Maternal diabetes: an independent risk factor for major cardiovascular malformations with increased mortality of affected infants.
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VACTERL/caudal regression/Currarino syndrome-like malformations in mice with mutation in the proprotein convertase Pcsk5Diabetes and congenital heart defects: a systematic review, meta-analysis, and modeling projectNovel Mode of Defective Neural Tube Closure in the Non-Obese Diabetic (NOD) Mouse StrainProteomic-based detection of a protein cluster dysregulated during cardiovascular development identifies biomarkers of congenital heart defectsAltered gene expression in rat cranial neural crest cells exposed to a teratogenic glucose concentration in vitro: paradoxical downregulation of antioxidative defense genesMaternal diabetes alters transcriptional programs in the developing embryo.Cardiac malformations in fetuses of gestational and pre gestational diabetic mothers.Oxidative stress during diabetic pregnancy disrupts cardiac neural crest migration and causes outflow tract defects.Late-gestation ventricular myocardial reduction in fetuses of hyperglycemic CD1 mice is associated with increased apoptosis.Primary prevention of neural-tube defects and some other congenital abnormalities by folic acid and multivitamins: history, missed opportunity and tasks.Increased DNA methyltransferase 3b (Dnmt3b)-mediated CpG island methylation stimulated by oxidative stress inhibits expression of a gene required for neural tube and neural crest development in diabetic pregnancy.Sirenomelia (symelia apus) with Potter's syndrome in connection with gestational diabetes mellitus: a case report and literature reviewDiabetic embryopathy: a role for the epigenome?Role of HIF-1α in maternal hyperglycemia-induced embryonic vasculopathyCellular Stress, Excessive Apoptosis, and the Effect of Metformin in a Mouse Model of Type 2 Diabetic EmbryopathyASK1 mediates the teratogenicity of diabetes in the developing heart by inducing ER stress and inhibiting critical factors essential for cardiac development.Superoxide Dismutase 1 In Vivo Ameliorates Maternal Diabetes Mellitus-Induced Apoptosis and Heart Defects Through Restoration of Impaired Wnt Signaling.Advances in understanding the molecular causes of diabetes-induced birth defects.Elevated glucose inhibits VEGF-A-mediated endocardial cushion formation: modulation by PECAM-1 and MMP-2.Descriptive epidemiology of non-syndromic complete atrioventricular canal defects.High glucose environment inhibits cranial neural crest survival by activating excessive autophagy in the chick embryoHyperglycemia slows embryonic growth and suppresses cell cycle via cyclin D1 and p21.Frontiers in research on maternal diabetes-induced neural tube defects: Past, present and futureIs Duane retraction syndrome part of the VACTERL association?Diabetic embryopathy: a developmental perspective from fertilization to adulthood.Mitochondrial Factors and VACTERL Association-Related Congenital MalformationsNew concepts in diabetic embryopathySpecific local cardiovascular changes of Nepsilon-(carboxymethyl)lysine, vascular endothelial growth factor, and Smad2 in the developing embryos coincide with maternal diabetes-induced congenital heart defects.Adults with genetic syndromes and cardiovascular abnormalities: clinical history and management.Understanding diabetic teratogenesis: where are we now and where are we going?Diabetes and apoptosis: neural crest cells and neural tube.Troponin T and pro-B-type natriuretic Peptide in fetuses of type 1 diabetic mothers.Responses of the embryonic epigenome to maternal diabetesDiabetic uterus environment may play a key role in alterations of DNA methylation of several imprinted genes at mid-gestation in mice.Proportion of selected congenital heart defects attributable to recognized risk factors.Type 2 diabetes mellitus induces congenital heart defects in murine embryos by increasing oxidative stress, endoplasmic reticulum stress, and apoptosis.The Dorsal Mesenchymal Protrusion and the Pathogenesis of Atrioventricular Septal Defects.microRNA expression profiling and functional annotation analysis of their targets modulated by oxidative stress during embryonic heart development in diabetic mice.Congenital heart disease in pregnancies complicated by maternal diabetes mellitus. An international clinical collaboration, literature review, and meta-analysis.Preconception care for women with diabetes and prevention of major congenital malformations.
P2860
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P2860
Maternal diabetes: an independent risk factor for major cardiovascular malformations with increased mortality of affected infants.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
Maternal diabetes: an independ ...... mortality of affected infants.
@en
Maternal diabetes: an independ ...... mortality of affected infants.
@nl
type
label
Maternal diabetes: an independ ...... mortality of affected infants.
@en
Maternal diabetes: an independ ...... mortality of affected infants.
@nl
prefLabel
Maternal diabetes: an independ ...... mortality of affected infants.
@en
Maternal diabetes: an independ ...... mortality of affected infants.
@nl
P2093
P2860
P921
P356
P1476
Maternal diabetes: an independ ...... mortality of affected infants.
@en
P2093
P2860
P304
P356
10.1002/TERA.1051
P577
2001-08-01T00:00:00Z