Nitric oxide, NOC-12, and S-nitrosoglutathione modulate the skeletal muscle calcium release channel/ryanodine receptor by different mechanisms. An allosteric function for O2 in S-nitrosylation of the channel.
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Regulation of cardiovascular cellular processes by S-nitrosylationHypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscleHuman spermatozoa contain multiple targets for protein S-nitrosylation: an alternative mechanism of the modulation of sperm function by nitric oxide?Potential molecular mechanisms underlying muscle fatigue mediated by reactive oxygen and nitrogen speciesROS-mediated decline in maximum Ca2+-activated force in rat skeletal muscle fibers following in vitro and in vivo stimulation.Stabilization of the skeletal muscle ryanodine receptor ion channel-FKBP12 complex by the 1,4-benzothiazepine derivative S107Regulation of Ryanodine Receptor Ion Channels Through Posttranslational Modifications.Dynamic denitrosylation via S-nitrosoglutathione reductase regulates cardiovascular function.Antioxidants protect calsequestrin-1 knockout mice from halothane- and heat-induced sudden death.Nitric oxide and blood: a review.Cross talk between Ca2+ and redox signalling cascades in muscle and neurons through the combined activation of ryanodine receptors/Ca2+ release channels.S-nitrosylation: integrator of cardiovascular performance and oxygen delivery.S-nitrosylation: NO-related redox signaling to protect against oxidative stress.Metabolic signalling in defence and stress: the central roles of soluble redox couples.Nitric oxide in blood. The nitrosative-oxidative disequilibrium hypothesis on the pathogenesis of cardiovascular disease.Regulation of the cardiac muscle ryanodine receptor by O(2) tension and S-nitrosoglutathione.Oxygen-coupled redox regulation of the skeletal muscle ryanodine receptor/Ca2+ release channel (RyR1): sites and nature of oxidative modification.Nitric Oxide-induced Activation of the Type 1 Ryanodine Receptor Is Critical for Epileptic Seizure-induced Neuronal Cell Death.Regulation of the skeletal muscle ryanodine receptor/Ca2+-release channel RyR1 by S-palmitoylation.Nitrosative modifications of protein and lipid signaling molecules by reactive nitrogen species.S-glutathionylation of ion channels: insights into the regulation of channel functions, thiol modification crosstalk, and mechanosensingNitric Oxide-Induced Calcium Release: Activation of Type 1 Ryanodine Receptor, a Calcium Release Channel, through Non-Enzymatic Post-Translational Modification by Nitric Oxide.GSNOR Deficiency Enhances In Situ Skeletal Muscle Strength, Fatigue Resistance, and RyR1 S-Nitrosylation Without Impacting Mitochondrial Content and Activity.Nitric oxide block of outward-rectifying K+ channels indicates direct control by protein nitrosylation in guard cells.Role of Cys³⁶⁰² in the function and regulation of the cardiac ryanodine receptor.Nitric oxide-induced calcium release: activation of type 1 ryanodine receptor by endogenous nitric oxide.Leaky ryanodine receptors in β-sarcoglycan deficient mice: a potential common defect in muscular dystrophy.RyR1 S-nitrosylation underlies environmental heat stroke and sudden death in Y522S RyR1 knockin mice.Effects on kidney filtration rate by agmatine requires activation of ryanodine channels for nitric oxide generation.S-glutathionylation decreases Mg2+ inhibition and S-nitrosylation enhances Ca2+ activation of RyR1 channels.Control of intracellular calcium in the presence of nitric oxide donors in isolated skeletal muscle fibres from mouse.Permissive contributions of NO and prostacyclin in CO-induced cerebrovascular dilation in piglets.Nitric oxide synthase inhibition affects sarcoplasmic reticulum Ca2+ release in skeletal muscle fibres from mouse.Hypoxic regulation of nitric oxide signaling in vascular smooth muscle.Signaling Mechanisms in the Nitric Oxide Donor- and Amphetamine-Induced Dopamine Release in Mesencephalic Primary Cultured Neurons.PDI-mediated S-nitrosylation of DRP1 facilitates DRP1-S616 phosphorylation and mitochondrial fission in CA1 neurons
P2860
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P2860
Nitric oxide, NOC-12, and S-nitrosoglutathione modulate the skeletal muscle calcium release channel/ryanodine receptor by different mechanisms. An allosteric function for O2 in S-nitrosylation of the channel.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Nitric oxide, NOC-12, and S-ni ...... -nitrosylation of the channel.
@en
Nitric oxide, NOC-12, and S-ni ...... -nitrosylation of the channel.
@nl
type
label
Nitric oxide, NOC-12, and S-ni ...... -nitrosylation of the channel.
@en
Nitric oxide, NOC-12, and S-ni ...... -nitrosylation of the channel.
@nl
prefLabel
Nitric oxide, NOC-12, and S-ni ...... -nitrosylation of the channel.
@en
Nitric oxide, NOC-12, and S-ni ...... -nitrosylation of the channel.
@nl
P2093
P2860
P356
P1476
Nitric oxide, NOC-12, and S-ni ...... -nitrosylation of the channel.
@en
P2093
Gerhard Meissner
Jerry P Eu
Jonathan S Stamler
Junhui Sun
P2860
P304
P356
10.1074/JBC.M211940200
P407
P577
2002-12-30T00:00:00Z