High glucose-mediated effects on endothelial cell proliferation occur via p38 MAP kinase.
about
p38 mitogen-activated protein kinase (MAPK) promotes cholesterol ester accumulation in macrophages through inhibition of macroautophagy.Beneficial effect of the oligomerized polyphenol oligonol on high glucose-induced changes in eNOS phosphorylation and dephosphorylation in endothelial cellsEffect of PIP3 on adhesion molecules and adhesion of THP-1 monocytes to HUVEC treated with high glucoseAntipermeability function of PEDF involves blockade of the MAP kinase/GSK/beta-catenin signaling pathway and uPAR expression.Effects of lycopene on number and function of human peripheral blood endothelial progenitor cells cultivated with high glucoseDiabetes alters vascular mechanotransduction: pressure-induced regulation of mitogen activated protein kinases in the rat inferior vena cava.Hyperglycemia alters PI3k and Akt signaling and leads to endothelial cell proliferative dysfunction.Ursodeoxycholic Acid (UDCA) Exerts Anti-Atherogenic Effects by Inhibiting RAGE Signaling in Diabetic Atherosclerosis.Effects of some anti-diabetic and cardioprotective agents on proliferation and apoptosis of human coronary artery endothelial cells.Insulin stimulates the clonogenic potential of angiogenic endothelial progenitor cells by IGF-1 receptor-dependent signaling.Imbalance of angiogenesis in diabetic complications: the mechanisms.Phosphatidylinositol-3,4,5-triphosphate and cellular signaling: implications for obesity and diabetes.High glucose promotes retinal endothelial cell migration through activation of Src, PI3K/Akt1/eNOS, and ERKs.Abnormal angiogenesis in diabetic nephropathy.Diabetes and Wound Angiogenesis.ERK regulation upon contact inhibition in fibroblasts.p21(WAF1/CIP1) Expression is Differentially Regulated by Metformin and Rapamycin.Diabetes-relevant regulation of cultured blood outgrowth endothelial cells.Endothelial dysfunction in diabetes: potential application of circulating markers as advanced diagnostic and prognostic toolsHigh-density lipoproteins attenuate high glucose-impaired endothelial cell signaling and functions: potential implications for improved vascular repair in diabetes.Aberrant angiogenesis: The gateway to diabetic complications.Effect of combined hormonal and insulin therapy on the steroid hormone receptors and growth factors signalling in diabetic mice prostate.Ablation of NF-kappaB expression by small interference RNA prevents the dysfunction of human umbilical vein endothelial cells induced by high glucose.
P2860
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P2860
High glucose-mediated effects on endothelial cell proliferation occur via p38 MAP kinase.
description
2003 nî lūn-bûn
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2003年の論文
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年學術文章
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name
High glucose-mediated effects on endothelial cell proliferation occur via p38 MAP kinase.
@en
High glucose-mediated effects on endothelial cell proliferation occur via p38 MAP kinase.
@nl
type
label
High glucose-mediated effects on endothelial cell proliferation occur via p38 MAP kinase.
@en
High glucose-mediated effects on endothelial cell proliferation occur via p38 MAP kinase.
@nl
prefLabel
High glucose-mediated effects on endothelial cell proliferation occur via p38 MAP kinase.
@en
High glucose-mediated effects on endothelial cell proliferation occur via p38 MAP kinase.
@nl
P2093
P2860
P921
P1476
High glucose-mediated effects on endothelial cell proliferation occur via p38 MAP kinase.
@en
P2093
P2860
P304
P356
10.1152/AJPENDO.00572.2002
P577
2003-06-03T00:00:00Z