Two msbB genes encoding maximal acylation of lipid A are required for invasive Shigella flexneri to mediate inflammatory rupture and destruction of the intestinal epithelium.
about
Lipid A modification systems in gram-negative bacteriaShigella: a model of virulence regulation in vivo.Structure-guided enzymology of the lipid A acyltransferase LpxM reveals a dual activity mechanismGut microbiota and immune crosstalk in metabolic diseaseHigh yield production process for Shigella outer membrane particlesShield as signal: lipopolysaccharides and the evolution of immunity to gram-negative bacteria.Yeast functional genomic screens lead to identification of a role for a bacterial effector in innate immunity regulationVirulence, inflammatory potential, and adaptive immunity induced by Shigella flexneri msbB mutantsInvolvement of the Escherichia coli O157:H7(pO157) ecf operon and lipid A myristoyl transferase activity in bacterial survival in the bovine gastrointestinal tract and bacterial persistence in farm water troughs.Murein lipoprotein is a critical outer membrane component involved in Salmonella enterica serovar typhimurium systemic infectionTargeted complement inhibition by C3d recognition ameliorates tissue injury without apparent increase in susceptibility to infection.The Shigella flexneri effector OspG interferes with innate immune responses by targeting ubiquitin-conjugating enzymesModulation of endotoxicity of Shigella generalized modules for membrane antigens (GMMA) by genetic lipid A modifications: relative activation of TLR4 and TLR2 pathways in different mutantsSpecies-specific activation of TLR4 by hypoacylated endotoxins governed by residues 82 and 122 of MD-2Common changes in global gene expression induced by RNA polymerase inhibitors in Shigella flexneriLcrV delivered via type III secretion system of live attenuated Yersinia pseudotuberculosis enhances immunogenicity against pneumonic plague.Characterization of lipid A profiles from Shigella flexneri variant X lipopolysaccharide.Pleiotropic effects of the lpxM mutation in Yersinia pestis resulting in modification of the biosynthesis of major immunoreactive antigens.Characterization of WRSs2 and WRSs3, new second-generation virG(icsA)-based Shigella sonnei vaccine candidates with the potential for reduced reactogenicity.Discovery of new biosynthetic pathways: the lipid A story.Analysis of virulence and inflammatory potential of Shigella flexneri purine biosynthesis mutantsSecretory expression of biologically active human Herpes virus interleukin-10 analogues in Escherichia coli via a modified Sec-dependent transporter constructHuman colonic myocytes are involved in postischemic inflammation through ADAM17-dependent TNFalpha production.RNA-seq analysis of the influence of anaerobiosis and FNR on Shigella flexneri.An msbB homologue carried in plasmid pO157 encodes an acyltransferase involved in lipid A biosynthesis in Escherichia coli O157:H7.Bioimage analysis of Shigella infection reveals targeting of colonic cryptsVaccines for viral and bacterial pathogens causing acute gastroenteritis: Part II: Vaccines for Shigella, Salmonella, enterotoxigenic E. coli (ETEC) enterohemorragic E. coli (EHEC) and Campylobacter jejuni.Design of a covalently linked human interleukin-10 fusion protein and its secretory expression in Escherichia coli.A specific role for TLR1 in protective T(H)17 immunity during mucosal infection.Norharmane Matrix Enhances Detection of Endotoxin by MALDI-MS for Simultaneous Profiling of Pathogen, Host, and Vector Systems.Shigella spp. and enteroinvasive Escherichia coli pathogenicity factors.Salmonella enterica serovar Typhimurium expressing mutant lipid A with decreased endotoxicity causes maturation of murine dendritic cellsProtective and destructive immunity in the periodontium: Part 1--innate and humoral immunity and the periodontium.Sensing gram-negative bacterial lipopolysaccharides: a human disease determinant?Characterization of htrB and msbB mutants of the light organ symbiont Vibrio fischeri.New perspectives for a new century: implications of pathogen responses for the future of antimicrobial therapy.Shigella's ways of manipulating the host intestinal innate and adaptive immune system: a tool box for survival?Clinical trials of Shigella vaccines: two steps forward and one step back on a long, hard road.Secondary acylation of Klebsiella pneumoniae lipopolysaccharide contributes to sensitivity to antibacterial peptidesPagP activation in the outer membrane triggers R3 core oligosaccharide truncation in the cytoplasm of Escherichia coli O157:H7.
P2860
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P2860
Two msbB genes encoding maximal acylation of lipid A are required for invasive Shigella flexneri to mediate inflammatory rupture and destruction of the intestinal epithelium.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Two msbB genes encoding maxima ...... of the intestinal epithelium.
@en
Two msbB genes encoding maxima ...... of the intestinal epithelium.
@nl
type
label
Two msbB genes encoding maxima ...... of the intestinal epithelium.
@en
Two msbB genes encoding maxima ...... of the intestinal epithelium.
@nl
prefLabel
Two msbB genes encoding maxima ...... of the intestinal epithelium.
@en
Two msbB genes encoding maxima ...... of the intestinal epithelium.
@nl
P2093
P1476
Two msbB genes encoding maxima ...... of the intestinal epithelium.
@en
P2093
Anders Kussak
Claude Parsot
Duncan J Maskell
Hélène D'Hauteville
John Mathison
Nicole Wuscher
Philippe J Sansonetti
Richard J Ulevitch
Shahid Khan
P304
P356
10.4049/JIMMUNOL.168.10.5240
P407
P577
2002-05-01T00:00:00Z