Apoptosis in the left ventricle of chronic volume overload causes endocardial endothelial dysfunction in rats.
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Arrhythmia and neuronal/endothelial myocyte uncoupling in hyperhomocysteinemiaEpigenetic silencing of TIMP4 in heart failure.H2S ameliorates oxidative and proteolytic stresses and protects the heart against adverse remodeling in chronic heart failureMatrix metalloproteinase-2 in the development of diabetic retinopathy and mitochondrial dysfunction.Metalloproteinase in myocardial adaptation and maladaptation.Synergism between arrhythmia and hyperhomo-cysteinemia in structural heart disease.A metabolomic study of rats with doxorubicin-induced cardiomyopathy and Shengmai injection treatment.Changes in plasma profiles of matrix metalloproteinases (MMPs) and tissue inhibitors of MMPs in stress-induced cardiomyopathyCell Life versus cell longevity: the mysteries surrounding the NAD+ precursor nicotinamide.Triple play: promoting neurovascular longevity with nicotinamide, WNT, and erythropoietin in diabetes mellitus.Physiologic and molecular characterization of a murine model of right ventricular volume overload.Nitrotyrosinylation, remodeling and endothelial-myocyte uncoupling in iNOS, cystathionine beta synthase (CBS) knockouts and iNOS/CBS double knockout mice.Oxidative stress and the development of diabetic retinopathy: contributory role of matrix metalloproteinase-2.The vitamin nicotinamide: translating nutrition into clinical care.Cardiac myocyte-fibroblast interactions and the coronary vasculature.Etiology-specific assessment of predictors of long-term survival in chronic systolic heart failureNAD+ : A big player in cardiac and skeletal muscle remodeling and aging.An elderly patient with severe aortic stenosis and myocardial infarction with a huge mobile thrombus as complication in the left ventricle.Defects in ryanodine receptor function are associated with systolic dysfunction in rats subjected to volume overload.Cyclical mechanical stretch-induced apoptosis in myocytes from young rats but necrosis in myocytes from old rats.Protease-activated receptor and endothelial-myocyte uncoupling in chronic heart failure.Acute aortocaval fistula: role of low perfusion pressure and subendocardial remodeling on left ventricular function.Pioglitazone prevents cardiac remodeling in high-fat, high-calorie-induced Type 2 diabetes mellitus.Emerging Potential Benefits of Modulating NAD+ Metabolism in Cardiovascular Disease.
P2860
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P2860
Apoptosis in the left ventricle of chronic volume overload causes endocardial endothelial dysfunction in rats.
description
2002 nî lūn-bûn
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2002年の論文
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年學術文章
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name
Apoptosis in the left ventricl ...... dothelial dysfunction in rats.
@en
Apoptosis in the left ventricl ...... dothelial dysfunction in rats.
@nl
type
label
Apoptosis in the left ventricl ...... dothelial dysfunction in rats.
@en
Apoptosis in the left ventricl ...... dothelial dysfunction in rats.
@nl
prefLabel
Apoptosis in the left ventricl ...... dothelial dysfunction in rats.
@en
Apoptosis in the left ventricl ...... dothelial dysfunction in rats.
@nl
P2093
P2860
P1476
Apoptosis in the left ventricl ...... dothelial dysfunction in rats.
@en
P2093
Derrick Chandler
Giorgio M Aru
Harpreet S Sood
Jeffrey R Henegar
Matthew J Hunt
Michael J Cox
Suresh C Tyagi
P2860
P304
P356
10.1152/AJPHEART.00483.2001
P577
2002-04-01T00:00:00Z