about
Difficulties in generating specific antibodies for immunohistochemical detection of nitrosylated tubulinsActivation state-dependent interaction between Gαq subunits and the Fhit tumor suppressor.Chronic morphine treatment attenuates cell growth of human BT474 breast cancer cells by rearrangement of the ErbB signalling network.Transcriptional regulation of the human mu-opioid receptor gene by interleukin-6.Epidermal growth factor treatment switches δ-opioid receptor-stimulated extracellular signal-regulated kinases 1 and 2 signaling from an epidermal growth factor to an insulin-like growth factor-1 receptor-dependent mechanism.Down-regulation of c-Cbl by morphine accounts for persistent ERK1/2 signaling in delta-opioid receptor-expressing HEK293 cellsdelta-Opioid receptor-stimulated Akt signaling in neuroblastoma x glioma (NG108-15) hybrid cells involves receptor tyrosine kinase-mediated PI3K activation.Ginkgo biloba extract EGb 761 exerts anti-angiogenic effects via activation of tyrosine phosphatases.Delta-opioid receptors activate ERK/MAP kinase via integrin-stimulated receptor tyrosine kinases.Chronic morphine treatment inhibits opioid receptor desensitization and internalization.Adenylyl cyclase supersensitivity in opioid-withdrawn NG108-15 hybrid cells requires Gs but is not mediated by the Gsalpha subunit.Morphine dependence in human neuroblastoma SH-SY5Y cells is associated with adaptive changes in both the quantity and functional interaction of PGE1 receptors and stimulatory G proteins.Alterations in the expression of G-proteins and regulation of adenylate cyclase in human neuroblastoma SH-SY5Y cells chronically exposed to low-efficacy mu-opioids.Regulation of stimulatory adenylyl cyclase signaling during forskolin-induced differentiation of mouse neuroblastoma x rat glioma (NG108-15) cells.Chronic morphine treatment increases stimulatory beta-2 adrenoceptor signaling in A431 cells stably expressing the mu opioid receptor.Identity of adenylyl cyclase isoform determines the G protein mediating chronic opioid-induced adenylyl cyclase supersensitivity.Chronic activation of inhibitory delta-opioid receptors cross-regulates the stimulatory adenylate cyclase-coupled prostaglandin E1 receptor system in neuroblastoma x glioma (NG108-15) hybrid cells.Coupling of prostaglandin E1 receptors to the stimulatory GTP-binding protein Gs is enhanced in neuroblastoma x glioma (NG108-15) hybrid cells chronically exposed to an opioid.delta-Opioid receptors stimulate ERK1/2 activity in NG108-15 hybrid cells by integrin-mediated transactivation of TrkA receptors.Spontaneous degenerative polioencephalomyelopathy in feeder pigs--a new motor neuron disease?Regulation of G proteins by chronic opiate and clonidine treatment in the guinea pig myenteric plexus.A novel approach to prevent endothelial hyperpermeability: the Crataegus extract WS® 1442 targets the cAMP/Rap1 pathway.Pertussis toxin blocks growth factor receptor signalling by attenuating p21ras activity.Chronic exposure of NG 108-15 cells to inhibitory acting drugs reduces stimulatory prostaglandin E1 receptor number.Suspected side effects of doxycycline use in dogs - a retrospective study of 386 casesRetinoic acid-induced differentiation of human neuroblastoma SH-SY5Y cells is associated with changes in the abundance of G proteinsSegment-dependent expression of muscarinic acetylcholine receptors and G-protein coupling in the equine respiratory tract[High incidence of jaundice in young calves in Southern Germany]
P50
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P50
description
hulumtues
@sq
researcher
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wetenschapper
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հետազոտող
@hy
name
Hermann Ammer
@ast
Hermann Ammer
@en
Hermann Ammer
@es
Hermann Ammer
@nl
Hermann Ammer
@sl
type
label
Hermann Ammer
@ast
Hermann Ammer
@en
Hermann Ammer
@es
Hermann Ammer
@nl
Hermann Ammer
@sl
prefLabel
Hermann Ammer
@ast
Hermann Ammer
@en
Hermann Ammer
@es
Hermann Ammer
@nl
Hermann Ammer
@sl
P106
P21
P31
P496
0000-0003-4935-0207