Overexpression of EIF5A2 promotes colorectal carcinoma cell aggressiveness by upregulating MTA1 through C-myc to induce epithelial-mesenchymaltransition. - Wikidata - awgldk - TriplyDB

Overexpression of EIF5A2 promotes colorectal carcinoma cell aggressiveness by upregulating MTA1 through C-myc to induce epithelial-mesenchymaltransition.

Overexpression of EIF5A2 promotes colorectal carcinoma cell aggressiveness by upregulating MTA1 through C-myc to induce epithelial-mesenchymaltransition.

http://www.wikidata.org/entity/Q43931495

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Systemic delivery of microRNA-101 potently inhibits hepatocellular carcinoma in vivo by repressing multiple targets Structure, expression and functions of MTA genes A novel mouse model for inhibition of DOHH-mediated hypusine modification reveals a crucial function in embryonic development, proliferation and oncogenic transformation The enforced expression of c-Myc in pig fibroblasts triggers mesenchymal-epithelial transition (MET) via F-actin reorganization and RhoA/Rock pathway inactivation MTA1 promotes epithelial to mesenchymal transition and metastasis in non-small-cell lung cancer.Increased intratumoral neutrophil in colorectal carcinomas correlates closely with malignant phenotype and predicts patients' adverse prognosis.Epstein-Barr virus-encoded latent membrane protein 2A promotes the epithelial-mesenchymal transition in nasopharyngeal carcinoma via metastatic tumor antigen 1 and mechanistic target of rapamycin signaling induction.Ablation of EIF5A2 induces tumor vasculature remodeling and improves tumor response to chemotherapy via regulation of matrix metalloproteinase 2 expression Cisplatin sensitivity is enhanced in non-small cell lung cancer cells by regulating epithelial-mesenchymal transition through inhibition of eukaryotic translation initiation factor 5A2.A hypusine-eIF5A-PEAK1 switch regulates the pathogenesis of pancreatic cancer Establishment of a TGFβ-induced post-transcriptional EMT gene signature.N1-guanyl-1,7-diaminoheptane sensitizes bladder cancer cells to doxorubicin by preventing epithelial-mesenchymal transition through inhibition of eukaryotic translation initiation factor 5A2 activation.Overexpression of eukaryotic translation initiation factor 5A2 (EIF5A2) correlates with cell aggressiveness and poor survival in gastric cancer Ano1/TMEM16A Overexpression Is Associated with Good Prognosis in PR-Positive or HER2-Negative Breast Cancer Patients following Tamoxifen Treatment.A Screen for Epigenetically Silenced microRNA Genes in Gastrointestinal Stromal Tumors.Biological Relevance and Therapeutic Potential of the Hypusine Modification System.MiR-30b suppresses tumor migration and invasion by targeting EIF5A2 in gastric cancer Expression of EIF5A2 associates with poor survival of nasopharyngeal carcinoma patients treated with induction chemotherapy.Stemness and chemotherapeutic drug resistance induced by EIF5A2 overexpression in esophageal squamous cell carcinoma Connecting the dots: chromatin and alternative splicing in EMT.The translation factor eIF5A and human cancer.Increased expression of Solute carrier family 12 member 5 via gene amplification contributes to tumour progression and metastasis and associates with poor survival in colorectal cancer.MiRNA-203 suppresses cell proliferation, migration and invasion in colorectal cancer via targeting of EIF5A2.The Dysregulation of Polyamine Metabolism in Colorectal Cancer Is Associated with Overexpression of c-Myc and C/EBPβ rather than Enterotoxigenic Bacteroides fragilis Infection MicroRNA-543 suppresses colorectal cancer growth and metastasis by targeting KRAS, MTA1 and HMGA2.Eukaryotic translation initiation factor 5A2 regulates the migration and invasion of hepatocellular carcinoma cells via pathways involving reactive oxygen species.Decreased expression of PTPN12 correlates with tumor recurrence and poor survival of patients with hepatocellular carcinoma.Identification and validation of AIB1 and EIF5A2 for noninvasive detection of bladder cancer in urine samples Role of Eukaryotic Initiation Factors during Cellular Stress and Cancer Progression EIF5A2 predicts outcome in localised invasive bladder cancer and promotes bladder cancer cell aggressiveness in vitro and in vivo.MicroRNA-9 regulates non-small cell lung cancer cell invasion and migration by targeting eukaryotic translation initiation factor 5A2.Biomarkers for predicting future metastasis of human gastrointestinal tumors.Roles of eukaryotic initiation factor 5A2 in human cancer.MTA family of proteins in prostate cancer: biology, significance, and therapeutic opportunities.Molecular functions and significance of the MTA family in hormone-independent cancer.The hypusine cascade promotes cancer progression and metastasis through the regulation of RhoA in squamous cell carcinoma.EIF5A2 is a novel chemoresistance gene in breast cancer.Role of EIF5A2, a downstream target of Akt, in promoting melanoma cell invasion.Eukaryotic translation initiation factors and cancer.MTA1 modulated by miR-30e contributes to epithelial-to-mesenchymal transition in hepatocellular carcinoma through an ErbB2-dependent pathway.

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Overexpression of EIF5A2 promotes colorectal carcinoma cell aggressiveness by upregulating MTA1 through C-myc to induce epithelial-mesenchymaltransition.

http://www.wikidata.org/entity/Q43931495

description

2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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2011年學術文章

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name

Overexpression of EIF5A2 promo ...... thelial-mesenchymaltransition.

@en

Overexpression of EIF5A2 promo ...... thelial-mesenchymaltransition.

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type

label

Overexpression of EIF5A2 promo ...... thelial-mesenchymaltransition.

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Overexpression of EIF5A2 promo ...... thelial-mesenchymaltransition.

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prefLabel

Overexpression of EIF5A2 promo ...... thelial-mesenchymaltransition.

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Overexpression of EIF5A2 promo ...... thelial-mesenchymaltransition.

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Overexpression of EIF5A2 promo ...... ithelial-mesenchymaltransition

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Dan Xie

http://www.w3.org/2001/XMLSchema#string

Hsiang-Fu Kung

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Marie C Lin

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Mu-Yan Cai

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Shi-Juan Mai

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Sui-Sui Dong

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Wei Zhu

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Xiu-Wu Bian

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Yi-Xin Zeng

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Zhu-Ting Tong

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562-575

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10.1136/GUTJNL-2011-300207

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