RAGE mediates the inactivation of nAChRs in sympathetic neurons under high glucose conditions.

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Transcriptomic alterations in the heart of non-obese type 2 diabetic Goto-Kakizaki rats RAGE-dependent potentiation of TRPV1 currents in sensory neurons exposed to high glucose.Alarmin HMGB1 induces systemic and brain inflammatory exacerbation in post-stroke infection rat model.

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RAGE mediates the inactivation of nAChRs in sympathetic neurons under high glucose conditions.

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RAGE mediates the inactivation ...... under high glucose conditions.

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RAGE mediates the inactivation ...... under high glucose conditions.

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RAGE mediates the inactivation ...... under high glucose conditions.

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RAGE mediates the inactivation ...... under high glucose conditions.

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RAGE mediates the inactivation ...... under high glucose conditions.

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RAGE mediates the inactivation ...... under high glucose conditions.

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P1476

RAGE mediates the inactivation ...... under high glucose conditions.

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Andrew R Chandna

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Christine Chang

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Darrell D Mousseau

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Manoj Nair

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Paul R Pennington

http://www.w3.org/2001/XMLSchema#string

Verónica A Campanucci

http://www.w3.org/2001/XMLSchema#string

Yasuhiko Yamamoto

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P2860

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

Loss of pain perception in diabetes is dependent on a receptor of the immunoglobulin superfamily

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

HMG-1 as a late mediator of endotoxin lethality in mice

Development and prevention of advanced diabetic nephropathy in RAGE-overexpressing mice

A null mutation for the alpha3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde m

Megacystis, mydriasis, and ion channel defect in mice lacking the alpha3 neuronal nicotinic acetylcholine receptor

Development of selective axonopathy in adult sensory neurons isolated from diabetic rats: role of glucose-induced oxidative stress.

Protein glycation, diabetes, and aging.

Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion.

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341-351

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scholarly article

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10.1111/EJN.12795

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English

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2014-11-28T00:00:00Z

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P5875

268820588

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P698

25431195

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RAGE mediates the inactivation of nAChRs in sympathetic neurons under high glucose conditions.

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P2860

P2860

RAGE mediates the inactivation of nAChRs in sympathetic neurons under high glucose conditions.

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