Oxidant regulation of gene expression and neural tube development: Insights gained from diabetic pregnancy on molecular causes of neural tube defects.
about
Bioactive Compounds and Their Neuroprotective Effects in Diabetic ComplicationsOxidative stress, unfolded protein response, and apoptosis in developmental toxicityLevels of polycyclic aromatic hydrocarbons in maternal serum and risk of neural tube defects in offspringAltered gene expression in rat cranial neural crest cells exposed to a teratogenic glucose concentration in vitro: paradoxical downregulation of antioxidative defense genesMaternal diabetes alters transcriptional programs in the developing embryo.Embryonic defence mechanisms against glucose-dependent oxidative stress require enhanced expression of Alx3 to prevent malformations during diabetic pregnancy.Oxidative stress during diabetic pregnancy disrupts cardiac neural crest migration and causes outflow tract defects.Deficient Vitamin E Uptake During Development Impairs Neural Tube Closure in Mice Lacking Lipoprotein Receptor SR-BI.Genetic variations in the GLUT3 gene associated with myelomeningocelePax3 stimulates p53 ubiquitination and degradation independent of transcriptionIncreased DNA methyltransferase 3b (Dnmt3b)-mediated CpG island methylation stimulated by oxidative stress inhibits expression of a gene required for neural tube and neural crest development in diabetic pregnancy.Transcriptome profiling of genes involved in neural tube closure during human embryonic development using long serial analysis of gene expression (long-SAGE).Birth defects and anti-heat shock protein 70 antibodies in early pregnancy.Use of a murine embryonic stem cell line that is sensitive to high glucose environment to model neural tube development in diabetic pregnancyMaternal diet modulates the risk for neural tube defects in a mouse model of diabetic pregnancyImpact of retinoic acid exposure on midfacial shape variation and manifestation of holoprosencephaly in Twsg1 mutant mice.Oxidative stress is responsible for maternal diabetes-impaired transforming growth factor beta signaling in the developing mouse heart.A transcriptional map of the impact of endurance exercise training on skeletal muscle phenotypeThe essential role of protein kinase Cδ in diabetes-induced neural tube defectsWhat neonatal complications should the pediatrician be aware of in case of maternal gestational diabetes?Advances in revealing the molecular targets downstream of oxidative stress-induced proapoptotic kinase signaling in diabetic embryopathy.AMP-activated protein kinase mediates effects of oxidative stress on embryo gene expression in a mouse model of diabetic embryopathy.Vitamin E mediates cell signaling and regulation of gene expression.Hyperglycemia impairs left-right axis formation and thereby disturbs heart morphogenesis in mouse embryos.Advances in understanding the molecular causes of diabetes-induced birth defects.Experimental mechanisms of diabetic embryopathy and strategies for developing therapeutic interventions.Association of copper-zinc superoxide dismutase (SOD1) and manganese superoxide dismutase (SOD2) genes with nonsyndromic myelomeningoceleNeural tube defects and maternal intake of micronutrients related to one-carbon metabolism or antioxidant activity.Frontiers in research on maternal diabetes-induced neural tube defects: Past, present and futureEmbryonic Stem Cell Proliferation Stimulated By Altered Anabolic Metabolism From Glucose Transporter 2-Transported Glucosamine.Novel single nucleotide polymorphisms in the superoxide dismutase 1 and 2 genes among children with myelomeningocele.Mouse embryonic stem cells established in physiological-glucose media express the high KM Glut2 glucose transporter expressed by normal embryosUnderstanding diabetic teratogenesis: where are we now and where are we going?Diabetes and apoptosis: neural crest cells and neural tube.Management of Type 2 diabetes and pregnancy.Genomics and genetics in the biology of adaptation to exercise.Lack of metformin effect on mouse embryo AMPK activity: implications for metformin treatment during pregnancy.The role of oxidative stress and antioxidants in diabetic complicationsMouse as a model for multifactorial inheritance of neural tube defects.Genetic and pharmacological inactivation of adenosine A2A receptor reveals an Egr-2-mediated transcriptional regulatory network in the mouse striatum.
P2860
Q26739955-314F07D5-D1B6-4AAB-8A36-CBB8C4FF93A7Q28084613-3D5F48C5-145F-449A-83EC-15C8BF58CA33Q28385786-A38DD6B6-12F3-48B8-9808-F3E88482C03CQ28576441-F86F81B9-77EA-465D-8B9D-B95305B0CF37Q33470737-BE93A101-4F53-44AC-BEA0-02D09445D177Q33670696-6813E980-0BDD-4A62-BDA6-386797E3999BQ33750553-7DAF3D43-AB3D-4C9D-94A6-381E93083665Q33899448-C21B8D16-5B18-48DC-8BDB-0822688917A7Q34111801-FF3331CF-C099-42F4-9565-1A3B0C5E8878Q34117536-DB34C845-C3AE-4D50-B047-4F6C88C73ACCQ34227874-EB663D28-7D10-47D9-A298-844BB7BA3AB6Q34341021-796387C1-CD95-44F4-93A5-C2F29637A1AAQ34446471-7A604970-FE2E-4219-A186-2F36D4F9CFD6Q34452969-40174FC9-73AB-4C74-839C-D27B7414BE90Q34560952-445C6AED-F0C1-4AD7-8E75-6A7EEED1271CQ35041346-4AEF17BA-2705-4B14-A128-3E8AA731CD0DQ35561759-ECA0FBBA-707B-4895-9C32-E9C948043702Q35658108-2095D133-4023-4A50-908F-0E3C4996DCF8Q35684260-2EFD9409-0B1D-4750-903E-EBE556B32B95Q35691385-DEE6506F-A203-46DF-A937-D6188AD5B4F6Q35847202-DC569B23-1441-460D-8C8B-7E04CF834581Q35927533-FD62A19E-2999-411A-A715-EEBB1C2C6176Q36064390-CC0059DA-6FCE-443F-A0C5-7FF86CCB5698Q36103084-75B95B9F-E18B-421B-BDB6-A5A0416C6630Q36319750-C2D42B09-A894-4AAA-8F8C-42454291AB79Q36330216-CAE2716B-998E-4FDA-A89B-D3796AE2BEBBQ36422334-01898017-6967-4155-966C-9D98B5BE3297Q36455973-26EE3F8E-6E12-44F0-8CDB-E4A057D82007Q36514459-868AD423-338A-4858-9BA4-24A149643E0AQ37014994-3062C366-B5A0-4911-BA67-0DD403AAA97DQ37204892-9A0BE543-3C56-408B-B6C4-030ADF2668E0Q37344264-81571C5C-2D2E-488B-BCFF-615F0C515B08Q37349983-C345202F-72CD-48A7-92E9-067CED09C153Q37351323-62B39141-43F7-4936-A26C-ABB5A464CCEBQ37608444-A775CDC7-5D7F-4A8E-92E6-BDF202E53AAFQ37609488-89E0FB28-9BFB-485F-8ACA-2DB8DC592E63Q37711344-99752CB6-D887-46A6-9D8E-D7810F6A3EA7Q37989150-043CD769-6ACA-4C34-BEB5-7970BCBD4C12Q38018292-B12DBC2B-ADD0-4E99-ADC2-BE3F3A24372EQ38323124-FA9C8F77-B2A4-4186-AF45-04B3BC78151A
P2860
Oxidant regulation of gene expression and neural tube development: Insights gained from diabetic pregnancy on molecular causes of neural tube defects.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Oxidant regulation of gene exp ...... causes of neural tube defects.
@en
Oxidant regulation of gene exp ...... causes of neural tube defects.
@nl
type
label
Oxidant regulation of gene exp ...... causes of neural tube defects.
@en
Oxidant regulation of gene exp ...... causes of neural tube defects.
@nl
prefLabel
Oxidant regulation of gene exp ...... causes of neural tube defects.
@en
Oxidant regulation of gene exp ...... causes of neural tube defects.
@nl
P2093
P2860
P1433
P1476
Oxidant regulation of gene exp ...... causes of neural tube defects.
@en
P2093
P2860
P2888
P304
P356
10.1007/S00125-003-1063-2
P577
2003-03-26T00:00:00Z