Ras links cellular morphogenesis to virulence by regulation of the MAP kinase and cAMP signalling pathways in the pathogenic fungus Candida albicans. - Wikidata - awgldk - TriplyDB

Ras links cellular morphogenesis to virulence by regulation of the MAP kinase and cAMP signalling pathways in the pathogenic fungus Candida albicans.

Ras links cellular morphogenesis to virulence by regulation of the MAP kinase and cAMP signalling pathways in the pathogenic fungus Candida albicans.

http://www.wikidata.org/entity/Q43808791

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An evolutionary proteomics approach identifies substrates of the cAMP-dependent protein kinase Regulation of phenotypic transitions in the fungal pathogen Candida albicans Hypoxia and fungal pathogenesis: to air or not to air?Unveiling the structural basis for translational ambiguity tolerance in a human fungal pathogen Mds3 regulates morphogenesis in Candida albicans through the TOR pathway The Hsp90 co-chaperone Sgt1 governs Candida albicans morphogenesis and drug resistance Mitochondrial Activity and Cyr1 Are Key Regulators of Ras1 Activation of C. albicans Virulence Pathways Cdc24, the GDP-GTP exchange factor for Cdc42, is required for invasive hyphal growth of Candida albicans.Polarized hyphal growth in Candida albicans requires the Wiskott-Aldrich Syndrome protein homolog Wal1p Candida and invasive candidiasis: back to basics.Hgc1 mediates dynamic Candida albicans-endothelium adhesion events during circulation.Cyclin Cln3p links G1 progression to hyphal and pseudohyphal development in Candida albicans.The G protein-coupled receptor Gpr1 and the Galpha protein Gpa2 act through the cAMP-protein kinase A pathway to induce morphogenesis in Candida albicans.Signaling through adenylyl cyclase is essential for hyphal growth and virulence in the pathogenic fungus Candida albicans.Identification of GIG1, a GlcNAc-induced gene in Candida albicans needed for normal sensitivity to the chitin synthase inhibitor nikkomycin Z.Candida albicans killing by RAW 264.7 mouse macrophage cells: effects of Candida genotype, infection ratios, and gamma interferon treatment.Modulation of morphogenesis in Candida albicans by various small molecules.Regulatory circuitry governing fungal development, drug resistance, and disease.A fungus-specific ras homolog contributes to the hyphal growth and virulence of Aspergillus fumigatus In vivo inhibitory effect on the biofilm formation of Candida albicans by liverwort derived riccardin D.Ras pathway signaling accelerates programmed cell death in the pathogenic fungus Candida albicans G protein-coupled receptor Gpr4 senses amino acids and activates the cAMP-PKA pathway in Cryptococcus neoformans.Cdc42p GTPase regulates the budded-to-hyphal-form transition and expression of hypha-specific transcripts in Candida albicans.Ash1 protein, an asymmetrically localized transcriptional regulator, controls filamentous growth and virulence of Candida albicans Ras GTPases modulate morphogenesis, sporulation and cellulase gene expression in the cellulolytic fungus Trichoderma reesei.Candida albicans cell-type switching and functional plasticity in the mammalian host.Candida albicans Czf1 and Efg1 coordinate the response to farnesol during quorum sensing, white-opaque thermal dimorphism, and cell death.Low concentrations of hydrogen peroxide or nitrite induced of Paracoccidioides brasiliensis cell proliferation in a Ras-dependent manner.Ras1 acts through duplicated Cdc42 and Rac proteins to regulate morphogenesis and pathogenesis in the human fungal pathogen Cryptococcus neoformans.Effect of tetrandrine against Candida albicans biofilms Conjugated linoleic acid inhibits hyphal growth in Candida albicans by modulating Ras1p cellular levels and downregulating TEC1 expression.Hyphal growth in Candida albicans does not require induction of hyphal-specific gene expression The quorum-sensing molecules farnesol/homoserine lactone and dodecanol operate via distinct modes of action in Candida albicans N-acetylglucosamine (GlcNAc) induction of hyphal morphogenesis and transcriptional responses in Candida albicans are not dependent on its metabolism Roles of Ras1 membrane localization during Candida albicans hyphal growth and farnesol response Ser or Leu: structural snapshots of mistranslation in Candida albicans.Morphogenesis in Candida albicans.Calcineurin is essential for virulence in Candida albicans.Regulation of filamentation in the human fungal pathogen Candida tropicalis.Novel roles for GlcNAc in cell signaling.

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Ras links cellular morphogenesis to virulence by regulation of the MAP kinase and cAMP signalling pathways in the pathogenic fungus Candida albicans.

http://www.wikidata.org/entity/Q43808791

description

2001 nî lūn-bûn

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2001年の論文

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年學術文章

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2001年學術文章

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name

Ras links cellular morphogenes ...... genic fungus Candida albicans.

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Ras links cellular morphogenes ...... genic fungus Candida albicans.

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type

label

Ras links cellular morphogenes ...... genic fungus Candida albicans.

@en

Ras links cellular morphogenes ...... genic fungus Candida albicans.

@nl

prefLabel

Ras links cellular morphogenes ...... genic fungus Candida albicans.

@en

Ras links cellular morphogenes ...... genic fungus Candida albicans.

@nl

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J.1365-2958.2001.02672.X

P1433

Molecular Microbiology

P1476

Ras links cellular morphogenes ...... genic fungus Candida albicans.

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P2093

Dignard D

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Harcus D

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Johnson L

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Leberer E

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Schröppel K

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Ushinsky S

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P2860

Mammalian Ras interacts directly with the serine/threonine kinase Raf

Ras2 signals via the Cdc42/Ste20/mitogen-activated protein kinase module to induce filamentous growth in Saccharomyces cerevisiae

Pheromone signalling and polarized morphogenesis in yeast.

Genetic analysis of yeast RAS1 and RAS2 genes.

Cyclic AMP-dependent protein kinase regulates pseudohyphal differentiation in Saccharomyces cerevisiae.

Yeast pseudohyphal growth is regulated by GPA2, a G protein alpha homolog

The three yeast A kinases have specific signaling functions in pseudohyphal growth

Unipolar cell divisions in the yeast S. cerevisiae lead to filamentous growth: regulation by starvation and RAS.

Involvement of distinct G-proteins, Gpa2 and Ras, in glucose- and intracellular acidification-induced cAMP signalling in the yeast Saccharomyces cerevisiae.

GPR1 encodes a putative G protein-coupled receptor that associates with the Gpa2p Galpha subunit and functions in a Ras-independent pathway.

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673-687

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scholarly article

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10.1046/J.1365-2958.2001.02672.X

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3

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42

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David Y. Thomas

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2001-11-01T00:00:00Z

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11632481

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11722734

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