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Chronic deficit in the expression of voltage-gated potassium channel Kv3.4 subunit in the hippocampus of pilocarpine-treated epileptic ratsAnticonvulsant activity of β-caryophyllene against pentylenetetrazol-induced seizures.Increased susceptibility to pentylenetetrazol following survival of cerebral malaria in mice.Anticonvulsant activity of Caryocar coriaceum Wittm. fixed pulp oil against pentylenetetrazol-induced seizures.The Impact of Previous Physical Training on Redox Signaling after Traumatic Brain Injury in Rats: A Behavioral and Neurochemical Approach.Lycopene protects against acute zearalenone-induced oxidative, endocrine, inflammatory and reproductive damages in male mice.Nitric oxide and potassium channels mediate GM1 ganglioside-induced vasorelaxation.Chronic administration of methylmalonate on young rats alters neuroinflammatory markers and spatial memory.Neuroprotective effect of physical exercise in a mouse model of Alzheimer's disease induced by β-amyloid₁₋₄₀ peptide.Creatine protects against the convulsive behavior and lactate production elicited by the intrastriatal injection of methylmalonate.Involvement of NO in the convulsive behavior and oxidative damage induced by the intrastriatal injection of methylmalonate.Adaptation to oxidative challenge induced by chronic physical exercise prevents Na+,K+-ATPase activity inhibition after traumatic brain injury.Additive anticonvulsant effects of creatine supplementation and physical exercise against pentylenetetrazol-induced seizures.Prostaglandin E2 modulates Na+,K+-ATPase activity in rat hippocampus: implications for neurological diseases.Kinetic characterization of l-[(3)H]glutamate uptake inhibition and increase oxidative damage induced by glutaric acid in striatal synaptosomes of rats.Methylmalonate Induces Inflammatory and Apoptotic Potential: A Link to Glial Activation and Neurological Dysfunction.Na+,K+-ATPase activity impairment after experimental traumatic brain injury: relationship to spatial learning deficits and oxidative stress.Neuromodulatory effect of creatine on extracellular action potentials in rat hippocampus: role of NMDA receptors.Increased xanthine oxidase-related ROS production and TRPV1 synthesis preceding DOMS post-eccentric exercise in rats.A neuronal disruption in redox homeostasis elicited by ammonia alters the glycine/glutamate (GABA) cycle and contributes to MMA-induced excitability.Fumonisin B1 facilitates seizures induced by pentylenetetrazol in mice.alpha-Tocopherol protects against pentylenetetrazol- and methylmalonate-induced convulsions.Cyclooxygenase-2/PGE2 pathway facilitates pentylenetetrazol-induced seizures.Effectiveness of creatine monohydrate on seizures and oxidative damage induced by methylmalonate.The involvement of the polyamines binding sites at the NMDA receptor in creatine-induced spatial learning enhancement.GM1 ganglioside prevents seizures, Na+,K+-ATPase activity inhibition and oxidative stress induced by glutaric acid and pentylenetetrazole.The role of kinin B1 receptor and the effect of angiotensin I-converting enzyme inhibition on acute gout attacks in rodents.Commentary on Kaushik et al.: Prostaglandin D2 is crucial for seizure suppression and postictal sleep. Novel evidence supporting a role for prostanoid receptors in seizure control.Atorvastatin withdrawal elicits oxidative/nitrosative damage in the rat cerebral cortex.Contrasting effects of Na+, K+-ATPase activation on seizure activity in acute versus chronic models.Acute creatine administration improves mitochondrial membrane potential and protects against pentylenetetrazol-induced seizures.Methylene blue prevents methylmalonate-induced seizures and oxidative damage in rat striatum.Guanosine Protects Against Traumatic Brain Injury-Induced Functional Impairments and Neuronal Loss by Modulating Excitotoxicity, Mitochondrial Dysfunction, and Inflammation.Creatine increases hippocampal Na(+),K(+)-ATPase activity via NMDA-calcineurin pathway.Time-dependent effects of treadmill exercise on aversive memory and cyclooxygenase pathway function.Long-term decrease in Na+,K+-ATPase activity after pilocarpine-induced status epilepticus is associated with nitration of its alpha subunit.Spermidine decreases Na⁺,K⁺-ATPase activity through NMDA receptor and protein kinase G activation in the hippocampus of rats.L-NAME prevents GM1 ganglioside-induced vasodilation in the rat brain.The involvement of Na+, K+-ATPase activity and free radical generation in the susceptibility to pentylenetetrazol-induced seizures after experimental traumatic brain injury.Exercise pre-conditioning reduces brain inflammation and protects against toxicity induced by traumatic brain injury: behavioral and neurochemical approach.
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Mauro Schneider Oliveira
@ast
Mauro Schneider Oliveira
@en
Mauro Schneider Oliveira
@es
Mauro Schneider Oliveira
@nl
Mauro Schneider Oliveira
@sl
type
label
Mauro Schneider Oliveira
@ast
Mauro Schneider Oliveira
@en
Mauro Schneider Oliveira
@es
Mauro Schneider Oliveira
@nl
Mauro Schneider Oliveira
@sl
prefLabel
Mauro Schneider Oliveira
@ast
Mauro Schneider Oliveira
@en
Mauro Schneider Oliveira
@es
Mauro Schneider Oliveira
@nl
Mauro Schneider Oliveira
@sl
P1053
G-3437-2012
P106
P21
P31
P3829
P496
0000-0002-5381-1208