Overeating, alcohol and sucrose consumption decrease in CB1 receptor deleted mice.
about
The endocannabinoid system as an emerging target of pharmacotherapyGrowth Hormone Secretagogue Receptor Dimers: A New Pharmacological Target(1,2,3)The CB1 receptor as an important mediator of hedonic reward processing.Decreased reward sensitivity in rats from the Fischer344 strain compared to Wistar rats is paralleled by differences in endocannabinoid signalingNeuropharmacology of the endocannabinoid signaling system-molecular mechanisms, biological actions and synaptic plasticity.CB1 receptors: emerging evidence for central and peripheral mechanisms that regulate energy balance, metabolism, and cardiovascular health.The orexigenic effect of ghrelin is mediated through central activation of the endogenous cannabinoid system.The incentive salience of alcohol: translating the effects of genetic variant in CNR1.Rimonabant (SR141716) has no effect on alcohol self-administration or endocrine measures in nontreatment-seeking heavy alcohol drinkers.Effects of melanocortin receptor activation and blockade on ethanol intake: a possible role for the melanocortin-4 receptor.Suppressing effect of the cannabinoid CB1 receptor antagonist, SR147778, on alcohol intake and motivational properties of alcohol in alcohol-preferring sP rats.The relationship of in vivo central CB1 receptor occupancy to changes in cortical monoamine release and feeding elicited by CB1 receptor antagonists in rats.CB1 cannabinoid receptor-mediated modulation of food intake in mice.Innate difference in the endocannabinoid signaling and its modulation by alcohol consumption in alcohol-preferring sP rats.CB1 receptors regulate alcohol-seeking behavior and alcohol self-administration of alcohol-preferring (P) rats.Role of the endocannabinoid system in obesity induced by neuropeptide Y overexpression in noradrenergic neurons.The utility of animal models to evaluate novel anti-obesity agents.Effects of the cannabinoid antagonist SR141716 (rimonabant) and d-amphetamine on palatable food and food pellet intake in non-human primatesEndocannabinoid receptor antagonists: potential for obesity treatment.Role of endocannabinoids and cannabinoid CB1 receptors in alcohol-related behaviors.The endocannabinoid signaling system: a potential target for next-generation therapeutics for alcoholism.Sex and cannabinoid CB1 genotype differentiate palatable food and cocaine self-administration behaviors in micePositron emission tomography shows elevated cannabinoid CB1 receptor binding in men with alcohol dependenceRoles for the endocannabinoid system in ethanol-motivated behavior.Cannabinoid Ligands and Alcohol Addiction: A Promising Therapeutic Tool or a Humbug?Structural analogs of pyrazole and sulfonamide cannabinoids: effects on acute food intake in miceThe endocannabinoid system in brain reward processesBlocking the cannabinoid receptors: drug candidates and therapeutic promises.The cannabinoid CB1 receptor antagonist, rimonabant, as a promising pharmacotherapy for alcohol dependence: preclinical evidence.Cannabinoids in eating disorders and obesity.Dissociable effects of CB1 receptor blockade on anxiety-like and consummatory behaviors in the novelty-induced hypophagia test in mice.Tolerance to Ethanol or Nicotine Results in Increased Ethanol Self-Administration and Long-Term Depression in the Dorsolateral StriatumRole of endocannabinoid signaling in anxiety and depression.Resilience to meet the challenge of addiction: psychobiology and clinical considerations.Using genetically engineered animal models in the postgenomic era to understand gene function in alcoholism.Agonism of the endocannabinoid system modulates binge-like alcohol intake in male C57BL/6J mice: involvement of the posterior ventral tegmental area.Cannabinoids and appetite: food craving and food pleasure.Genes and Alcohol Consumption: Studies with Mutant Mice.Neurobiology of consummatory behavior: mechanisms underlying overeating and drug use.Translational strategies for therapeutic development in nicotine addiction: rethinking the conventional bench to bedside approach.
P2860
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P2860
Overeating, alcohol and sucrose consumption decrease in CB1 receptor deleted mice.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Overeating, alcohol and sucrose consumption decrease in CB1 receptor deleted mice.
@en
Overeating, alcohol and sucrose consumption decrease in CB1 receptor deleted mice.
@nl
type
label
Overeating, alcohol and sucrose consumption decrease in CB1 receptor deleted mice.
@en
Overeating, alcohol and sucrose consumption decrease in CB1 receptor deleted mice.
@nl
prefLabel
Overeating, alcohol and sucrose consumption decrease in CB1 receptor deleted mice.
@en
Overeating, alcohol and sucrose consumption decrease in CB1 receptor deleted mice.
@nl
P2093
P1433
P1476
Overeating, alcohol and sucrose consumption decrease in CB1 receptor deleted mice.
@en
P2093
Jeanne Maruani
Martine Poncelet
Philippe Soubrié
Roselyne Calassi
P304
P356
10.1016/S0304-3940(03)00397-5
P407
P577
2003-06-01T00:00:00Z