I(h) channels contribute to the different functional properties of identified dopaminergic subpopulations in the midbrain.
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The selective antagonist EPPTB reveals TAAR1-mediated regulatory mechanisms in dopaminergic neurons of the mesolimbic systemAutism-associated SHANK3 haploinsufficiency causes Ih channelopathy in human neuronsVirogenetic and optogenetic mechanisms to define potential therapeutic targets in psychiatric disordersCocaine increases dopaminergic neuron and motor activity via midbrain α1 adrenergic signaling.Dopamine neurons share common response function for reward prediction error.Ethanol action on dopaminergic neurons in the ventral tegmental area: interaction with intrinsic ion channels and neurotransmitter inputs.Converging roles of ion channels, calcium, metabolic stress, and activity pattern of Substantia nigra dopaminergic neurons in health and Parkinson's diseaseGene expression in human hippocampus from cocaine abusers identifies genes which regulate extracellular matrix remodelingCorticotropin-releasing factor binding protein within the ventral tegmental area is expressed in a subset of dopaminergic neuronsPresynaptic GABAA receptors facilitate GABAergic transmission to dopaminergic neurons in the ventral tegmental area of young ratsMolecular organization and timing of Wnt1 expression define cohorts of midbrain dopamine neuron progenitors in vivoDopaminergic Neurons Exhibit an Age-Dependent Decline in Electrophysiological Parameters in the MitoPark Mouse Model of Parkinson's Disease.Perinatal Exposure to Neuregulin-1 Results in Disinhibition of Adult Midbrain Dopaminergic Neurons: Implication in Schizophrenia ModelingCocaine sensitization increases I h current channel subunit 2 (HCN₂) protein expression in structures of the mesocorticolimbic system.Mesolimbic dopamine neurons in the brain reward circuit mediate susceptibility to social defeat and antidepressant actionRecombinase-driver rat lines: tools, techniques, and optogenetic application to dopamine-mediated reinforcement.Ionic currents influencing spontaneous firing and pacemaker frequency in dopamine neurons of the ventrolateral periaqueductal gray and dorsal raphe nucleus (vlPAG/DRN): A voltage-clamp and computational modelling study.Cell type-specific gene expression of midbrain dopaminergic neurons reveals molecules involved in their vulnerability and protectionCellular mechanisms underlying burst firing in substantia nigra dopamine neurons.Dynamic, nonlinear feedback regulation of slow pacemaking by A-type potassium current in ventral tegmental area neurons.Alpha-2 noradrenergic receptor activation inhibits the hyperpolarization-activated cation current (Ih) in neurons of the ventral tegmental area.Functional integration of grafted neural stem cell-derived dopaminergic neurons monitored by optogenetics in an in vitro Parkinson modelAging decreases L-type calcium channel currents and pacemaker firing fidelity in substantia nigra dopamine neurons.Phasic dopamine release in appetitive behaviors and drug addiction.Pacemaking in dopaminergic ventral tegmental area neurons: depolarizing drive from background and voltage-dependent sodium conductances.Cocaine inhibition of nicotinic acetylcholine receptors influences dopamine release.Differentiation of human ES and Parkinson's disease iPS cells into ventral midbrain dopaminergic neurons requires a high activity form of SHH, FGF8a and specific regionalization by retinoic acid.Dynamic regulation of midbrain dopamine neuron activity: intrinsic, synaptic, and plasticity mechanismsIh current is necessary to maintain normal dopamine fluctuations and sleep consolidation in Drosophila.Loss of mitochondrial fission depletes axonal mitochondria in midbrain dopamine neurons.Cell type analysis of functional fetal dopamine cell suspension transplants in the striatum and substantia nigra of patients with Parkinson's disease.Kappa opioids selectively control dopaminergic neurons projecting to the prefrontal cortexFunctional upregulation of Ca(2+)-activated K(+) channels in the development of substantia nigra dopamine neurons.The h-current in the substantia Nigra pars compacta neurons: a re-examinationHCN Channel Targets for Novel Antidepressant Treatment.Non-linear developmental trajectory of electrical phenotype in rat substantia nigra pars compacta dopaminergic neurons.Hyperpolarization-activated cation current (Ih) is an ethanol target in midbrain dopamine neurons of mice.Operant self-stimulation of dopamine neurons in the substantia nigra.In vivo ethanol experience increases D(2) autoinhibition in the ventral tegmental area.Functional reduction of SK3-mediated currents precedes AMPA-receptor-mediated excitotoxicity in dopaminergic neurons.
P2860
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P2860
I(h) channels contribute to the different functional properties of identified dopaminergic subpopulations in the midbrain.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
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2002年学术文章
@zh
2002年学术文章
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@zh-hans
2002年学术文章
@zh-my
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name
I(h) channels contribute to th ...... ubpopulations in the midbrain.
@en
type
label
I(h) channels contribute to th ...... ubpopulations in the midbrain.
@en
prefLabel
I(h) channels contribute to th ...... ubpopulations in the midbrain.
@en
P50
P1476
I(h) channels contribute to th ...... subpopulations in the midbrain
@en
P2093
Henrike Neuhoff
P304
P356
10.1523/JNEUROSCI.22-04-01290.2002
P407
P577
2002-02-01T00:00:00Z