Critical role of cAMP-response element-binding protein for angiotensin II-induced hypertrophy of vascular smooth muscle cells. - Wikidata - awgldk - TriplyDB

Critical role of cAMP-response element-binding protein for angiotensin II-induced hypertrophy of vascular smooth muscle cells.

Critical role of cAMP-response element-binding protein for angiotensin II-induced hypertrophy of vascular smooth muscle cells.

http://www.wikidata.org/entity/Q43925886

about

WISP1, a pro-mitogenic, pro-survival factor, mediates tumor necrosis factor-alpha (TNF-alpha)-stimulated cardiac fibroblast proliferation but inhibits TNF-alpha-induced cardiomyocyte death Identification of a cAMP-response element in the regulator of G-protein signaling-2 (RGS2) promoter as a key cis-regulatory element for RGS2 transcriptional regulation by angiotensin II in cultured vascular smooth muscles Involvement of cAMP-response element binding protein-1 in arachidonic acid-induced vascular smooth muscle cell motility Rare Copy Number Variants Identified Suggest the Regulating Pathways in Hypertension-Related Left Ventricular Hypertrophy Caveolin 1 is critical for abdominal aortic aneurysm formation induced by angiotensin II and inhibition of lysyl oxidase.Ribonucleic acid interference knockdown of interleukin 6 attenuates cold-induced hypertension T-type Ca²+ signalling regulates aldosterone-induced CREB activation and cell death through PP2A activation in neonatal cardiomyocytes Akt increases sox2 expression in adult hippocampal neural progenitor cells, but increased sox2 does not promote proliferation.The α and Δ isoforms of CREB1 are required to maintain normal pulmonary vascular resistance.Tissue kallikrein protects against pressure overload-induced cardiac hypertrophy through kinin B2 receptor and glycogen synthase kinase-3beta activation Angiotensin II regulates brain (pro)renin receptor expression through activation of cAMP response element-binding protein.Classic interleukin-6 receptor signaling and interleukin-6 trans-signaling differentially control angiotensin II-dependent hypertension, cardiac signal transducer and activator of transcription-3 activation, and vascular hypertrophy in vivo.Small RNA sequencing reveals microRNAs that modulate angiotensin II effects in vascular smooth muscle cells Activation of Nrf2 contributes to the protective effect of Exendin-4 against angiotensin II-induced vascular smooth muscle cell senescence.Antenatal hypoxia induces epigenetic repression of glucocorticoid receptor and promotes ischemic-sensitive phenotype in the developing heart.Alpha-CaMKII plays a critical role in determining the aggressive behavior of human osteosarcoma.Role of interferon regulatory factor 4 in the regulation of pathological cardiac hypertrophy NADPH oxidases and angiotensin II receptor signaling The cyclic AMP response element-binding protein (CREB) mediates smooth muscle cell proliferation in response to angiotensin II.Overexpression of CREB protein protects from tunicamycin-induced apoptosis in various rat cell types.STIM-1 and ORAI-1 channel mediate angiotensin-II-induced expression of Egr-1 in vascular smooth muscle cells.NF-κB and CREB are required for angiotensin II type 1 receptor upregulation in neurons.Essential role of ATF-1 in induction of NOX1, a catalytic subunit of NADPH oxidase: involvement of mitochondrial respiratory chain Platelet-derived growth factor BB induces nuclear export and proteasomal degradation of CREB via phosphatidylinositol 3-kinase/Akt signaling in pulmonary artery smooth muscle cells.Transcription factor cAMP response element modulator (Crem) restrains Pdgf-dependent proliferation of vascular smooth muscle cells in mice.Angiotensin II stimulates melanogenesis via the protein kinase C pathway.Mechanisms of angiotensin II-induced expression of B2 kinin receptors.Extracellular receptor kinase and cAMP response element binding protein activation in the neonatal rat heart after perinatal cocaine exposure.Resveratrol attenuates angiotensin II-induced interleukin-6 expression and perivascular fibrosis.

P2860

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P2860

Critical role of cAMP-response element-binding protein for angiotensin II-induced hypertrophy of vascular smooth muscle cells.

http://www.wikidata.org/entity/Q43925886

description

2002 nî lūn-bûn

@nan

2002年の論文

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2002年学术文章

@wuu

2002年学术文章

@zh

2002年学术文章

@zh-cn

2002年学术文章

@zh-hans

2002年学术文章

@zh-my

2002年学术文章

@zh-sg

2002年學術文章

@yue

2002年學術文章

@zh-hant

name

Critical role of cAMP-response ...... vascular smooth muscle cells.

@en

Critical role of cAMP-response ...... vascular smooth muscle cells.

@nl

type

label

Critical role of cAMP-response ...... vascular smooth muscle cells.

@en

Critical role of cAMP-response ...... vascular smooth muscle cells.

@nl

prefLabel

Critical role of cAMP-response ...... vascular smooth muscle cells.

@en

Critical role of cAMP-response ...... vascular smooth muscle cells.

@nl

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P356

P1433

Journal of Biological Chemistry

P1476

Critical role of cAMP-response ...... vascular smooth muscle cells.

@en

P2093

Akira Takeshita

http://www.w3.org/2001/XMLSchema#string

Kotaro Takeda

http://www.w3.org/2001/XMLSchema#string

Naoko Iino

http://www.w3.org/2001/XMLSchema#string

Tomotake Tokuno

http://www.w3.org/2001/XMLSchema#string

Toshihiro Ichiki

http://www.w3.org/2001/XMLSchema#string

Yuko Funakoshi

http://www.w3.org/2001/XMLSchema#string

P2860

Mitogen- and stress-activated protein kinase-1 (MSK1) is directly activated by MAPK and SAPK2/p38, and may mediate activation of CREB

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

A cluster of phosphorylation sites on the cyclic AMP-regulated nuclear factor CREB predicted by its sequence

Coupling of the RAS-MAPK pathway to gene activation by RSK2, a growth factor-regulated CREB kinase

CREB is a regulatory target for the protein kinase Akt/PKB

Rsk-2 activity is necessary for epidermal growth factor-induced phosphorylation of CREB protein and transcription of c-fos gene

Cyclic AMP stimulates somatostatin gene transcription by phosphorylation of CREB at serine 133

Angiotensin II induces hypertrophy, not hyperplasia, of cultured rat aortic smooth muscle cells.

Multiple protein kinase A-regulated events are required for transcriptional induction by cAMP.

Distinct effects of cAMP and mitogenic signals on CREB-binding protein recruitment impart specificity to target gene activation via CREB.

P304

18710-18717

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scholarly article

P356

10.1074/JBC.M110430200

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P433

21

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P478

277

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P577

2002-03-20T00:00:00Z

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P698

11907026

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