The mu-opioid receptor polymorphism A118G predicts cortisol responses to naloxone and stress. - Wikidata - awgldk - TriplyDB

The mu-opioid receptor polymorphism A118G predicts cortisol responses to naloxone and stress.

The mu-opioid receptor polymorphism A118G predicts cortisol responses to naloxone and stress.

http://www.wikidata.org/entity/Q44350222

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Cortisol Stress Response in Men and Women Modulated Differentially by the Mu-Opioid Receptor Gene Polymorphism OPRM1 A118G.Stress and the HPA axis: role of glucocorticoids in alcohol dependence.Endogenous opiates and behavior: 2006 Limited effects of beta-endorphin compared to loperamide or fentanyl in a neuroendocrine biomarker assay in non-human primates.Influence of beta-Endorphin on anxious behavior in mice: interaction with EtOH Drug-induced and genetic alterations in stress-responsive systems: Implications for specific addictive diseases.Subjective health complaints in patients with lumbar radicular pain and disc herniation are associated with a sex - OPRM1 A118G polymorphism interaction: a prospective 1-year observational study Effect of OPRM1 and stressful life events on symptoms of major depression in African American adolescents OPRM1 SNP (A118G): involvement in disease development, treatment response, and animal models.A new biomarker of hedonic eating? A preliminary investigation of cortisol and nausea responses to acute opioid blockade.Human behavioral pharmacology, past, present, and future: symposium presented at the 50th annual meeting of the Behavioral Pharmacology Society.Effects of opioid receptor gene variation on targeted nalmefene treatment in heavy drinkers.Association of BDNF Val66Met polymorphism with HPA and SAM axis reactivity to psychological and physical stress.Opioid partial agonist buprenorphine dampens responses to psychosocial stress in humans Human Mu Opioid Receptor (OPRM1 A118G) polymorphism is associated with brain mu-opioid receptor binding potential in smokers Neurotransmitter and neuromodulator genes associated with a history of depressive symptoms in individuals with alcohol dependence OPRM1 gene variation influences hypothalamic-pituitary-adrenal axis function in response to a variety of stressors in rhesus macaques.Mouse model of OPRM1 (A118G) polymorphism increases sociability and dominance and confers resilience to social defeat Genetics of stress response and stress-related disorders.Pharmacological consequence of the A118G μ opioid receptor polymorphism on morphine- and fentanyl-mediated modulation of Ca²⁺ channels in humanized mouse sensory neurons.Sex differences in acute hormonal and subjective response to naltrexone: The impact of menstrual cycle phase Decreased response to social defeat stress in μ-opioid-receptor knockout mice.Opiate addiction and cocaine addiction: underlying molecular neurobiology and genetics.β-endorphin modulates the effect of stress on novelty-suppressed feeding Genetic association of FKBP5 and CRHR1 with cortisol response to acute psychosocial stress in healthy adults.Self-rated health and interleukin-6: Longitudinal relationships in older adults.Variation in OPRM1 and risk of suicidal behavior in drug-dependent individuals.Blunted opiate modulation of hypothalamic-pituitary-adrenocortical activity in men and women who smoke.Investigating the molecular basis of major depressive disorder etiology: a functional convergent genetic approach The genetics of the opioid system and specific drug addictions Role of a functional human gene polymorphism in stress responsivity and addictions Stress, alcohol and drug interaction: an update of human research.Genetic variability of the mu-opioid receptor influences intrathecal fentanyl analgesia requirements in laboring women OPRM1 Asn40Asp predicts response to naltrexone treatment: a haplotype-based approach Mu-opioid receptor A118G polymorphism in healthy volunteers affects hypothalamic-pituitary-adrenal axis adrenocorticotropic hormone stress response to metyrapone Mouse model of OPRM1 (A118G) polymorphism has sex-specific effects on drug-mediated behavior.Role of the HPA axis and the A118G polymorphism of the mu-opioid receptor in stress-induced drinking behavior.Strategies for performing genotype-phenotype association studies in nonhuman primates.Neurobiological Aspects of Mindfulness in Pain Autoregulation: Unexpected Results from a Randomized-Controlled Trial and Possible Implications for Meditation Research.Pharmacogenetically driven treatments for alcoholism: are we there yet?

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The mu-opioid receptor polymorphism A118G predicts cortisol responses to naloxone and stress.

http://www.wikidata.org/entity/Q44350222

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The mu-opioid receptor polymor ...... ponses to naloxone and stress.

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The mu-opioid receptor polymor ...... ponses to naloxone and stress.

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The mu-opioid receptor polymor ...... ponses to naloxone and stress.

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Neuropsychopharmacology

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Gary S Wand

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Lynn Oswald

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Magdalena Uhart

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Rachel Y Chong

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Xiaoju Yang

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P2860

Single-nucleotide polymorphism in the human mu opioid receptor gene alters beta-endorphin binding and activity: possible implications for opiate addiction

Hypothalamic-pituitary-adrenal axis, neuroendocrine factors and stress

Protective and damaging effects of stress mediators

Longitudinal data analysis for discrete and continuous outcomes

Glucocorticoids and hippocampal atrophy in neuropsychiatric disorders.

Altered pituitary-adrenal axis responses to provocative challenge tests in adult survivors of childhood abuse.

Endogenous opioids and reward.

The 'Trier Social Stress Test'--a tool for investigating psychobiological stress responses in a laboratory setting.

Endogenous opiates: 1999.

Stress and depression: possible links to neuron death in the hippocampus.

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