Host versus in vitro signals and intrastrain allelic differences in the expression of a Candida albicans virulence gene.
about
The diploid genome sequence of Candida albicansThe parasexual cycle in Candida albicans provides an alternative pathway to meiosis for the formation of recombinant strains.Profile of Candida albicans-secreted aspartic proteinase elicited during vaginal infection.Interaction of Blastomyces dermatitidis, Sporothrix schenckii, and Histoplasma capsulatum with Acanthamoeba castellanii.Rapid mechanisms for generating genome diversity: whole ploidy shifts, aneuploidy, and loss of heterozygosityHeterozygosity and functional allelic variation in the Candida albicans efflux pump genes CDR1 and CDR2.Mutations in alternative carbon utilization pathways in Candida albicans attenuate virulence and confer pleiotropic phenotypesEnvironmental sensing and signal transduction pathways regulating morphopathogenic determinants of Candida albicans.Candida albicans proteinases and host/pathogen interactions.Lipase 8 affects the pathogenesis of Candida albicans.Unraveling the secret lives of bacteria: use of in vivo expression technology and differential fluorescence induction promoter traps as tools for exploring niche-specific gene expressionThe Snf1-activating kinase Sak1 is a key regulator of metabolic adaptation and in vivo fitness of Candida albicans.Assessing the genome-wide effect of promoter region tandem repeat natural variation on gene expression.Inactivation of the phospholipase B gene PLB5 in wild-type Candida albicans reduces cell-associated phospholipase A2 activity and attenuates virulence.An MDR1 promoter allele with higher promoter activity is common in clinically isolated strains of Candida albicansDevelopment and characterization of an in vivo central venous catheter Candida albicans biofilm model.A single SNP, G929T (Gly310Val), determines the presence of a functional and a non-functional allele of HIS4 in Candida albicans SC5314: detection of the non-functional allele in laboratory strains.Differential susceptibility of mitogen-activated protein kinase pathway mutants to oxidative-mediated killing by phagocytes in the fungal pathogen Candida albicans.Demonstration of loss of heterozygosity by single-nucleotide polymorphism microarray analysis and alterations in strain morphology in Candida albicans strains during infection.Genome-wide single-nucleotide polymorphism map for Candida albicans.Tetracycline-inducible expression of individual secreted aspartic proteases in Candida albicans allows isoenzyme-specific inhibitor screening.Phenotypic Consequences of a Spontaneous Loss of Heterozygosity in a Common Laboratory Strain of Candida albicans.Loss of heterozygosity at an unlinked genomic locus is responsible for the phenotype of a Candida albicans sap4Δ sap5Δ sap6Δ mutant.Divergence of Stp1 and Stp2 transcription factors in Candida albicans places virulence factors required for proper nutrient acquisition under amino acid control.A family of oligopeptide transporters is required for growth of Candida albicans on proteins.
P2860
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P2860
Host versus in vitro signals and intrastrain allelic differences in the expression of a Candida albicans virulence gene.
description
2002 nî lūn-bûn
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2002年の論文
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name
Host versus in vitro signals a ...... ndida albicans virulence gene.
@en
Host versus in vitro signals a ...... ndida albicans virulence gene.
@nl
type
label
Host versus in vitro signals a ...... ndida albicans virulence gene.
@en
Host versus in vitro signals a ...... ndida albicans virulence gene.
@nl
prefLabel
Host versus in vitro signals a ...... ndida albicans virulence gene.
@en
Host versus in vitro signals a ...... ndida albicans virulence gene.
@nl
P2093
P2860
P1476
Host versus in vitro signals a ...... ndida albicans virulence gene.
@en
P2093
Herbert Hof
Joachim Morschhäuser
Marianne Kretschmar
Peter Staib
Thomas Nichterlein
P2860
P304
P356
10.1046/J.1365-2958.2002.02967.X
P407
P577
2002-06-01T00:00:00Z