Alterations in food intake by opioid and dopamine signaling pathways between the ventral tegmental area and the shell of the nucleus accumbens.
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The New Era of Drug Therapy for Obesity: The Evidence and the ExpectationsKnockdown of ventral tegmental area mu-opioid receptors in rats prevents effects of social defeat stress: implications for amphetamine cross-sensitization, social avoidance, weight regulation and expression of brain-derived neurotrophic factor.The action of leptin in the ventral tegmental area to decrease food intake is dependent on Jak-2 signalingBehavioral functions of the mesolimbic dopaminergic system: an affective neuroethological perspective.Metabolic hormones, dopamine circuits, and feeding.Energy regulatory signals and food reward.Dopaminergic dysregulation in mice selectively bred for excessive exercise or obesity.Sugar addiction: the state of the science.Amygdalar opioids modulate hypothalamic melanocortin-induced anorexiaNucleus accumbens dopamine/glutamate interaction switches modes to generate desire versus dread: D(1) alone for appetitive eating but D(1) and D(2) together for fearIncretins and amylin: neuroendocrine communication between the gut, pancreas, and brain in control of food intake and blood glucose.GLP-1 neurons in the nucleus of the solitary tract project directly to the ventral tegmental area and nucleus accumbens to control for food intake.Reward processing by the opioid system in the brain.How can drug addiction help us understand obesity?Modulation of food reward by adiposity signals.Pain and suicidality: insights from reward and addiction neuroscience.Insulin acts at different CNS sites to decrease acute sucrose intake and sucrose self-administration in rats.Hypothalamic injection of non-opioid peptides increases gene expression of the opioid enkephalin in hypothalamic and mesolimbic nuclei: Possible mechanism underlying their behavioral effects.A systematic investigation of the differential roles for ventral tegmentum serotonin 1- and 2-type receptors on food intake in the rat.The role of nucleus accumbens adenosine-opioid interaction in mediating palatable food intake.Opioids in the nucleus accumbens stimulate ethanol intake.Opioid-dependent anticipatory negative contrast and binge-like eating in rats with limited access to highly preferred food.Consuming a low-fat diet from weaning to adulthood reverses the programming of food preferences in male, but not in female, offspring of 'junk food'-fed rat dams.Quantitative Mass Spectrometry Reveals Food Intake-Induced Neuropeptide Level Changes in Rat Brain: Functional Assessment of Selected Neuropeptides as Feeding Regulators.Selective dopaminergic lesions of the ventral tegmental area impair preference for sucrose but not for male sexual pheromones in female mice.Descending projections from the nucleus accumbens shell suppress activity of taste-responsive neurons in the hamster parabrachial nuclei.Relationship between substance use and body mass index in young males.
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P2860
Alterations in food intake by opioid and dopamine signaling pathways between the ventral tegmental area and the shell of the nucleus accumbens.
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2004年の論文
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Alterations in food intake by ...... hell of the nucleus accumbens.
@en
Alterations in food intake by ...... hell of the nucleus accumbens.
@nl
type
label
Alterations in food intake by ...... hell of the nucleus accumbens.
@en
Alterations in food intake by ...... hell of the nucleus accumbens.
@nl
prefLabel
Alterations in food intake by ...... hell of the nucleus accumbens.
@en
Alterations in food intake by ...... hell of the nucleus accumbens.
@nl
P2093
P921
P1433
P1476
Alterations in food intake by ...... hell of the nucleus accumbens.
@en
P2093
Allen S Levine
Amy F MacDonald
Charles J Billington
P356
10.1016/J.BRAINRES.2004.05.043
P407
P577
2004-08-01T00:00:00Z