Cardiac dysfunction in terms of left ventricular mechanical work and energetics in hypothyroid rats.
about
Decreased cardiac SERCA2 expression, SR Ca uptake, and contractile function in hypothyroidism are attenuated in SERCA2 overexpressing transgenic rats.Restoration of mechanical and energetic function in failing aortic-banded rat hearts by gene transfer of calcium cycling proteins.Mechanical work and energetic analysis of eccentric cardiac remodeling in a volume overload heart failure in rats.Regulation of myocardial SERCA2a expression in ventricular hypertrophy and heart failure.Rescue of Ca2+ overload-induced left ventricular dysfunction by targeted ablation of phospholamban.Effects of ischemic postconditioning on the hemodynamic parameters and heart nitric oxide levels of hypothyroid rats.A cardioprotective agent of a novel calpain inhibitor, SNJ-1945, exerts beta1 actions on left ventricular mechanical work and energetics.Cardioprotective effects of a novel calpain inhibitor SNJ-1945 for reperfusion injury after cardioplegic cardiac arrest.Left ventricular function of isoproterenol-induced hypertrophied rat hearts perfused with blood: mechanical work and energetics.Reversible effects of isoproterenol-induced hypertrophy on in situ left ventricular function in rat hearts.Left ventricular mechanoenergetics after hyperpolarized cardioplegic arrest by nicorandil and after depolarized cardioplegic arrest by KCl.Modest actomyosin energy conservation increases myocardial postischemic function.Calpain inhibitor-1 protects the rat heart from ischemia-reperfusion injury: analysis by mechanical work and energetics.Na+/Ca2+ exchange inhibition protects the rat heart from ischemia-reperfusion injury by blocking energy-wasting processes.Effects of formaldehyde on cardiovascular system in in situ rat hearts.Cardiac sarcoplasmic reticulum Ca2+-ATPase: heat production and phospholamban alterations promoted by cold exposure and thyroid hormone.Detrimental effects after dobutamine infusion on rat left ventricular function: mechanical work and energetics.Preservation of mechanical and energetic function after adenoviral gene transfer in normal rat hearts.Transcoronary gene transfer of SERCA2a increases coronary blood flow and decreases cardiomyocyte size in a type 2 diabetic rat model.Half-logistic time constant: a more reliable lusitropic index than monoexponential time constant regardless of temperature in canine left ventricle.Targeted gene transfer increases contractility and decreases oxygen cost of contractility in normal rat hearts.
P2860
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P2860
Cardiac dysfunction in terms of left ventricular mechanical work and energetics in hypothyroid rats.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Cardiac dysfunction in terms o ...... nergetics in hypothyroid rats.
@en
Cardiac dysfunction in terms o ...... nergetics in hypothyroid rats.
@nl
type
label
Cardiac dysfunction in terms o ...... nergetics in hypothyroid rats.
@en
Cardiac dysfunction in terms o ...... nergetics in hypothyroid rats.
@nl
prefLabel
Cardiac dysfunction in terms o ...... nergetics in hypothyroid rats.
@en
Cardiac dysfunction in terms o ...... nergetics in hypothyroid rats.
@nl
P2093
P2860
P1476
Cardiac dysfunction in terms o ...... nergetics in hypothyroid rats.
@en
P2093
Chikako Takenaka
Miyako Takaki
Shigeki Taniguchi
Susumu Sakata
Takehisa Abe
Tsuyoshi Tsuji
Yoshimi Ohga
P2860
P304
P356
10.1152/AJPHEART.00046.2002
P577
2002-08-01T00:00:00Z