Nucleocytoplasmic shuttling and CRM1-dependent MHC class I peptide presentation of human cytomegalovirus pp65.

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Role of human cytomegalovirus tegument proteins in virion assembly Noncanonical expression of a murine cytomegalovirus early protein CD8 T-cell epitope as an immediate early epitope based on transcription from an upstream gene.CRM1 Inhibitors for Antiviral Therapy.

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Nucleocytoplasmic shuttling and CRM1-dependent MHC class I peptide presentation of human cytomegalovirus pp65.

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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name

Nucleocytoplasmic shuttling an ...... of human cytomegalovirus pp65.

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Nucleocytoplasmic shuttling an ...... of human cytomegalovirus pp65.

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Nucleocytoplasmic shuttling an ...... of human cytomegalovirus pp65.

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Nucleocytoplasmic shuttling an ...... of human cytomegalovirus pp65.

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Nucleocytoplasmic shuttling an ...... of human cytomegalovirus pp65.

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Nucleocytoplasmic shuttling an ...... of human cytomegalovirus pp65.

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P1476

Nucleocytoplasmic shuttling an ...... of human cytomegalovirus pp65.

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P2093

Bodo Plachter

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Nadine Frankenberg

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Peter Lischka

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Sandra Pepperl-Klindworth

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P2860

No strings attached: the ESCRT machinery in viral budding and cytokinesis

Leptomycin B inactivates CRM1/exportin 1 by covalent modification at a cysteine residue in the central conserved region

RNA-binding of the human cytomegalovirus transactivator protein UL69, mediated by arginine-rich motifs, is not required for nuclear export of unspliced RNA.

DRiPs solidify: progress in understanding endogenous MHC class I antigen processing

CRM1 is an export receptor for leucine-rich nuclear export signals

The HIV-1 Rev activation domain is a nuclear export signal that accesses an export pathway used by specific cellular RNAs

Inhibitors of the proteasome block the degradation of most cell proteins and the generation of peptides presented on MHC class I molecules

A novel transferable nuclear export signal mediates CRM1-independent nucleocytoplasmic shuttling of the human cytomegalovirus transactivator protein pUL69

Nuclear export is evolutionarily conserved in CVC paired-like homeobox proteins and influences protein stability, transcriptional activation, and extracellular secretion.

Identification of a signal for rapid export of proteins from the nucleus

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s00430-012-0269-7

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567-579

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scholarly article

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10.1007/S00430-012-0269-7

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201

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Thomas Stamminger

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2012-09-11T00:00:00Z

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22965172

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P921

Cytomegalovirus

Nucleocytoplasmic shuttling and CRM1-dependent MHC class I peptide presentation of human cytomegalovirus pp65.

about

P2860

P2860

Nucleocytoplasmic shuttling and CRM1-dependent MHC class I peptide presentation of human cytomegalovirus pp65.

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