Genetic analysis of immortalizing functions of Epstein-Barr virus in human B lymphocytes.
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The Epstein-Barr virus nuclear protein 2 acidic domain forms a complex with a novel cellular coactivator that can interact with TFIIEMediation of Epstein-Barr virus EBNA-LP transcriptional coactivation by Sp100SCFSkp2 complex targeted by Epstein-Barr virus essential nuclear antigenExpression of Epstein-Barr virus nuclear antigen-1 induces B cell neoplasia in transgenic miceThe 62- and 80-kDa subunits of transcription factor IIH mediate the interaction with Epstein-Barr virus nuclear protein 2Characterization of the Epstein-Barr virus-inducible gene encoding the human leukocyte adhesion and activation antigen BLAST-1 (CD48)The Epstein-Barr virus oncogene product latent membrane protein 1 engages the tumor necrosis factor receptor-associated death domain protein to mediate B lymphocyte growth transformation and activate NF-kappaBTruncated mammalian Notch1 activates CBF1/RBPJk-repressed genes by a mechanism resembling that of Epstein-Barr virus EBNA2Epstein-Barr virus LMP1 blocks p16INK4a-RB pathway by promoting nuclear export of E2F4/5Epstein-Barr virus immortalization: Notch2 interacts with CBF1 and blocks differentiationEBNA2 and activated Notch induce expression of BATFThe EBV nuclear antigen 1 (EBNA1) enhances B cell immortalization several thousandfoldEpstein-Barr virus provides a new paradigm: a requirement for the immediate inhibition of apoptosis.The Role of Gammaherpesviruses in Cancer PathogenesisStructural and Functional Characterization of a Complex between the Acidic Transactivation Domain of EBNA2 and the Tfb1/p62 Subunit of TFIIHProtein kinase A associates with HA95 and affects transcriptional coactivation by Epstein-Barr virus nuclear proteins.Modulation of histone acetyltransferase activity through interaction of epstein-barr nuclear antigen 3C with prothymosin alphaRBP-L, a transcription factor related to RBP-JkappaExosomes derived from Burkitt's lymphoma cell lines induce proliferation, differentiation, and class-switch recombination in B cells.Interaction of Epstein-Barr virus nuclear antigen leader protein (EBNA-LP) with HS1-associated protein X-1: implication of cytoplasmic function of EBNA-LP.Mutational analysis of the J recombination signal sequence binding protein (RBP-J)/Epstein-Barr virus nuclear antigen 2 (EBNA2) and RBP-J/Notch interaction.Antisense to the Epstein-Barr virus (EBV)-encoded latent membrane protein 1 (LMP-1) sensitizes EBV-immortalized B cells to transforming growth factor-beta and chemotherapeutic agents.Promoter-specific targeting of human SWI-SNF complex by Epstein-Barr virus nuclear protein 2.Silencing of the Epstein-Barr virus latent membrane protein 1 gene by the Max-Mad1-mSin3A modulator of chromatin structureHyperphosphorylation of EBNA2 by Epstein-Barr virus protein kinase suppresses transactivation of the LMP1 promoter.Epstein-barr virus nuclear antigen 3C activates the latent membrane protein 1 promoter in the presence of Epstein-Barr virus nuclear antigen 2 through sequences encompassing an spi-1/Spi-B binding site.Interferon regulatory factor 7 is induced by Epstein-Barr virus latent membrane protein 1Sequence and functional analysis of EBNA-LP and EBNA2 proteins from nonhuman primate lymphocryptovirusesEpstein-barr virus transformation: involvement of latent membrane protein 1-mediated activation of NF-kappaB.Activated Notch1 modulates gene expression in B cells similarly to Epstein-Barr viral nuclear antigen 2.Dual expression of Epstein-Barr virus, latent membrane protein-1 and human papillomavirus-16 E6 transform primary mouse embryonic fibroblasts through NF-κB signalingEpstein-Barr virus-encoded nuclear antigen 2 activates the viral latent membrane protein promoter by modulating the activity of a negative regulatory element.Genetic dissection of cell growth arrest functions mediated by the Epstein-Barr virus lytic gene product, ZtaAn Epstein-Barr virus that expresses only the first 231 LMP1 amino acids efficiently initiates primary B-lymphocyte growth transformationA viral microRNA cluster strongly potentiates the transforming properties of a human herpesvirusNotch1IC partially replaces EBNA2 function in B cells immortalized by Epstein-Barr virus.The latency-associated nuclear antigen encoded by Kaposi's sarcoma-associated herpesvirus activates two major essential Epstein-Barr virus latent promotersActivated Notch1 can transiently substitute for EBNA2 in the maintenance of proliferation of LMP1-expressing immortalized B cells.EBNA-LP associates with cellular proteins including DNA-PK and HA95.Identification of major phosphorylation sites of Epstein-Barr virus nuclear antigen leader protein (EBNA-LP): ability of EBNA-LP to induce latent membrane protein 1 cooperatively with EBNA-2 is regulated by phosphorylation
P2860
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P2860
Genetic analysis of immortalizing functions of Epstein-Barr virus in human B lymphocytes.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年学术文章
@wuu
1989年学术文章
@zh
1989年学术文章
@zh-cn
1989年学术文章
@zh-hans
1989年学术文章
@zh-my
1989年学术文章
@zh-sg
1989年學術文章
@yue
1989年學術文章
@zh-hant
name
Genetic analysis of immortalizing functions of Epstein-Barr virus in human B lymphocytes.
@en
Genetic analysis of immortalizing functions of Epstein-Barr virus in human B lymphocytes.
@nl
type
label
Genetic analysis of immortalizing functions of Epstein-Barr virus in human B lymphocytes.
@en
Genetic analysis of immortalizing functions of Epstein-Barr virus in human B lymphocytes.
@nl
prefLabel
Genetic analysis of immortalizing functions of Epstein-Barr virus in human B lymphocytes.
@en
Genetic analysis of immortalizing functions of Epstein-Barr virus in human B lymphocytes.
@nl
P356
P1433
P1476
Genetic analysis of immortalizing functions of Epstein-Barr virus in human B lymphocytes.
@en
P2093
Hammerschmidt W
P2888
P304
P356
10.1038/340393A0
P407
P577
1989-08-01T00:00:00Z
P6179
1013091913