Bioenergetics in cardiac hypertrophy: mitochondrial respiration as a pathological target of NO*.
about
Activation of Hsp90/NOS and increased NO generation does not impair mitochondrial respiratory chain by competitive binding at cytochrome c oxidase in low oxygen concentrations.Prologue: Nitric oxide--hormones, metabolism, and function.Cardioprotection and mitochondrial S-nitrosation: effects of S-nitroso-2-mercaptopropionyl glycine (SNO-MPG) in cardiac ischemia-reperfusion injuryThe complex II inhibitor atpenin A5 protects against cardiac ischemia-reperfusion injury via activation of mitochondrial KATP channelsROS and RNS signaling in heart disorders: could antioxidant treatment be successful?
P2860
Bioenergetics in cardiac hypertrophy: mitochondrial respiration as a pathological target of NO*.
description
2001 nî lūn-bûn
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2001年の論文
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2001年学术文章
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2001年学术文章
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name
Bioenergetics in cardiac hyper ...... a pathological target of NO*.
@en
Bioenergetics in cardiac hyper ...... a pathological target of NO*.
@nl
type
label
Bioenergetics in cardiac hyper ...... a pathological target of NO*.
@en
Bioenergetics in cardiac hyper ...... a pathological target of NO*.
@nl
prefLabel
Bioenergetics in cardiac hyper ...... a pathological target of NO*.
@en
Bioenergetics in cardiac hyper ...... a pathological target of NO*.
@nl
P2093
P2860
P1476
Bioenergetics in cardiac hyper ...... a pathological target of NO*.
@en
P2093
Anderson PG
Brookes PS
Darley-Usmar VM
P2860
P304
P356
10.1152/AJPHEART.2001.281.6.H2261
P577
2001-12-01T00:00:00Z