Extracellular signal-regulated kinase inhibition by statins inhibits neutrophil activation by ANCA.
about
Do the pleiotropic effects of statins in the vasculature predict a role in inflammatory diseases?Effect of HMG-CoA reductase inhibitors on activation of human gammadeltaT cells induced by Mycobacterium tuberculosis antigens.Statins Decrease Oxidative Stress and ICD Therapies.Inhibitory effect of LXR activation on cell proliferation and cell cycle progression through lipogenic activity.p38MAPK, ERK and PI3K signaling pathways are involved in C5a-primed neutrophils for ANCA-mediated activation.The interaction between C5a and sphingosine-1-phosphate in neutrophils for antineutrophil cytoplasmic antibody mediated activation.A pro-inflammatory role of C5L2 in C5a-primed neutrophils for ANCA-induced activation.High mobility group box 1 contributes to anti-neutrophil cytoplasmic antibody-induced neutrophils activation through receptor for advanced glycation end products (RAGE) and Toll-like receptor 4.Do statins offer therapeutic potential in inflammatory arthritis?Atorvastatin, Losartan and Captopril Lead to Upregulation of TGF-β, and Downregulation of IL-6 in Coronary Artery Disease and HypertensionOutcome of rheumatoid arthritis following adjunct statin therapyHigh-mobility group box 1 potentiates antineutrophil cytoplasmic antibody-inducing neutrophil extracellular traps formation.Potential therapeutic role for statins in respiratory disease.ANCA-associated vasculitis: new options beyond steroids and cytotoxic drugs.Methacoline Challenge test as an Evaluator of Response to Statins in Bronchial Hyperresponsiveness.How anti-neutrophil cytoplasmic autoantibodies activate neutrophils.Inhibition of p38-mitogen-activated protein kinase may protect from clozapine-induced agranulocytosis.EPR studies on hydroxyl radical-scavenging activities of pravastatin and fluvastatin.Simvastatin abrogates inflamed neutrophil adhesive properties, in association with the inhibition of Mac-1 integrin expression and modulation of Rho kinase activity.Targeting novel mechanisms of pain in sickle cell disease.Autophagy is induced by anti-neutrophil cytoplasmic Abs and promotes neutrophil extracellular traps formation.Complement Factor H Inhibits Anti-Neutrophil Cytoplasmic Autoantibody-Induced Neutrophil Activation by Interacting With Neutrophils.Promotion of hypercoagulability in antineutrophil cytoplasmic antibody-associated vasculitis by C5a-induced tissue factor-expressing microparticles and neutrophil extracellular traps.Neutrophil extracellular traps can activate alternative complement pathways.
P2860
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P2860
Extracellular signal-regulated kinase inhibition by statins inhibits neutrophil activation by ANCA.
description
2003 nî lūn-bûn
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2003年の論文
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年学术文章
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name
Extracellular signal-regulated ...... neutrophil activation by ANCA.
@en
Extracellular signal-regulated ...... neutrophil activation by ANCA.
@nl
type
label
Extracellular signal-regulated ...... neutrophil activation by ANCA.
@en
Extracellular signal-regulated ...... neutrophil activation by ANCA.
@nl
prefLabel
Extracellular signal-regulated ...... neutrophil activation by ANCA.
@en
Extracellular signal-regulated ...... neutrophil activation by ANCA.
@nl
P2093
P50
P1433
P1476
Extracellular signal-regulated ...... neutrophil activation by ANCA.
@en
P2093
Madhavi Rane
Susanne Rolle
P304
P356
10.1046/J.1523-1755.2003.00718.X
P407
P577
2003-01-01T00:00:00Z
P5875
P6179
1050508143