A coding variant in SR-BI (I179N) significantly increases atherosclerosis in mice.

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Single amino acid mutation of SR-BI decreases infectivity of hepatitis C virus derived from cell culture in a cell culture model.

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A coding variant in SR-BI (I179N) significantly increases atherosclerosis in mice.

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http://www.wikidata.org/entity/Q43751225

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2013 nî lūn-bûn

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2013年学术文章

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A coding variant in SR-BI (I179N) significantly increases atherosclerosis in mice.

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A coding variant in SR-BI

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type

Item

label

A coding variant in SR-BI (I179N) significantly increases atherosclerosis in mice.

@en

A coding variant in SR-BI

@nl

prefLabel

A coding variant in SR-BI (I179N) significantly increases atherosclerosis in mice.

@en

A coding variant in SR-BI

@nl

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A coding variant in SR-BI (I179N) significantly increases atherosclerosis in mice.

@en

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Anthony P Kent

http://www.w3.org/2001/XMLSchema#string

Antonino Picataggi

http://www.w3.org/2001/XMLSchema#string

Daniel J Rader

http://www.w3.org/2001/XMLSchema#string

Geoffrey F Lim

http://www.w3.org/2001/XMLSchema#string

Ioannis M Stylianou

http://www.w3.org/2001/XMLSchema#string

John S Millar

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Biological, clinical and population relevance of 95 loci for blood lipids

Identification of scavenger receptor SR-BI as a high density lipoprotein receptor

Plasma HDL cholesterol and risk of myocardial infarction: a mendelian randomisation study

Novel ENU-induced point mutation in scavenger receptor class B, member 1, results in liver specific loss of SCARB1 protein.

Impact of gene variants on sex-specific regulation of human Scavenger receptor class B type 1 (SR-BI) expression in liver and association with lipid levels in a population-based study.

Scavenger receptor BI facilitates hepatic very low density lipoprotein production in mice.

Deficiency of scavenger receptor BI leads to impaired lymphocyte homeostasis and autoimmune disorders in mice.

Genetic basis of atherosclerosis: insights from mice and humans

Cholesterol efflux pathways and other potential mechanisms involved in the athero-protective effect of high density lipoproteins.

Increased HDL cholesterol and apoA-I in humans and mice treated with a novel SR-BI inhibitor.

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s00335-013-9459-x

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scholarly article

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10.1007/S00335-013-9459-X

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7-8

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2013-05-31T00:00:00Z

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1038742478

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atherosclerosis

A coding variant in SR-BI (I179N) significantly increases atherosclerosis in mice.

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A coding variant in SR-BI (I179N) significantly increases atherosclerosis in mice.

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