Macrophage depletion impairs oligodendrocyte remyelination following lysolecithin-induced demyelination. - Wikidata - awgldk - TriplyDB

Macrophage depletion impairs oligodendrocyte remyelination following lysolecithin-induced demyelination.

Macrophage depletion impairs oligodendrocyte remyelination following lysolecithin-induced demyelination.

http://www.wikidata.org/entity/Q43700744

about

Oligodendrogenesis in the normal and pathological central nervous system The innate immune system in demyelinating disease Inhibition of p53 transcriptional activity: a potential target for future development of therapeutic strategies for primary demyelination Macrophages promote axon regeneration with concurrent neurotoxicity TWEAK, via its receptor Fn14, is a novel regulator of mesenchymal progenitor cells and skeletal muscle regeneration Cellular and Molecular Mechanisms Underpinning Macrophage Activation during Remyelination Regenerative Capacity of Macrophages for Remyelination CNS Remyelination and the Innate Immune System Myelin damage and repair in pathologic CNS: challenges and prospects The Alzheimer's Disease Neuroimaging Initiative informatics core: A decade in review Remyelination therapy for multiple sclerosis Promoting remyelination in multiple sclerosis-recent advances Infiltrating blood-derived macrophages are vital cells playing an anti-inflammatory role in recovery from spinal cord injury in mice Nogo receptor inhibition enhances functional recovery following lysolecithin-induced demyelination in mouse optic chiasm Delayed nerve stimulation promotes axon-protective neurofilament phosphorylation, accelerates immune cell clearance and enhances remyelination in vivo in focally demyelinated nerves Myelin-mediated inhibition of oligodendrocyte precursor differentiation can be overcome by pharmacological modulation of Fyn-RhoA and protein kinase C signalling Oligodendrocytes: biology and pathology Dealing with Danger in the CNS: The Response of the Immune System to Injury.Inefficient clearance of myelin debris by microglia impairs remyelinating processes Progressive multiple sclerosis: from pathogenic mechanisms to treatment.Therapeutic inhibition of soluble brain TNF promotes remyelination by increasing myelin phagocytosis by microglia Importance of oligodendrocyte protection, BBB breakdown and inflammation for remyelination.Fingolimod (FTY720) enhances remyelination following demyelination of organotypic cerebellar slices.Administration of a Sigma Receptor Agonist Delays MCAO-Induced Neurodegeneration and White Matter Injury Glatiramer acetate-reactive T lymphocytes regulate oligodendrocyte progenitor cell number in vitro: role of IGF-2 Oligodendrocyte PTEN is required for myelin and axonal integrity, not remyelination Glatiramer acetate increases phagocytic activity of human monocytes in vitro and in multiple sclerosis patients Remyelination can be extensive in multiple sclerosis despite a long disease course.Oligodendrocyte fate after spinal cord injury.Immune regulatory mechanisms influence early pathology in spinal cord injury and in spontaneous autoimmune encephalomyelitis.Metabolic reprograming of mononuclear phagocytes in progressive multiple sclerosis.Dysfunction of platelet-derived growth factor receptor α (PDGFRα) represses the production of oligodendrocytes from arylsulfatase A-deficient multipotential neural precursor cells Neural precursors exhibit distinctly different patterns of cell migration upon transplantation during either the acute or chronic phase of EAE: a serial MR imaging study.Experimental demyelination and remyelination of murine spinal cord by focal injection of lysolecithin.Experimental autoimmune encephalomyelitis (EAE) as a model for multiple sclerosis (MS).Prox1 Inhibits Proliferation and Is Required for Differentiation of the Oligodendrocyte Cell Lineage in the Mouse.TLR3 Agonist Poly-IC Induces IL-33 and Promotes Myelin Repair Adoptive transfer of Th1-conditioned lymphocytes promotes axonal remodeling and functional recovery after spinal cord injury.Targeting CD64 mediates elimination of M1 but not M2 macrophages in vitro and in cutaneous inflammation in mice and patient biopsies.Regeneration of myelin sheaths of normal length and thickness in the zebrafish CNS correlates with growth of axons in caliber.

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P2860

Macrophage depletion impairs oligodendrocyte remyelination following lysolecithin-induced demyelination.

http://www.wikidata.org/entity/Q43700744

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2001 nî lūn-bûn

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Macrophage depletion impairs o ...... ecithin-induced demyelination.

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Macrophage depletion impairs o ...... ecithin-induced demyelination.

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Macrophage depletion impairs o ...... ecithin-induced demyelination.

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Macrophage depletion impairs o ...... ecithin-induced demyelination.

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Macrophage depletion impairs o ...... ecithin-induced demyelination.

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Macrophage depletion impairs o ...... ecithin-induced demyelination.

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Franklin RJ

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Setzu A

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Van Rooijen N

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P2860

Promotion of wound repair in old mice by local injection of macrophages

The role of macrophages in demyelination.

The liposome-mediated macrophage 'suicide' technique.

West Nile virus neuroinvasion and encephalitis induced by macrophage depletion in mice.

Liposome mediated depletion of macrophages: mechanism of action, preparation of liposomes and applications.

Distinctive patterns of PDGF-A, FGF-2, IGF-I, and TGF-beta1 gene expression during remyelination of experimentally-induced spinal cord demyelination.

Macrophages enhance muscle satellite cell proliferation and delay their differentiation.

Native and modified low density lipoproteins increase the functional expression of the macrophage class B scavenger receptor, CD36.

Macrophages in CNS remyelination: friend or foe?

Suppression of experimental allergic encephalomyelitis in Lewis rats after elimination of macrophages.

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10.1002/GLIA.1085

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3

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35

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