Differential subcellular actions of ACE inhibitors and AT(1) receptor antagonists on cardiac remodeling induced by chronic inhibition of NO synthesis in rats.

Differential subcellular actions of ACE inhibitors and AT(1) receptor antagonists on cardiac remodeling induced by chronic inhibition of NO synthesis in rats.

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http://www.wikidata.org/entity/Q43744724
Upregulation of adrenomedullin and its receptor components during cardiomyocyte hypertrophy induced by chronic inhibition of nitric oxide synthesis in ratsAngiotensin II type 1 receptor blocker attenuates the activation of ERK and NADPH oxidase by mechanical strain in mesangial cells in the absence of angiotensin II.Caveolin-1 ablation reduces the adverse cardiovascular effects of N-omega-nitro-L-arginine methyl ester and angiotensin II.Pharmacological and clinical studies with temocapril, an angiotensin converting enzyme inhibitor that is excreted in the bile.Sub-acute effect of N(G)-nitro-l-arginine methyl-ester (L-NAME) on biochemical indices in rats: Protective effects of Kolaviron and extract of Curcuma longa L.Brachial artery diameter and the right ventricle: the Multi-Ethnic Study of Atherosclerosis-right ventricle study.Potential impact of carbohydrate and fat intake on pathological left ventricular hypertrophy.Nitric oxide regulates angiotensin-I converting enzyme under static conditions but not under shear stress.Hepatic morphological alterations, glycogen content and cytochrome P450 activities in rats treated chronically with N(omega)-nitro-L-arginine methyl ester (L-NAME).Cardiac hypertrophy and oxidative stress: a leap of faith or stark reality?Cardiovascular status following combined angiotensin-converting enzyme and AT1 receptor inhibition in conscious spontaneously hypertensive rats.Thyroxine-induced cardiac hypertrophy: influence of adrenergic nervous system versus renin-angiotensin system on myocyte remodeling.Cardiopulmonary effects of chronic administration of the NO synthase inhibitor L-NAME in the chick embryo.The antagonism of aldosterone receptor prevents the development of hypertensive heart failure induced by chronic inhibition of nitric oxide synthesis in rats.Olmesartan attenuates pressure-overload- or post-infarction-induced cardiac remodeling in mice.
P2860

Differential subcellular actions of ACE inhibitors and AT(1) receptor antagonists on cardiac remodeling induced by chronic inhibition of NO synthesis in rats.

Item
http://www.wikidata.org/entity/Q43744724
description
2001 nî lūn-bûn
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2001年の論文
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2001年学术文章
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2001年学术文章
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2001年学术文章
@zh-cn
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@zh-my
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2001年學術文章
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name
Differential subcellular actions of ACE inhibitors and AT
@nl
Differential subcellular actio ...... ition of NO synthesis in rats.
@en
type
label
Differential subcellular actions of ACE inhibitors and AT
@nl
Differential subcellular actio ...... ition of NO synthesis in rats.
@en
prefLabel
Differential subcellular actions of ACE inhibitors and AT
@nl
Differential subcellular actio ...... ition of NO synthesis in rats.
@en
P1433
P1476
P2093
P304
P31
P356
P407
P433
P478
P50
P577
P698
P356
P1433
P1476
Differential subcellular actio ...... bition of NO synthesis in rats
@en
P2093
P304
P31
P356
P407
P433
P478
P50
P577
P698