Sodium bicarbonate treatment and ubiquitin gene expression in acidotic human subjects with chronic renal failure.

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Correction of chronic metabolic acidosis for chronic kidney disease patients The temporal responses of protein synthesis, gene expression and cell signalling in human quadriceps muscle and patellar tendon to disuse Consequences and therapy of the metabolic acidosis of chronic kidney disease.

P2860

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Sodium bicarbonate treatment and ubiquitin gene expression in acidotic human subjects with chronic renal failure.

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http://www.wikidata.org/entity/Q44086550

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2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

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2002年學術文章

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name

Sodium bicarbonate treatment a ...... ts with chronic renal failure.

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Sodium bicarbonate treatment a ...... ts with chronic renal failure.

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type

Item

label

Sodium bicarbonate treatment a ...... ts with chronic renal failure.

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Sodium bicarbonate treatment a ...... ts with chronic renal failure.

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Sodium bicarbonate treatment a ...... ts with chronic renal failure.

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Sodium bicarbonate treatment a ...... ts with chronic renal failure.

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P1476

Sodium bicarbonate treatment a ...... cts with chronic renal failure

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K A Graham

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K Bartlett

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R G Roberts

http://www.w3.org/2001/XMLSchema#string

R Wilkinson

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T H J Goodship

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P2860

Isolation of biologically active ribonucleic acid from sources enriched in ribonuclease

Metabolic acidosis stimulates muscle protein degradation by activating the adenosine triphosphate-dependent pathway involving ubiquitin and proteasomes

Metabolic acidosis stimulates protein degradation in rat muscle by a glucocorticoid-dependent mechanism.

Ubiquitin and intracellular protein degradation.

Sepsis stimulates nonlysosomal, energy-dependent proteolysis and increases ubiquitin mRNA levels in rat skeletal muscle

Molecular cloning of cDNA for proteasomes (multicatalytic proteinase complexes) from rat liver: primary structure of the largest component (C2).

The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway

Muscle wasting in a rat model of long-lasting sepsis results from the activation of lysosomal, Ca2+ -activated, and ubiquitin-proteasome proteolytic pathways.

Muscle wasting in insulinopenic rats results from activation of the ATP-dependent, ubiquitin-proteasome proteolytic pathway by a mechanism including gene transcription.

Sepsis is associated with increased mRNAs of the ubiquitin-proteasome proteolytic pathway in human skeletal muscle

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488-492

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scholarly article

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10.1046/J.1365-2362.2002.01008.X

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Christopher Redfern

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2002-07-01T00:00:00Z

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P698

12153548

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P921

human subject research

Sodium bicarbonate treatment and ubiquitin gene expression in acidotic human subjects with chronic renal failure.

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P2860

P2860

Sodium bicarbonate treatment and ubiquitin gene expression in acidotic human subjects with chronic renal failure.

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