Injury primes the innate immune system for enhanced Toll-like receptor reactivity.
about
Astrocyte elevated gene-1 (AEG-1) is induced by lipopolysaccharide as toll-like receptor 4 (TLR4) ligand and regulates TLR4 signallingTrauma equals danger--damage control by the immune systemIntestine immune homeostasis after alcohol and burn injuryDecreased pulmonary inflammation following ethanol and burn injury in mice deficient in TLR4 but not TLR2 signalingTranscriptome characterization by RNA-Seq reveals the involvement of the complement components in noise-traumatized rat cochleaeUp-regulation of cell surface Toll-like receptors on circulating gammadelta T-cells following burn injury.Pretreatment with stress cortisol enhances the human systemic inflammatory response to bacterial endotoxin.Positional differences in the wound transcriptome of skin and oral mucosa.Sepsis prediction in critically ill patients by platelet activation markers on ICU admission: a prospective pilot study.Wound trauma mediated inflammatory signaling attenuates a tissue regenerative response in MRL/MpJ mice.Trauma hemorrhagic shock-induced lung injury involves a gut-lymph-induced TLR4 pathway in miceRegulatory T cells suppress antigen-driven CD4 T cell reactivity following injury.Burn injury triggered dysfunction in dendritic cell response to TLR9 activation and resulted in skewed T cell functionsHigh-mobility group box-1 induces proinflammatory cytokines production of Kupffer cells through TLRs-dependent signaling pathway after burn injuryComparison of the effects of aging and IL-6 on the hepatic inflammatory response in two models of systemic injury: scald injury versus i.p. LPS administrationImmune system phenotyping of radiation and radiation combined injury in outbred miceToll-like receptor 3 is a potent negative regulator of axonal growth in mammals.MyD88 and Src are differentially regulated in Kupffer cells of males and proestrus females following hypoxia.Altered adherent leukocyte profile on biomaterials in Toll-like receptor 4 deficient mice.Injury-induced GR-1+ macrophage expansion and activation occurs independently of CD4 T-cell influence.Burn-induced alterations in toll-like receptor-mediated responses by bronchoalveolar lavage cells.Heme oxygenase-1 protects rat liver against warm ischemia/reperfusion injury via TLR2/TLR4-triggered signaling pathways.Sepsis: Something old, something new, and a systems view.Histone deacetylase inhibitor treatment attenuates MAP kinase pathway activation and pulmonary inflammation following hemorrhagic shock in a rodent model.Injury, sepsis, and the regulation of Toll-like receptor responses.Assessment of pharmacological activities of two medicinal plant of Bangladesh: Launaea sarmentosa and Aegialitis rotundifolia roxb in the management of pain, pyrexia and inflammation.Production of autoantibodies by murine B-1a cells stimulated with Helicobacter pylori urease through toll-like receptor 2 signaling.A protective role for inflammasome activation following injury.Resolution of LPS-induced airway inflammation and goblet cell hyperplasia is independent of IL-18.Pathogenesis of malaria and clinically similar conditions.Toll and IMD pathways synergistically activate an innate immune response in Drosophila melanogasterDynamics of short-term gene expression profiling in liver following thermal injury.Plasmodium yoelii blood-stage primes macrophage-mediated innate immune response through modulation of toll-like receptor signalling.Burn enhances toll-like receptor induced responses by circulating leukocytes.Psychoneuroimmunology: then and now.Inhibition of toll-like receptor and cytokine signaling--a unifying theme in ischemic tolerance.Activation of toll-like receptor 4 is necessary for trauma hemorrhagic shock-induced gut injury and polymorphonuclear neutrophil primingInnate immunity and organ transplantation: the potential role of toll-like receptors.Inflammation in atherosclerosis and other conditions: a response to danger.Burn plus lipopolysaccharide augments endoplasmic reticulum stress and NLRP3 inflammasome activation and reduces PGC-1α in liver
P2860
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P2860
Injury primes the innate immune system for enhanced Toll-like receptor reactivity.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Injury primes the innate immune system for enhanced Toll-like receptor reactivity.
@en
Injury primes the innate immune system for enhanced Toll-like receptor reactivity.
@nl
type
label
Injury primes the innate immune system for enhanced Toll-like receptor reactivity.
@en
Injury primes the innate immune system for enhanced Toll-like receptor reactivity.
@nl
prefLabel
Injury primes the innate immune system for enhanced Toll-like receptor reactivity.
@en
Injury primes the innate immune system for enhanced Toll-like receptor reactivity.
@nl
P2093
P1476
Injury primes the innate immune system for enhanced Toll-like receptor reactivity.
@en
P2093
Elizabeth J Purcell
James A Lederer
John A Mannick
Sara J Kriynovich
Thomas J Murphy
P304
P356
10.4049/JIMMUNOL.171.3.1473
P407
P577
2003-08-01T00:00:00Z