Brain catecholamine metabolism in catechol-O-methyltransferase (COMT)-deficient mice. - Wikidata - awgldk - TriplyDB

Brain catecholamine metabolism in catechol-O-methyltransferase (COMT)-deficient mice.

Brain catecholamine metabolism in catechol-O-methyltransferase (COMT)-deficient mice.

http://www.wikidata.org/entity/Q43889421

about

Why does the giant panda eat bamboo? A comparative analysis of appetite-reward-related genes among mammals Computational systems analysis of dopamine metabolism A catechol-O-methyltransferase that is essential for auditory function in mice and humans.Functional analysis of genetic variation in catechol-O-methyltransferase (COMT): effects on mRNA, protein, and enzyme activity in postmortem human brain Catechol-O-methyltransferase polymorphisms and some implications for cognitive therapeutics Norepinephrine and stimulant addiction Reduced natriuretic response to acute sodium loading in COMT gene deleted mice Diversity of Dopaminergic Neural Circuits in Response to Drug Exposure Improvement of prepulse inhibition and executive function by the COMT inhibitor tolcapone depends on COMT Val158Met polymorphism A transposon in Comt generates mRNA variants and causes widespread expression and behavioral differences among mice Genetic variation in the human brain dopamine system influences motor learning and its modulation by L-Dopa Importance of membrane-bound catechol-O-methyltransferase in L-DOPA metabolism: a pharmacokinetic study in two types of Comt gene modified mice Orientation and cellular distribution of membrane-bound catechol-O-methyltransferase in cortical neurons: implications for drug development Schizophrenia genes, gene expression, and neuropathology: on the matter of their convergence Animal models of gene-environment interaction in schizophrenia: A dimensional perspective.Generation and characterization of humanized mice carrying COMT158 Met/Val alleles.The impact of adult vitamin D deficiency on behaviour and brain function in male Sprague-Dawley rats Genetics of emotion.The 22q11.2 microdeletion: fifteen years of insights into the genetic and neural complexity of psychiatric disorders.Animal models of gene-environment interactions in schizophrenia.22q11.2 microdeletions: linking DNA structural variation to brain dysfunction and schizophrenia.Mesocorticolimbic monoamine correlates of methamphetamine sensitization and motivation.Neurobehavioral evidence for changes in dopamine system activity during adolescence.Estrogen shapes dopamine-dependent cognitive processes: implications for women's health DAT1 and COMT effects on delay discounting and trait impulsivity in male adolescents with attention deficit/hyperactivity disorder and healthy controls.Working memory contributions to reinforcement learning impairments in schizophrenia Haplotypic association spanning the 22q11.21 genes COMT and ARVCF with schizophrenia.COMT polymorphisms and anxiety-related personality traits.Sexually dimorphic effects of catechol-O-methyltransferase (COMT) inhibition on dopamine metabolism in multiple brain regions Dopaminergic genes predict individual differences in susceptibility to confirmation bias.Catechol O-methyltransferase val158-met genotype and individual variation in the brain response to amphetamine.A neurogenetics approach to defining differential susceptibility to institutional care The catechol-O-methyltransferase polymorphism: relations to the tonic-phasic dopamine hypothesis and neuropsychiatric phenotypes.COMT polymorphism modulates the resting-state EEG alpha oscillatory response to acute nicotine in male non-smokers How much of reinforcement learning is working memory, not reinforcement learning? A behavioral, computational, and neurogenetic analysis.Pharmacogenetic tools for the development of target-oriented cognitive-enhancing drugs.Variability in Dopamine Genes Dissociates Model-Based and Model-Free Reinforcement Learning Marine Ο-3 polyunsaturated fatty acids induce sex-specific changes in reinforcer-controlled behaviour and neurotransmitter metabolism in a spontaneously hypertensive rat model of ADHD.Is COMT a susceptibility gene for schizophrenia?Observed positive parenting behaviors and youth genotype: evidence for gene-environment correlations and moderation by parent personality traits.

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P2860

Brain catecholamine metabolism in catechol-O-methyltransferase (COMT)-deficient mice.

http://www.wikidata.org/entity/Q43889421

description

2002 nî lūn-bûn

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2002年学术文章

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2002年学术文章

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2002年學術文章

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2002年學術文章

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name

Brain catecholamine metabolism in catechol-O-methyltransferase (COMT)-deficient mice.

@en

Brain catecholamine metabolism in catechol-O-methyltransferase

@nl

type

label

Brain catecholamine metabolism in catechol-O-methyltransferase (COMT)-deficient mice.

@en

Brain catecholamine metabolism in catechol-O-methyltransferase

@nl

prefLabel

Brain catecholamine metabolism in catechol-O-methyltransferase (COMT)-deficient mice.

@en

Brain catecholamine metabolism in catechol-O-methyltransferase

@nl

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European Journal of Neuroscience

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Brain catecholamine metabolism in catechol-O-methyltransferase (COMT)-deficient mice.

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Atso Raasmaja

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Joseph A Gogos

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Juha Hyttinen

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Maria Karayiorgou

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Marko Huotari

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Markus Forsberg

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Olli Koponen

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Pekka T Männistö

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P2860

Aggressive behavior and altered amounts of brain serotonin and norepinephrine in mice lacking MAOA

Catechol-O-methyltransferase-deficient mice exhibit sexually dimorphic changes in catecholamine levels and behavior.

Cleavage of structural proteins during the assembly of the head of bacteriophage T4

Catechol-O-methyltransferase (COMT) in rat brain: immunoelectron microscopic study with an antiserum against rat recombinant COMT protein

Effect of COMT Val108/158 Met genotype on frontal lobe function and risk for schizophrenia

Methylation pharmacogenetics: catechol O-methyltransferase, thiopurine methyltransferase, and histamine N-methyltransferase.

Schizophrenia susceptibility associated with interstitial deletions of chromosome 22q11.

Reduction of background problems in nonradioactive northern and Southern blot analyses enables higher sensitivity than 32P-based hybridizations.

Association of codon 108/158 catechol-O-methyltransferase gene polymorphism with the psychiatric manifestations of velo-cardio-facial syndrome.

Genotype determining low catechol-O-methyltransferase activity as a risk factor for obsessive-compulsive disorder.

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