Peripheral administration of an angiotensin II AT(1) receptor antagonist decreases the hypothalamic-pituitary-adrenal response to isolation Stress.
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Angiotensin type 1 receptor inhibition enhances the extinction of fear memory.Peripherally administered angiotensin II AT1 receptor antagonists are anti-stress compounds in vivoRenal dopaminergic defect in C57Bl/6J miceCentral angiotensin II stimulation promotes β amyloid production in Sprague Dawley rats.Inhibitory effects of olmesartan on catecholamine secretion from the perfused rat adrenal medullaBlockade of brain angiotensin II AT1 receptors ameliorates stress, anxiety, brain inflammation and ischemia: Therapeutic implications.Long-term angiotensin II AT1 receptor inhibition produces adipose tissue hypotrophy accompanied by increased expression of adiponectin and PPARgamma.Angiotensin II AT(1) receptor blockers ameliorate inflammatory stress: a beneficial effect for the treatment of brain disorders.Novel mechanism within the paraventricular nucleus reduces both blood pressure and hypothalamic pituitary-adrenal axis responses to acute stress.Angiotensin II AT1 receptor blocker candesartan prevents the fast up-regulation of cerebrocortical benzodiazepine-1 receptors induced by acute inflammatory and restraint stress.Effects of life-long caloric restriction and voluntary exercise on age-related changes in levels of catecholamine biosynthetic enzymes and angiotensin II receptors in the rat adrenal medulla and hypothalamus.Regulation of angiotensin II type 2 receptor gene expression in the adrenal medulla by acute and repeated immobilization stress.Angiotensin II AT(1) receptor blockers as treatments for inflammatory brain disorders.Influence of Fimasartan (a Novel AT(1) Receptor Blocker) on Catecholamine Release in the Adrenal Medulla of Spontaneously Hypertensive Rats.Brain and peripheral angiotensin II play a major role in stress.Adrenocortical dysregulation as a major player in insulin resistance and onset of obesity.Angiotensin II AT1 receptor blockade decreases lipopolysaccharide-induced inflammation in the rat adrenal glandTargeting brain angiotensin and corticotrophin-releasing hormone systems interaction for the treatment of mood and alcohol use disorders.Estrogen reduces aldosterone, upregulates adrenal angiotensin II AT2 receptors and normalizes adrenomedullary Fra-2 in ovariectomized rats.Angiotensin II AT1 receptor blockade prevents the hypothalamic corticotropin-releasing factor response to isolation stress.Angiotensin II type 1 receptors may not influence response of spinal autonomic neurons to axonal damageGene expression profiling following maternal deprivation: involvement of the brain Renin-Angiotensin system.A peripherally administered, centrally acting angiotensin II AT2 antagonist selectively increases brain AT1 receptors and decreases brain tyrosine hydroxylase transcription, pituitary vasopressin and ACTH.Anti-inflammatory effects of angiotensin receptor blockers in the brain and the periphery.Angiotensin II AT(1) receptor blockade selectively enhances brain AT(2) receptor expression, and abolishes the cold-restraint stress-induced increase in tyrosine hydroxylase mRNA in the locus coeruleus of spontaneously hypertensive rats.In vivo Angiotensin II AT1 receptor blockade selectively inhibits LPS-induced innate immune response and ACTH release in rat pituitary gland.Telmisartan ameliorates glutamate-induced neurotoxicity: roles of AT(1) receptor blockade and PPARγ activationInteraction between irbesartan, peroxisome proliferator-activated receptor (PPAR-γ), and adiponectin in the regulation of blood pressure and renal function in spontaneously hypertensive rats.Effects of losartan on catecholamine release in the isolated rat adrenal glandRenin-Angiotensin system hyperactivation can induce inflammation and retinal neural dysfunction.Candesartan prevents impairment of recall caused by repeated stress in rats.Anti-inflammatory effects of angiotensin II AT1 receptor antagonism prevent stress-induced gastric injury.Losartan may prevent the elevation of plasma glucose, corticosterone and catecholamine levels induced by chronic stress.A centrally acting, anxiolytic angiotensin II AT1 receptor antagonist prevents the isolation stress-induced decrease in cortical CRF1 receptor and benzodiazepine binding.Coupling corticotropin-releasing-hormone and angiotensin converting enzyme 2 dampens stress responsiveness in male mice.Angiotensin II AT2 Receptors Contribute to Regulate the Sympathoadrenal and Hormonal Reaction to Stress Stimuli.AT1 and AT2 Receptors in the Prelimbic Cortex Modulate the Cardiovascular Response Evoked by Acute Exposure to Restraint Stress in Rats.Anti-stress and nootropic activity of drugs affecting the renin-angiotensin system in rats based on indirect biochemical evidence.Chronic administration of an angiotensin II receptor antagonist resets the hypothalamic-pituitary-adrenal (HPA) axis and improves the affect of patients with diabetes mellitus type 2: preliminary results.Chronic AT1 blockade improves glucose homeostasis in obese OLETF rats.
P2860
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P2860
Peripheral administration of an angiotensin II AT(1) receptor antagonist decreases the hypothalamic-pituitary-adrenal response to isolation Stress.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
Peripheral administration of an angiotensin II AT
@nl
Peripheral administration of a ...... response to isolation Stress.
@en
type
label
Peripheral administration of an angiotensin II AT
@nl
Peripheral administration of a ...... response to isolation Stress.
@en
prefLabel
Peripheral administration of an angiotensin II AT
@nl
Peripheral administration of a ...... response to isolation Stress.
@en
P2093
P356
P1433
P1476
Peripheral administration of a ...... response to isolation Stress.
@en
P2093
A Carranza
A Falcón-Neri
A V Juorio
J A Terrón
J M Saavedra
M Barontini
Y Nishimura
P304
P356
10.1210/ENDO.142.9.8366
P407
P577
2001-09-01T00:00:00Z