Increased nonoxidative glycolysis despite continued fatty acid uptake during demand-induced myocardial ischemia.
about
Myocardial energy metabolism during ischemia and the mechanisms of metabolic therapies.Malonyl-CoA decarboxylase inhibition as a novel approach to treat ischemic heart disease.Impact of anaerobic glycolysis and oxidative substrate selection on contractile function and mechanical efficiency during moderate severity ischemiaTransmural distribution of metabolic abnormalities and glycolytic activity during dobutamine-induced demand ischemia.Effects of exercise training and diet on lipid kinetics during free fatty acid-induced insulin resistance in older obese humans with impaired glucose tolerance.Dipropionylcysteine ethyl ester compensates for loss of citric acid cycle intermediates during post ischemia reperfusion in the pig heartQuantitative assessment of anaplerosis from propionate in pig heart in vivo.Effect of hyperglycemia and fatty acid oxidation inhibition during aerobic conditions and demand-induced ischemia.beta-Hydroxybutyrate inhibits myocardial fatty acid oxidation in vivo independent of changes in malonyl-CoA content.Moderate severity heart failure does not involve a downregulation of myocardial fatty acid oxidation.Regulation of pyruvate dehydrogenase activity and citric acid cycle intermediates during high cardiac power generation.Mechanistic model of cardiac energy metabolism predicts localization of glycolysis to cytosolic subdomain during ischemia.Modulation of NFKB1/p50 by ROS leads to impaired ATP production during MI compared to cardiac hypertrophy.Regulation of cardiac malonyl-CoA content and fatty acid oxidation during increased cardiac power.Malonyl-CoA decarboxylase inhibition suppresses fatty acid oxidation and reduces lactate production during demand-induced ischemia.Differential effects of heptanoate and hexanoate on myocardial citric acid cycle intermediates following ischemia-reperfusion.Parallel activation of mitochondrial oxidative metabolism with increased cardiac energy expenditure is not dependent on fatty acid oxidation in pigs.Acute lipoprotein lipase deletion in adult mice leads to dyslipidemia and cardiac dysfunctionCircadian rhythms in myocardial metabolism and contractile function: influence of workload and oleate
P2860
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P2860
Increased nonoxidative glycolysis despite continued fatty acid uptake during demand-induced myocardial ischemia.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Increased nonoxidative glycoly ...... d-induced myocardial ischemia.
@en
Increased nonoxidative glycoly ...... d-induced myocardial ischemia.
@nl
type
label
Increased nonoxidative glycoly ...... d-induced myocardial ischemia.
@en
Increased nonoxidative glycoly ...... d-induced myocardial ischemia.
@nl
prefLabel
Increased nonoxidative glycoly ...... d-induced myocardial ischemia.
@en
Increased nonoxidative glycoly ...... d-induced myocardial ischemia.
@nl
P2093
P2860
P1476
Increased nonoxidative glycoly ...... d-induced myocardial ischemia.
@en
P2093
Hazel Huang
Margaret P Chandler
Tracy A McElfresh
William C Stanley
P2860
P304
P356
10.1152/AJPHEART.00976.2001
P577
2002-05-01T00:00:00Z