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High hydrostatic pressure inhibits the biosynthesis of eukaryotic elongation factor-2Variability in the upstream promoter and intron sequences of the human, mouse and chick type X collagen genesChanged lamellipodial extension, adhesion plaques and migration in epidermal keratinocytes containing constitutively expressed sense and antisense hyaluronan synthase 2 (Has2) genes[Age-related macular degeneration--a difficult problem for the patient and for the ophthalmologist].Proteomic changes in articular cartilage of human endemic osteoarthritis in China.Contrast agent enhanced pQCT of articular cartilage.Geldanamycin activates Hsp70 response and attenuates okadaic acid-induced cytotoxicity in human retinal pigment epithelial cells.Morphology and phenotype expression of types I, II, III, and X collagen and MMP-13 of chondrocytes cultured from articular cartilage of Kashin-Beck Disease.Proteomics of chondrocytes with special reference to phosphorylation changes of proteins in stretched human chondrosarcoma cells.Bioreactor improves the growth and viability of chondrocytes in the knitted poly-L,D-lactide scaffold.Improved adherence and spreading of Saos-2 cells on polypropylene surfaces achieved by surface texturing and carbon nitride coating.Adaptation of canine femoral head articular cartilage to long distance running exercise in young beagles.Cellular responses to T-2 toxin and/or deoxynivalenol that induce cartilage damage are not specific to chondrocytes.Rho-kinase inhibitor Y-27632 and hypoxia synergistically enhance chondrocytic phenotype and modify S100 protein profiles in human chondrosarcoma cells.Hypotonic challenge modulates cell volumes differently in the superficial zone of intact articular cartilage and cartilage explant.Regular joint loading in youth assists in the establishment and strengthening of the collagen network of articular cartilage and contributes to the prevention of osteoarthrosis later in life: a hypothesis.Hyperosmolaric contrast agents in cartilage tomography may expose cartilage to overload-induced cell death.Genome-wide gene expression analysis suggests an important role of suppressed immunity in pathogenesis of Kashin-Beck disease.The effect of collagen degradation on chondrocyte volume and morphology in bovine articular cartilage following a hypotonic challenge.TiO2 coating promotes human mesenchymal stem cell proliferation without the loss of their capacity for chondrogenic differentiation.Effects of medium and temperature on cellular responses in the superficial zone of hypo-osmotically challenged articular cartilage.Osteoblast behavior on various ultra short pulsed laser deposited surface coatings.The expression of p-ATF2 involved in the chondeocytes apoptosis of an endemic osteoarthritis, Kashin-Beck disease.Effects of Freeze-Thaw Cycle with and without Proteolysis Inhibitors and Cryopreservant on the Biochemical and Biomechanical Properties of Articular Cartilage.In vitro glycation of articular cartilage alters the biomechanical response of chondrocytes in a depth-dependent manner.Immobilisation causes longlasting matrix changes both in the immobilised and contralateral joint cartilage.Gene expression signature in endemic osteoarthritis by microarray analysisHydrostatic pressure-induced changes in cellular protein synthesis.Current perspectives on cartilage and chondrocyte mechanobiology.Hsp70 accumulation in chondrocytic cells exposed to high continuous hydrostatic pressure coincides with mRNA stabilization rather than transcriptional activationEffects of Articular Cartilage Constituents on Phosphotungstic Acid Enhanced Micro-Computed Tomography.Identified molecular mechanism of interaction between environmental risk factors and differential expression genes in cartilage of Kashin-Beck disease.Scaffold-free approach produces neocartilage tissue of similar quality as the use of HyStem™ and Hydromatrix™ scaffoldsRecent advances in the research of an endemic osteochondropathy in China: Kashin-Beck disease.Field synopsis and meta-analyses of genetic epidemiological evidence for Kashin-Beck disease, an endemic osteoarthropathy in China.Identification of differentially expressed genes and pathways between primary osteoarthritis and endemic osteoarthritis (Kashin-Beck disease).Gene expression profiles and molecular mechanism of cultured human chondrocytes' exposure to T-2 toxin and deoxynivalenol.The potential of induced pluripotent stem cells as a tool to study skeletal dysplasias and cartilage-related pathologic conditions.Chondrocytic cells express the taurine transporter on their plasma membrane and regulate its expression under anisotonic conditions.Chondrogenic differentiation of human pluripotent stem cells in chondrocyte co-culture.
P50
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description
Zweeds onderzoeker
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Mikko J. Lammi
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Mikko J. Lammi
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Mikko J. Lammi
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P1153
7006572063
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P27
P31
P496
0000-0002-6181-9904