Type 1 interferon (IFNalpha/beta) and type 2 nitric oxide synthase regulate the innate immune response to a protozoan parasite.
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Using Proteomics to Understand How Leishmania Parasites Survive inside the Host and Establish InfectionImmunization of Mastomys coucha with Brugia malayi recombinant trehalose-6-phosphate phosphatase results in significant protection against homologous challenge infectionInnate killing of Leishmania donovani by macrophages of the splenic marginal zone requires IRF-7Spotlights on immunological effects of reactive nitrogen species: When inflammation says nitric oxideSelective associations with signaling proteins determine stimulatory versus costimulatory activity of NKG2DInterferon-alphabeta mediates partial control of early pulmonary Mycobacterium bovis bacillus Calmette-Guérin infectionLeishmania major infection in humanized mice induces systemic infection and provokes a nonprotective human immune responseGenetic Manipulation of Leishmania donovani to Explore the Involvement of Argininosuccinate Synthase in Oxidative Stress ManagementFibroblasts as host cells in latent leishmaniosisEvolution of lesion formation, parasitic load, immune response, and reservoir potential in C57BL/6 mice following high- and low-dose challenge with Leishmania major.Leishmania major infection of inbred mice: unmasking genetic determinants of infectious diseases.A defective TLR4 signaling for IFN-β expression is responsible for the innately lower ability of BALB/c macrophages to produce NO in response to LPS as compared to C57BL/6.Human dendritic cells exhibit a pronounced type I IFN signature following Leishmania major infection that is required for IL-12 inductionIdentification of nitric oxide synthase 2 as an innate resistance locus against ectromelia virus infection.Regulatory T cell-mediated resolution of lung injury: identification of potential target genes via expression profiling.Intranasal Poly-IC treatment exacerbates tuberculosis in mice through the pulmonary recruitment of a pathogen-permissive monocyte/macrophage population.Direct activation of innate and antigen-presenting functions of microglia following infection with Theiler's virus.Early gene expression of NK cell-activating chemokines in mice resistant to Leishmania major.Influence of vector-encoded cytokines on anti-Salmonella immunity: divergent effects of interleukin-2 and tumor necrosis factor alphaThe SPRY domain-containing SOCS box protein SPSB2 targets iNOS for proteasomal degradation.Plasmodium falciparum-infected erythrocytes and IL-12/IL-18 induce diverse transcriptomes in human NK cells: IFN-α/β pathway versus TREM signaling.Interferon-inducible effector mechanisms in cell-autonomous immunity.Expression of inducible nitric oxide synthase in skin lesions of patients with american cutaneous leishmaniasis.Type I IFN receptor regulates neutrophil functions and innate immunity to Leishmania parasites.Regulation of impaired protein kinase C signaling by chemokines in murine macrophages during visceral leishmaniasis.Topical resiquimod protects against visceral infection with Leishmania infantum chagasi in mice.The transcription factor nuclear factor of activated T cells c3 modulates the function of macrophages in sepsis.Role of protein kinase R in the killing of Leishmania major by macrophages in response to neutrophil elastase and TLR4 via TNFα and IFNβ.Regulatory role of T cells producing both interferon gamma and interleukin 10 in persistent infectionCutaneous Na+ storage strengthens the antimicrobial barrier function of the skin and boosts macrophage-driven host defense.Role of cytokines in the innate immune response to intracellular pathogensIFNs and STATs in innate immunity to microorganisms.Trypanosoma congolense Infections: Induced Nitric Oxide Inhibits Parasite Growth In VivoLeishmania promotes its own virulence by inducing expression of the host immune inhibitory ligand CD200Leishmania-infected macrophages are targets of NK cell-derived cytokines but not of NK cell cytotoxicityTGF-beta: the perpetrator of immune suppression by regulatory T cells and suicidal T cells.Gene expression induced by Toll-like receptors in macrophages requires the transcription factor NFAT5Both the Fas ligand and inducible nitric oxide synthase are needed for control of parasite replication within lesions in mice infected with Leishmania major whereas the contribution of tumor necrosis factor is minimal.Reactive oxygen species and nitric oxide in cutaneous leishmaniasis.T helper1/t helper2 cells and resistance/susceptibility to leishmania infection: is this paradigm still relevant?
P2860
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P2860
Type 1 interferon (IFNalpha/beta) and type 2 nitric oxide synthase regulate the innate immune response to a protozoan parasite.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Type 1 interferon (IFNalpha/be ...... ponse to a protozoan parasite.
@en
Type 1 interferon
@nl
type
label
Type 1 interferon (IFNalpha/be ...... ponse to a protozoan parasite.
@en
Type 1 interferon
@nl
prefLabel
Type 1 interferon (IFNalpha/be ...... ponse to a protozoan parasite.
@en
Type 1 interferon
@nl
P2093
P1433
P1476
Type 1 interferon (IFNalpha/be ...... ponse to a protozoan parasite.
@en
P2093
Diefenbach A
Donhauser N
MacMicking J
Röllinghoff M
Schindler H
P356
10.1016/S1074-7613(00)80460-4
P407
P577
1998-01-01T00:00:00Z