N-acetylaspartate is an axon-specific marker of mature white matter in vivo: a biochemical and immunohistochemical study on the rat optic nerve.
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Dietary supplementation with n-3 fatty acids from weaning limits brain biochemistry and behavioural changes elicited by prenatal exposure to maternal inflammation in the mouse model.Magnetic resonance spectroscopic determination of a neuronal and axonal marker in white matter predicts reversibility of deficits in secondary normal pressure hydrocephalusRelationship between fractional anisotropy of cerebral white matter and metabolite concentrations measured using (1)H magnetic resonance spectroscopy in healthy adults.Cerebral plasticity in multiple sclerosis: insights from fMRI.Occipital proton magnetic resonance spectroscopy (1H-MRS) reveals normal metabolite concentrations in retinal visual field defects.Polyphenon E, non-futile at neuroprotection in multiple sclerosis but unpredictably hepatotoxic: Phase I single group and phase II randomized placebo-controlled studies.Imaging of multiple sclerosis: role in neurotherapeutics.Magnetic resonance spectroscopy in the monitoring of multiple sclerosis.Whole-brain N-acetylaspartate as a surrogate marker of neuronal damage in diffuse neurologic disorders.Magnetic resonance imaging as a tool to examine the neuropathology of multiple sclerosis.N-Acetylaspartate in the CNS: from neurodiagnostics to neurobiology.Serial proton MR spectroscopy and diffusion tensor imaging in infantile Balo's concentric sclerosis.N-acetylaspartic acid in cerebrospinal fluid of multiple sclerosis patients determined by gas-chromatography-mass spectrometry.Age-modulated association between prefrontal NAA and the BDNF gene.1H-MR spectroscopy in traumatic brain injury.In vivo evidence of glutamate toxicity in multiple sclerosis.Normal appearing and diffusely abnormal white matter in patients with multiple sclerosis assessed with quantitative MR.Multiple regional 1H-MR spectroscopy in multiple system atrophy: NAA/Cr reduction in pontine base as a valuable diagnostic markerAcute metabolic brain changes following traumatic brain injury and their relevance to clinical severity and outcome.Increased concentrations of glutamate and glutamine in normal-appearing white matter of patients with multiple sclerosis and normal MR imaging brain scans.N-acetylaspartate (NAA) and N-acetylaspartylglutamate (NAAG) promote growth and inhibit differentiation of glioma stem-like cellsMetabolic maturation of the human brain from birth through adolescence: insights from in vivo magnetic resonance spectroscopy.Processing speed is correlated with cerebral health markers in the frontal lobes as quantified by neuroimaging.Moderate relationships between NAA and cognitive ability in healthy adults: implications for cognitive spectroscopyTraumatology of the optic nerve and contribution of crystallins to axonal regeneration.Neuropathologic correlates of magnetic resonance imaging in multiple sclerosis.Adapting brain metabolism to myelination and long-range signal transduction.Endogenous aspartoacylase expression is responsive to glutamatergic activity in vitro and in vivo.Prevention of axonal injury using calpain inhibitor in chronic progressive experimental autoimmune encephalomyelitis.Cerebrospinal fluid and blood biomarkers of neuroaxonal damage in multiple sclerosis.Short-term cuprizone feeding verifies N-acetylaspartate quantification as a marker of neurodegeneration.Apolipoprotein E4, Gender, Body Mass Index, Inflammation, Insulin Resistance, and Air Pollution Interactions: Recipe for Alzheimer's Disease Development in Mexico City Young Females.Age-related change in brain metabolite abnormalities in autism: a meta-analysis of proton magnetic resonance spectroscopy studies.Evolving Wallerian degeneration after transient retinal ischemia in mice characterized by diffusion tensor imaging.Megalencephalic leukoencephalopathy with subcortical cysts in an adult: quantitative proton MR spectroscopy and diffusion tensor MRI.Diffuse neuroaxonal involvement in mucolipidosis IV as assessed by proton magnetic resonance spectroscopic imaging.Cerebral structure and metabolism and long-term outcome in small-for-gestational-age preterm neonates.MRS of pilocytic astrocytoma: The peak at 2 ppm may not be NAA.Reduced N-acetylaspartate levels in mice lacking aralar, a brain- and muscle-type mitochondrial aspartate-glutamate carrier.Early reduction of total N-acetyl-aspartate-compounds in patients with classical vanishing white matter disease. A long-term follow-up MRS study.
P2860
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P2860
N-acetylaspartate is an axon-specific marker of mature white matter in vivo: a biochemical and immunohistochemical study on the rat optic nerve.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
N-acetylaspartate is an axon-s ...... study on the rat optic nerve.
@en
N-acetylaspartate is an axon-s ...... study on the rat optic nerve.
@nl
type
label
N-acetylaspartate is an axon-s ...... study on the rat optic nerve.
@en
N-acetylaspartate is an axon-s ...... study on the rat optic nerve.
@nl
prefLabel
N-acetylaspartate is an axon-s ...... study on the rat optic nerve.
@en
N-acetylaspartate is an axon-s ...... study on the rat optic nerve.
@nl
P2093
P2860
P356
P1433
P1476
N-acetylaspartate is an axon-s ...... study on the rat optic nerve.
@en
P2093
Bruce D Trapp
Carl Bjartmar
Erica Dupree
Jan Battistuta
Nobuo Terada
P2860
P356
10.1002/ANA.10052
P577
2002-01-01T00:00:00Z