IL-12, while beneficial, is not essential for the host response to VSV encephalitis.
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Interleukin (IL)-12 receptor beta1 or IL-12 receptor beta 2 deficiency in mice indicates that IL-12 and IL-23 are not essential for host recovery from viral encephalitis.Disruption of IFN-gamma- mediated antiviral activity in neurons: the role of cannabinoidsGene expression contributing to recruitment of circulating cells in response to vesicular stomatitis virus infection of the CNS.Melatonin decreases nitric oxide production and lipid peroxidation and increases interleukin-1 beta in the brain of mice infected by the Venezuelan equine encephalomyelitis virus.
P2860
IL-12, while beneficial, is not essential for the host response to VSV encephalitis.
description
2002 nî lūn-bûn
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2002年の論文
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2002年学术文章
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IL-12, while beneficial, is not essential for the host response to VSV encephalitis.
@en
IL-12, while beneficial, is not essential for the host response to VSV encephalitis.
@nl
type
label
IL-12, while beneficial, is not essential for the host response to VSV encephalitis.
@en
IL-12, while beneficial, is not essential for the host response to VSV encephalitis.
@nl
prefLabel
IL-12, while beneficial, is not essential for the host response to VSV encephalitis.
@en
IL-12, while beneficial, is not essential for the host response to VSV encephalitis.
@nl
P1476
IL-12, while beneficial, is not essential for the host response to VSV encephalitis.
@en
P2093
David A Chesler
P356
10.1016/S0165-5728(02)00257-6
P577
2002-10-01T00:00:00Z