Overexpression of MicroRNA-1 improves the efficacy of mesenchymal stem cell transplantation after myocardial infarction. - Wikidata - awgldk - TriplyDB

Overexpression of MicroRNA-1 improves the efficacy of mesenchymal stem cell transplantation after myocardial infarction.

Overexpression of MicroRNA-1 improves the efficacy of mesenchymal stem cell transplantation after myocardial infarction.

http://www.wikidata.org/entity/Q44415960

about

How to Improve the Survival of Transplanted Mesenchymal Stem Cell in Ischemic Heart?Roles of the canonical myomiRs miR-1, -133 and -206 in cell development and disease Exosomes in mesenchymal stem cells, a new therapeutic strategy for cardiovascular diseases?miRNome in myocardial infarction: Future directions and perspective Cell adhesion and long-term survival of transplanted mesenchymal stem cells: a prerequisite for cell therapy Cardiac stem cell therapy for cardiac repair."Second-generation" stem cells for cardiac repair microRNA-378 promotes mesenchymal stem cell survival and vascularization under hypoxic-ischemic conditions in vitro.Monocyte Chemoattractant Protein-Induced Protein 1 (MCPIP1) Enhances Angiogenic and Cardiomyogenic Potential of Murine Bone Marrow-Derived Mesenchymal Stem Cells.miR-23 regulate the pathogenesis of patients with coronary artery disease Let-7b-mediated pro-survival of transplanted mesenchymal stem cells for cardiac regeneration.microRNA-based diagnostics and therapy in cardiovascular disease-Summing up the facts.Can the outcomes of mesenchymal stem cell-based therapy for myocardial infarction be improved? Providing weapons and armour to cells.Possible Muscle Repair in the Human Cardiovascular System.Long nonding RNA UCA1 regulates neural stem cell differentiation by controlling miR-1/Hes1 expression.Antioxidant Therapeutic Strategies for Cardiovascular Conditions Associated with Oxidative Stress.MiR-23a Regulates the Vasculogenesis of Coronary Artery Disease by Targeting Epidermal Growth Factor Receptor.MiR-214 regulates the pathogenesis of patients with coronary artery disease by targeting VEGF.miR-10a rejuvenates aged human mesenchymal stem cells and improves heart function after myocardial infarction through KLF4.Priming stem cells with protein kinase C activator enhances early stem cell-chondrocyte interaction by increasing adhesion molecules

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Overexpression of MicroRNA-1 improves the efficacy of mesenchymal stem cell transplantation after myocardial infarction.

http://www.wikidata.org/entity/Q44415960

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Overexpression of MicroRNA-1 i ...... n after myocardial infarction.

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Overexpression of MicroRNA-1 i ...... n after myocardial infarction.

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Overexpression of MicroRNA-1 i ...... n after myocardial infarction.

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Overexpression of MicroRNA-1 i ...... n after myocardial infarction.

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Overexpression of MicroRNA-1 i ...... n after myocardial infarction.

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Overexpression of MicroRNA-1 i ...... n after myocardial infarction.

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Feng Huang

http://www.w3.org/2001/XMLSchema#string

Liang Tang

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Mei-Ling Li

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Qi-Ming Liu

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Sheng-Hua Zhou

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Xin-Qun Hu

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Yan-Shu Zhao

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Zhen-Fei Fang

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Zhen-Jiang Liu

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P2860

Notch promotes neural lineage entry by pluripotent embryonic stem cells.

MicroRNA1 influences cardiac differentiation in Drosophila and regulates Notch signaling

The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation

MicroRNA regulation of cell lineages in mouse and human embryonic stem cells

Normal microRNA maturation and germ-line stem cell maintenance requires Loquacious, a double-stranded RNA-binding domain protein

Multilineage potential of adult human mesenchymal stem cells

MicroRNAs: small RNAs with a big role in gene regulation

MicroRNAs: genomics, biogenesis, mechanism, and function

MicroRNA-1 and -499 regulate differentiation and proliferation in human-derived cardiomyocyte progenitor cells

The muscle-specific microRNAs miR-1 and miR-133 produce opposing effects on apoptosis by targeting HSP60, HSP70 and caspase-9 in cardiomyocytes

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