Inflammatory stimuli induce a new K+ outward current in cultured rat microglia.
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The Role of Ion Channels in Microglial Activation and Proliferation - A Complex Interplay between Ligand-Gated Ion Channels, K(+) Channels, and Intracellular Ca(2.)Predominant functional expression of Kv1.3 by activated microglia of the hippocampus after Status epilepticus.Prostaglandin E2 and 4-aminopyridine prevent the lipopolysaccharide-induced outwardly rectifying potassium current and interleukin-1beta production in cultured rat microgliaIon channels in cultured microglia.Microglia in neurodegeneration: molecular aspects.HERG-like K+ channels in microglia.Differential effects of Th1, monocyte/macrophage and Th2 cytokine mixtures on early gene expression for molecules associated with metabolism, signaling and regulation in central nervous system mixed glial cell cultures.Expression and contributions of the Kir2.1 inward-rectifier K(+) channel to proliferation, migration and chemotaxis of microglia in unstimulated and anti-inflammatory states.Responses of rat and mouse primary microglia to pro- and anti-inflammatory stimuli: molecular profiles, K+ channels and migration.Microglial K(+) channel expression in young adult and aged mice.Differential Kv1.3, KCa3.1, and Kir2.1 expression in "classically" and "alternatively" activated microglia.Characterization and transduction mechanisms of purinoceptors in activated rat microglia.Characterization and possible function of adenosine 5'-triphosphate receptors in activated rat microglia.Charge compensation for NADPH oxidase activity in microglia in rat brain slices does not involve a proton current.K+ channels and the microglial respiratory burst.Inhibition of store-operated Ca(2+) influx by acidic extracellular pH in cultured human microglia.Purinergic receptors on microglial cells: functional expression in acute brain slices and modulation of microglial activation in vitro.The secret life of ion channels: Kv1.3 potassium channels and proliferation.Sodium channel in isolated human brain macrophages (microglia).Novel objective classification of reactive microglia following hypoglossal axotomy using hierarchical cluster analysis.Epidermal growth factor is a motility factor for microglial cells in vitro: evidence for EGF receptor expression.Altered outward-rectifying K(+) current reveals microglial activation induced by HIV-1 Tat protein.Electrophysiological properties of microglial cells in normal and pathologic rat brain slices.Comparing Effects of Transforming Growth Factor β1 on Microglia From Rat and Mouse: Transcriptional Profiles and Potassium Channels.
P2860
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P2860
Inflammatory stimuli induce a new K+ outward current in cultured rat microglia.
description
1992 nî lūn-bûn
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1992年の論文
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1992年学术文章
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1992年学术文章
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1992年学术文章
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1992年學術文章
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name
Inflammatory stimuli induce a new K+ outward current in cultured rat microglia.
@en
Inflammatory stimuli induce a new K+ outward current in cultured rat microglia.
@nl
type
label
Inflammatory stimuli induce a new K+ outward current in cultured rat microglia.
@en
Inflammatory stimuli induce a new K+ outward current in cultured rat microglia.
@nl
prefLabel
Inflammatory stimuli induce a new K+ outward current in cultured rat microglia.
@en
Inflammatory stimuli induce a new K+ outward current in cultured rat microglia.
@nl
P2093
P1433
P1476
Inflammatory stimuli induce a new K+ outward current in cultured rat microglia.
@en
P2093
P J Gebicke-Haerter
W Nörenberg
P304
P356
10.1016/0304-3940(92)90587-W
P407
P577
1992-12-01T00:00:00Z