Active calcium accumulation underlies severe weakness in a panel of mice with slow-channel syndrome.
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Zebrafish: A Model for the Study of Toxicants Affecting Muscle Development and FunctionUncoupling nicotine mediated motoneuron axonal pathfinding errors and muscle degeneration in zebrafish.Decoding pathogenesis of slow-channel congenital myasthenic syndromes using recombinant expression and mice models.Calpain activation impairs neuromuscular transmission in a mouse model of the slow-channel myasthenic syndromeMotoneuron axon pathfinding errors in zebrafish: differential effects related to concentration and timing of nicotine exposureAcetylcholinesterase deficiency contributes to neuromuscular junction dysfunction in type 1 diabetic neuropathy.Novel beta subunit mutation causes a slow-channel syndrome by enhancing activation and decreasing the rate of agonist dissociationTryptophan scanning of the acetylcholine receptor's betaM4 transmembrane domain: decoding allosteric linkage at the lipid-protein interface with ion-channel gating.Defective Acetylcholine Receptor Subunit Switch Precedes Atrophy of Slow-Twitch Skeletal Muscle Fibers Lacking ERK1/2 Kinases in Soleus Muscle.Acute severe animal model of anti-muscle-specific kinase myasthenia: combined postsynaptic and presynaptic changes.Transgenic mouse model reveals an unsuspected role of the acetylcholine receptor in statin-induced neuromuscular adverse drug reactions.Skeletal muscle IP3R1 receptors amplify physiological and pathological synaptic calcium signals.The human adult subtype ACh receptor channel has high Ca2+ permeability and predisposes to endplate Ca2+ overloading.A human congenital myasthenia-causing mutation (epsilon L78P) of the muscle nicotinic acetylcholine receptor with unusual single channel properties.Fluoxetine is neuroprotective in slow-channel congenital myasthenic syndrome.Selective DNAzyme-mediated cleavage of AChR mutant transcripts by targeting the mutation site or through mismatches in the binding arm.Acetylcholine receptor gating in a zebrafish model for slow-channel syndrome.Selective cleavage of AChR cRNAs harbouring mutations underlying the slow channel myasthenic syndrome by hammerhead ribozymes.Animal Models of the Neuromuscular Junction, Vitally Informative for Understanding Function and the Molecular Mechanisms of Congenital Myasthenic Syndromes.
P2860
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P2860
Active calcium accumulation underlies severe weakness in a panel of mice with slow-channel syndrome.
description
2002 nî lūn-bûn
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2002年の論文
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2002年学术文章
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2002年学术文章
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2002年学术文章
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name
Active calcium accumulation un ...... ce with slow-channel syndrome.
@en
Active calcium accumulation un ...... ce with slow-channel syndrome.
@nl
type
label
Active calcium accumulation un ...... ce with slow-channel syndrome.
@en
Active calcium accumulation un ...... ce with slow-channel syndrome.
@nl
prefLabel
Active calcium accumulation un ...... ce with slow-channel syndrome.
@en
Active calcium accumulation un ...... ce with slow-channel syndrome.
@nl
P2093
P1476
Active calcium accumulation un ...... ice with slow-channel syndrome
@en
P2093
Christopher M Gomez
Jason Groshong
Ricardo A Maselli
Robert L Wollmann
Roberto Zayas
Thierry Cens
P304
P356
10.1523/JNEUROSCI.22-15-06447.2002
P407
P577
2002-08-01T00:00:00Z