Increased susceptibility of mice lacking T-bet to infection with Mycobacterium tuberculosis correlates with increased IL-10 and decreased IFN-gamma production.
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Complexity and Controversies over the Cytokine Profiles of T Helper Cell Subpopulations in TuberculosisDefining features of protective CD4 T cell responses to Mycobacterium tuberculosisThe role of IL-10 in Mycobacterium avium subsp. paratuberculosis infectionEx vivo cytokine mRNA levels correlate with changing clinical status of ethiopian TB patients and their contacts over timeCutting edge: control of Mycobacterium tuberculosis infection by a subset of lung parenchyma-homing CD4 T cellsCD4 T cells promote rather than control tuberculosis in the absence of PD-1-mediated inhibitionAcute immune response to Mycobacterium massiliense in C57BL/6 and BALB/c mice.CCL5 participates in early protection against Mycobacterium tuberculosis.Transcription factor T-bet regulates inflammatory arthritis through its function in dendritic cells.Role of interleukin 10 transcriptional regulation in inflammation and autoimmune diseaseIL-17 and Th17 cells in tuberculosisToll-like receptor 2-dependent extracellular signal-regulated kinase signaling in Mycobacterium tuberculosis-infected macrophages drives anti-inflammatory responses and inhibits Th1 polarization of responding T cellsThe TBX21 transcription factor T-1993C polymorphism is associated with decreased IFN-γ and IL-4 production by primary human lymphocytes.The transcription factor NFATp plays a key role in susceptibility to TB in mice.Nocardia brasiliensis induces an immunosuppressive microenvironment that favors chronic infection in BALB/c mice.Constitutive expression of SMAR1 confers susceptibility to Mycobacterium tuberculosis infection in a transgenic mouse model.Life and death in the granuloma: immunopathology of tuberculosis.Host responses to Mycobacterium avium subsp. paratuberculosis: a complex arsenal.Trawling for treasure: tales of T-bet.Differential expression of interleukin-4 (IL-4) and IL-4 delta 2 mRNA, but not transforming growth factor beta (TGF-beta), TGF-beta RII, Foxp3, gamma interferon, T-bet, or GATA-3 mRNA, in patients with fast and slow responses to antituberculosis treDominant role of the sst1 locus in pathogenesis of necrotizing lung granulomas during chronic tuberculosis infection and reactivation in genetically resistant hosts.Stat4-dependent, T-bet-independent regulation of IL-10 in NK cellsSignal transducer and activator of transcription 4 is required for the transcription factor T-bet to promote T helper 1 cell-fate determination.Nickel nanoparticles cause exaggerated lung and airway remodeling in mice lacking the T-box transcription factor, TBX21 (T-bet)T-bet regulates immunity to Francisella tularensis live vaccine strain infection, particularly in lungs.Transcriptional regulation of the mucosal immune system mediated by T-bet.Modulation of cell death by M. tuberculosis as a strategy for pathogen survivalT-bet in disease.The role of protein modifications of T-bet in cytokine production and differentiation of T helper cells.Innate and Adaptive Cellular Immune Responses to Mycobacterium tuberculosis Infection.Th1 Differentiation Drives the Accumulation of Intravascular, Non-protective CD4 T Cells during Tuberculosis.IL-36γ/IL-1F9, an innate T-bet target in myeloid cells.IL-21 Receptor Signaling Is Essential for Optimal CD4+ T Cell Function and Control of Mycobacterium tuberculosis Infection in Mice.Defective positioning in granulomas but not lung-homing limits CD4 T-cell interactions with Mycobacterium tuberculosis-infected macrophages in rhesus macaques.Expression pattern of transcription factors and intracellular cytokines reveals that clinically cured tuberculosis is accompanied by an increase in Mycobacterium-specific Th1, Th2, and Th17 cells.Potential Role of Hepatitis C Virus Alternate Reading Frame Protein in Negative Regulation of T-Bet Gene Expression.TBX21 polymorphisms are associated with virus persistence in hepatitis C virus infection patients from a high-risk Chinese population.Diverse roles for T-bet in the effector responses required for resistance to infection.T-bet protects against exacerbation of schistosome egg-induced immunopathology by regulating Th17-mediated inflammation.T-bet-dependent regulation of CD8+ T-cell expansion during experimental Trypanosoma cruzi infection.
P2860
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P2860
Increased susceptibility of mice lacking T-bet to infection with Mycobacterium tuberculosis correlates with increased IL-10 and decreased IFN-gamma production.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Increased susceptibility of mi ...... ecreased IFN-gamma production.
@en
Increased susceptibility of mi ...... ecreased IFN-gamma production.
@nl
type
label
Increased susceptibility of mi ...... ecreased IFN-gamma production.
@en
Increased susceptibility of mi ...... ecreased IFN-gamma production.
@nl
prefLabel
Increased susceptibility of mi ...... ecreased IFN-gamma production.
@en
Increased susceptibility of mi ...... ecreased IFN-gamma production.
@nl
P2093
P1476
Increased susceptibility of mi ...... ecreased IFN-gamma production.
@en
P2093
Brandon M Sullivan
Kristine Vasquez
Laurie H Glimcher
Ousman Jobe
Roderick Bronson
Vanja Lazarevic
P304
P356
10.4049/JIMMUNOL.175.7.4593
P407
P50
P577
2005-10-01T00:00:00Z