Persistent sodium and calcium currents cause plateau potentials in motoneurons of chronic spinal rats.
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Recovery of neuronal and network excitability after spinal cord injury and implications for spasticityInhibition of evoked glutamate release by neurosteroid allopregnanolone via inhibition of L-type calcium channels in rat medial prefrontal cortexL-type calcium channels: the low downAlternative splicing in the voltage-gated sodium channel DmNav regulates activation, inactivation, and persistent current.Postinhibitory rebound neurons and networks are disrupted in retrovirus-induced spongiform neurodegeneration.Increased persistent Na+ current contributes to seizure in the slamdance bang-sensitive Drosophila mutant.Proprioceptive neuropathy affects normalization of the H-reflex by exercise after spinal cord injury.Flexor reflex decreases during sympathetic stimulation in chronic human spinal cord injury.Disturbances of motor unit rate modulation are prevalent in muscles of spastic-paretic stroke survivorsManagement of spasticity after spinal cord injury: current techniques and future directions.Amphetamine increases persistent inward currents in human motoneurons estimated from paired motor-unit activityCharacterization of motor units in behaving adult mice shows a wide primary range.Relative location of inhibitory synapses and persistent inward currents determines the magnitude and mode of synaptic amplification in motoneurons.Self-assembling peptide amphiphile promotes plasticity of serotonergic fibers following spinal cord injury.External urethral sphincter motoneuron properties in adult female rats studied in vitroPersistent currents and discharge patterns in rat hindlimb motoneuronsCharacteristics and organization of discharge properties in rat hindlimb motoneurons.Neuromodulation impact on nonlinear firing behavior of a reduced model motoneuron with the active dendrite.Swimming against the tide: investigations of the C-bouton synapse.Motor neuron firing dysfunction in spastic patients with primary lateral sclerosisThe retrograde frequency response of passive dendritic trees constrains the nonlinear firing behaviour of a reduced neuron model.Persistent inward currents in spinal motoneurons: important for normal function but potentially harmful after spinal cord injury and in amyotrophic lateral sclerosis.Origins of spontaneous firing of motor units in the spastic-paretic biceps brachii muscle of stroke survivorsWind-up of stretch reflexes as a measure of spasticity in chronic spinalized rats: The effects of passive exercise and modafinil.Adrenergic receptors modulate motoneuron excitability, sensory synaptic transmission and muscle spasms after chronic spinal cord injury.Motoneuron excitability and muscle spasms are regulated by 5-HT2B and 5-HT2C receptor activity.Molecular Mechanisms Underlying Cell Death in Spinal Networks in Relation to Locomotor Activity After Acute Injury in vitro.Dendritic spine dysgenesis contributes to hyperreflexia after spinal cord injury.Polysynaptic excitatory postsynaptic potentials that trigger spasms after spinal cord injury in rats are inhibited by 5-HT1B and 5-HT1F receptorsP2Y1 receptor-mediated potentiation of inspiratory motor output in neonatal rat in vitro.Calcium currents of olfactory bulb juxtaglomerular cells: profile and multiple conductance plateau potential simulation.Altered obstacle negotiation after low thoracic hemisection in the cat.Asymmetry in signal propagation between the soma and dendrites plays a key role in determining dendritic excitability in motoneurons.Effects of persistent inward currents, accommodation, and adaptation on motor unit behavior: a simulation studySerotonin receptor and KCC2 gene expression in lumbar flexor and extensor motoneurons posttransection with and without passive cycling.Modulation of inhibitory strength and kinetics facilitates regulation of persistent inward currents and motoneuron excitability following spinal cord injuryPersistent inward currents in spinal motoneurons and their influence on human motoneuron firing patterns.Serotonin modulates multiple calcium current subtypes in commissural interneurons of the neonatal mouseEffect of prolonged riluzole exposure on cultured motoneurons in a mouse model of ALS.Differential contributions of somatic and dendritic calcium-dependent potassium currents to the control of motoneuron excitability following spinal cord injury.
P2860
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P2860
Persistent sodium and calcium currents cause plateau potentials in motoneurons of chronic spinal rats.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Persistent sodium and calcium ...... eurons of chronic spinal rats.
@en
Persistent sodium and calcium ...... eurons of chronic spinal rats.
@nl
type
label
Persistent sodium and calcium ...... eurons of chronic spinal rats.
@en
Persistent sodium and calcium ...... eurons of chronic spinal rats.
@nl
prefLabel
Persistent sodium and calcium ...... eurons of chronic spinal rats.
@en
Persistent sodium and calcium ...... eurons of chronic spinal rats.
@nl
P2860
P356
P1476
Persistent sodium and calcium ...... eurons of chronic spinal rats.
@en
P2093
David J Bennett
P2860
P304
P356
10.1152/JN.00236.2003
P407
P577
2003-04-30T00:00:00Z