Activation of PPARγ by pioglitazone potentiates the effects of naltrexone on alcohol drinking and relapse in msP rats. - Wikidata - awgldk - TriplyDB

Activation of PPARγ by pioglitazone potentiates the effects of naltrexone on alcohol drinking and relapse in msP rats.

Activation of PPARγ by pioglitazone potentiates the effects of naltrexone on alcohol drinking and relapse in msP rats.

http://www.wikidata.org/entity/Q44226877

about

Stress-Induced Reinstatement of Drug Seeking: 20 Years of Progress.The effects of pioglitazone, a PPARγ receptor agonist, on the abuse liability of oxycodone among nondependent opioid users.PPAR agonists regulate brain gene expression: relationship to their effects on ethanol consumption Neuroimmune pathways in alcohol consumption: evidence from behavioral and genetic studies in rodents and humans Peroxisome proliferator-activated receptors α and γ are linked with alcohol consumption in mice and withdrawal and dependence in humans PPARγ activation attenuates opioid consumption and modulates mesolimbic dopamine transmission Fetal alcohol spectrum disorders and neuroimmune changes PPAR Agonists: I. Role of Receptor Subunits in Alcohol Consumption in Male and Female Mice.Preclinical Medication Development: New Targets and New Drugs.Peroxisome proliferator-activated receptor (PPAR) agonists as promising new medications for drug addiction: preclinical evidence.Emerging targets for addiction neuropharmacology: From mechanisms to therapeutics Combined pharmacotherapies for the management of alcoholism: rationale and evidence to date.Investigational therapies for the pharmacological treatment of alcoholism.Inflammatory responses to alcohol in the CNS: nuclear receptors as potential therapeutics for alcohol-induced neuropathologies Genetic Deletion of Neuronal PPARγ Enhances the Emotional Response to Acute Stress and Exacerbates Anxiety: An Effect Reversed by Rescue of Amygdala PPARγ Function.Localization of PPAR isotypes in the adult mouse and human brain.Glial and neuroinflammatory targets for treating substance use disorders.Analgesic tolerance to morphine is regulated by PPARγ.PPAR-gamma agonist pioglitazone modifies craving intensity and brain white matter integrity in patients with primary cocaine use disorder: a double-blind randomized controlled pilot trial.Multi-modal MRI classifiers identify excessive alcohol consumption and treatment effects in the brain.Ethanol and Cytokines in the Central Nervous System.Inhibition of fatty acid amide hydrolase in the central amygdala alleviates co-morbid expression of innate anxiety and excessive alcohol intake.Protection against alcohol-induced neuronal and cognitive damage by the PPARγ receptor agonist pioglitazone.PPARγ agonism attenuates cocaine cue reactivity.Pioglitazone, a PPARγ agonist, reduces nicotine craving in humans, with marginal effects on abuse potential.Peroxisome Proliferator Activated Receptor Agonists Modulate Transposable Element Expression in Brain and Liver Anti-stress neuropharmacological mechanisms and targets for addiction treatment: A translational framework

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P2860

Activation of PPARγ by pioglitazone potentiates the effects of naltrexone on alcohol drinking and relapse in msP rats.

http://www.wikidata.org/entity/Q44226877

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

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2013年學術文章

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name

Activation of PPARγ by pioglit ...... nking and relapse in msP rats.

@en

Activation of PPARγ by pioglit ...... nking and relapse in msP rats.

@nl

type

label

Activation of PPARγ by pioglit ...... nking and relapse in msP rats.

@en

Activation of PPARγ by pioglit ...... nking and relapse in msP rats.

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prefLabel

Activation of PPARγ by pioglit ...... nking and relapse in msP rats.

@en

Activation of PPARγ by pioglit ...... nking and relapse in msP rats.

@nl

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Alcoholism: Clinical and Experimental Research

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Activation of PPARγ by pioglit ...... nking and relapse in msP rats.

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Andrea Cippitelli

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George Gaitanaris

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Gregory Demopulos

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Lorenzo Somaini

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Roberto Ciccocioppo

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Serena Stopponi

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P2860

Glial cell line-derived neurotrophic factor mediates the desirable actions of the anti-addiction drug ibogaine against alcohol consumption

An evaluation of mu-opioid receptor (OPRM1) as a predictor of naltrexone response in the treatment of alcohol dependence: results from the Combined Pharmacotherapies and Behavioral Interventions for Alcohol Dependence (COMBINE) study

The mechanisms of action of PPARs

Ultra-low dose naloxone upregulates interleukin-10 expression and suppresses neuroinflammation in morphine-tolerant rat spinal cords

Cocaine-predictive stimulus induces drug-seeking behavior and neural activation in limbic brain regions after multiple months of abstinence: reversal by D(1) antagonists

Variation at the rat Crhr1 locus and sensitivity to relapse into alcohol seeking induced by environmental stress

Peroxisome proliferator-activated receptor gamma activation decreases neuroinflammation in brain after stress in rats

Fat and beyond: the diverse biology of PPARgamma

Naltrexone in the treatment of alcohol dependence.

Anti-inflammatory actions of peroxisome proliferator-activated receptor gamma agonists in Alzheimer's disease.

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1351-1360

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10.1111/ACER.12091

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8

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37

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Nazzareno Cannella

Marsida Kallupi

Giordano de Guglielmo

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2013-03-29T00:00:00Z

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236101622

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