Direct effects of interleukin-13 on signaling pathways for physiological responses in cultured human airway smooth muscle cells. - Wikidata - awgldk - TriplyDB

Direct effects of interleukin-13 on signaling pathways for physiological responses in cultured human airway smooth muscle cells.

Direct effects of interleukin-13 on signaling pathways for physiological responses in cultured human airway smooth muscle cells.

http://www.wikidata.org/entity/Q43660408

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Preclinical development of CAT-354, an IL-13 neutralizing antibody, for the treatment of severe uncontrolled asthma The effect of IL-13 and IL-13R130Q, a naturally occurring IL-13 polymorphism, on the gene expression of human airway smooth muscle cells IL-4 and IL-13 signaling in allergic airway disease Point: alterations in airway smooth muscle phenotype do/do not cause airway hyperresponsiveness in asthma Airway smooth muscle in asthma: just a target for bronchodilation?Airway hyperresponsiveness; smooth muscle as the principal actor CD26 (dipeptidyl-peptidase IV)-dependent recruitment of T cells in a rat asthma model ERK1/2 signaling pathway modulates the airway smooth muscle cell phenotype in the rat model of chronic asthma.IL-13 expression by blood T cells and not eosinophils is increased in asthma compared to non-asthmatic eosinophilic bronchitis.IL-9 induces CCL11 expression via STAT3 signalling in human airway smooth muscle cells.Regulation of human mast cell and basophil function by anaphylatoxins C3a and C5a.Interleukin-4 activates large-conductance, calcium-activated potassium (BKCa) channels in human airway smooth muscle cells.Importance of cytokines in murine allergic airway disease and human asthma.Expression of IL-4 receptor alpha on smooth muscle cells is not necessary for development of experimental allergic asthma.Human airway musculature on a chip: an in vitro model of allergic asthmatic bronchoconstriction and bronchodilation.Asthma, airway biology, and nasal disorders in AJRCCM 2001.Pro-asthmatic cytokines regulate unliganded and ligand-dependent glucocorticoid receptor signaling in airway smooth muscle.Effects of cytokines on contractile and dilator responses of airway smooth muscle.src homology 2 domain-containing tyrosine phosphatase SHP-1 controls the development of allergic airway inflammation.Interleukin-13: prospects for new treatments.Selective stimulation of IL-4 receptor on smooth muscle induces airway hyperresponsiveness in mice.New insights into the role of the mast cell in asthma.Mechanical stimuli and IL-13 interact at integrin adhesion complexes to regulate expression of smooth muscle myosin heavy chain in airway smooth muscle tissue.Interleukin-13 in asthma pathogenesis.G Protein βγ-subunit signaling mediates airway hyperresponsiveness and inflammation in allergic asthma.Mitogen-activated protein kinases and asthma.Central role of Muc5ac expression in mucous metaplasia and its regulation by conserved 5' elements The re-emergence of the mast cell as a pivotal cell in asthma pathogenesis.Abrogation of airway hyperresponsiveness but not inflammation by rho kinase insufficiency.IL-4 inhibits calcium transients in bovine trachealis cells by a ryanodine receptor-dependent mechanism Targeting phosphoinositide 3-kinase γ in airway smooth muscle cells to suppress interleukin-13-induced mouse airway hyperresponsiveness.Bitter taste receptor agonists alter mitochondrial function and induce autophagy in airway smooth muscle cells.The proximal STAT6 and NF-kappaB sites are responsible for IL-13- and TNF-alpha-induced RhoA transcriptions in human bronchial smooth muscle cells.Downregulation of SUMF2 gene in ovalbumin-induced rat model of allergic inflammation IL-13 Augments Compressive Stress-Induced Tissue Factor Expression in Human Airway Epithelial Cells Airway smooth muscle in the pathophysiology and treatment of asthma.Th2 cytokine-induced upregulation of 11beta-hydroxysteroid dehydrogenase-1 facilitates glucocorticoid suppression of proasthmatic airway smooth muscle function.Inhibition of PI3K promotes dilation of human small airways in a rho kinase-dependent manner.Interleukin-4, interleukin-13, signal transducer and activator of transcription factor 6, and allergic asthma.IL-13 R110Q, a Naturally Occurring IL-13 Polymorphism, Confers Enhanced Functional Activity in Cultured Human Bronchial Smooth Muscle Cells

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Direct effects of interleukin-13 on signaling pathways for physiological responses in cultured human airway smooth muscle cells.

http://www.wikidata.org/entity/Q43660408

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Church TL

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Jouvin MH

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Cloning and characterization of a specific interleukin (IL)-13 binding protein structurally related to the IL-5 receptor alpha chain

Interleukin-13: central mediator of allergic asthma

Genetic susceptibility to asthma--bronchial hyperresponsiveness coinherited with a major gene for atopy

Autocrine interaction between IL-5 and IL-1beta mediates altered responsiveness of atopic asthmatic sensitized airway smooth muscle

Identification of a novel inhibitor of mitogen-activated protein kinase kinase

Variation in the interleukin 4-receptor alpha gene confers susceptibility to asthma and atopy in ethnically diverse populations.

Regulation of TH1- and TH2-type cytokine expression and action in atopic asthmatic sensitized airway smooth muscle

Control of cytoskeletal mechanics by extracellular matrix, cell shape, and mechanical tension

Mechanotransduction across the cell surface and through the cytoskeleton.

Mechanism of cytokine-induced modulation of beta-adrenoceptor responsiveness in airway smooth muscle.

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