High glucose increases glomerular filtration barrier permeability by activating protein kinase G type Iα subunits in a Nox4-dependent manner.
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Thioredoxin-interacting protein: pathophysiology and emerging pharmacotherapeutics in cardiovascular disease and diabetes.The emerging role of fibroblast growth factor 21 in diabetic nephropathy.Role of Protein Kinase G and Reactive Oxygen Species in the Regulation of Podocyte Function in Health and Disease.Have dipeptidyl peptidase-4 inhibitors ameliorated the vascular complications of type 2 diabetes in large-scale trials? The potential confounding effect of stem-cell chemokines.Intracellular calcium signaling regulates glomerular filtration barrier permeability: the role of the PKGIα-dependent pathway.NMDA receptors participate in the progression of diabetic kidney disease by decreasing Cdc42-GTP activation in podocytes.
P2860
High glucose increases glomerular filtration barrier permeability by activating protein kinase G type Iα subunits in a Nox4-dependent manner.
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2013 nî lūn-bûn
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name
High glucose increases glomeru ...... ts in a Nox4-dependent manner.
@en
High glucose increases glomeru ...... ts in a Nox4-dependent manner.
@nl
type
label
High glucose increases glomeru ...... ts in a Nox4-dependent manner.
@en
High glucose increases glomeru ...... ts in a Nox4-dependent manner.
@nl
prefLabel
High glucose increases glomeru ...... ts in a Nox4-dependent manner.
@en
High glucose increases glomeru ...... ts in a Nox4-dependent manner.
@nl
P50
P1476
High glucose increases glomeru ...... its in a Nox4-dependent manner
@en
P2093
Maciej Jankowski
P304
P356
10.1016/J.YEXCR.2013.09.005
P407
P577
2013-09-13T00:00:00Z