Early programming of T cell populations responding to bacterial infection.
about
DOCK8 deficiency impairs CD8 T cell survival and function in humans and miceCytolysin-dependent escape of the bacterium from the phagosome is required but not sufficient for induction of the Th1 immune response against Listeria monocytogenes infection: distinct role of Listeriolysin O determined by cytolysin gene replacementEpidemiological models for the spread of anti-malarial resistanceThe Timing of T Cell Priming and CyclingA Human Trypanosome Suppresses CD8+ T Cell Priming by Dendritic Cells through the Induction of Immune Regulatory CD4+ Foxp3+ T CellsProlonged antigen presentation is required for optimal CD8+ T cell responses against malaria liver stage parasitesListeria monocytogenes: a model pathogen to study antigen-specific memory CD8 T cell responsesModeling T- and B-cell growth and differentiationImpaired response to Listeria in H2-M3-deficient mice reveals a nonredundant role of MHC class Ib-specific T cells in host defenseIntegrating Antimicrobial Therapy with Host Immunity to Fight Drug-Resistant Infections: Classical vs. Adaptive TreatmentIn vivo depletion of CD11c+ dendritic cells abrogates priming of CD8+ T cells by exogenous cell-associated antigensVaccination by delayed treatment of infection.High epitope expression levels increase competition between T cellsSalmonella pathogenicity island 2-mediated overexpression of chimeric SspH2 proteins for simultaneous induction of antigen-specific CD4 and CD8 T cells.Selectively reduced intracellular proliferation of Salmonella enterica serovar typhimurium within APCs limits antigen presentation and development of a rapid CD8 T cell response.Delayed expansion and contraction of CD8+ T cell response during infection with virulent Salmonella typhimurium.IFN-gamma induces the erosion of preexisting CD8 T cell memory during infection with a heterologous intracellular bacteriumA reduced antigen load in vivo, rather than weak inflammation, causes a substantial delay in CD8+ T cell priming against Mycobacterium bovis (bacillus Calmette-Guérin).Tracking the total CD8 T cell response to infection reveals substantial discordance in magnitude and kinetics between inbred and outbred hostsEvaluating the cellular targets of anti-4-1BB agonist antibody during immunotherapy of a pre-established tumor in mice.Attenuated Salmonella choleraesuis-mediated RNAi targeted to conserved regions against foot-and-mouth disease virus in guinea pigs and swineStochastic models of lymphocyte proliferation and deathMaturation-dependent licensing of naive T cells for rapid TNF production.Exploring the collaboration between antibiotics and the immune response in the treatment of acute, self-limiting infectionsLong-term protection against tuberculosis following vaccination with a severely attenuated double lysine and pantothenate auxotroph of Mycobacterium tuberculosis.Dendritic cells maximize the memory CD8 T cell response to infection.Recruitment times, proliferation, and apoptosis rates during the CD8(+) T-cell response to lymphocytic choriomeningitis virusNovel recombinant Mycobacterium bovis BCG, ovine atadenovirus, and modified vaccinia virus Ankara vaccines combine to induce robust human immunodeficiency virus-specific CD4 and CD8 T-cell responses in rhesus macaquesThe CD4 molecule on CD8+ T lymphocytes directly enhances the immune response to viral and cellular antigensConditional lethality yields a new vaccine strain of Listeria monocytogenes for the induction of cell-mediated immunityInterleukin-2 and inflammation induce distinct transcriptional programs that promote the differentiation of effector cytolytic T cells.Essential role of the Wnt pathway effector Tcf-1 for the establishment of functional CD8 T cell memory.Heterologous immunity between virusesSimulation studies for a multistage dynamic process of immune memory response to influenza: experiment in silico.Early expression of stem cell-associated genes within the CD8 compartment after treatment with a tumor vaccineCD8(+)-T-cell response to secreted and nonsecreted antigens delivered by recombinant Listeria monocytogenes during secondary infection.Preexisting inflammation due to Mycobacterium bovis BCG infection differentially modulates T-cell priming against a replicating or nonreplicating immunogen.Proline-rich tyrosine kinase-2 is critical for CD8 T-cell short-lived effector fateMemory CD8 T-cell differentiation during viral infection.Short-term antigen presentation and single clonal burst limit the magnitude of the CD8(+) T cell responses to malaria liver stages.
P2860
Q24293628-D864E8B2-8446-4158-8C7D-B1F09E22E0EBQ24675620-AD478E41-2D57-4189-B035-0947FE365424Q24805506-5B2D1589-5903-4DB1-87CE-5393E53C370EQ26776548-CE3644BC-716D-431C-BE51-04637F0B0B34Q27313504-3ACA240B-81CA-47E7-8C5F-E01ABBD8BAFFQ27314913-B788558C-4152-42EF-A3DA-EB331792924DQ28083008-4D895B6A-402E-413E-BA8E-BD6E7CCDB7CAQ28293446-0C103083-8FD1-4E4A-8DCF-4C62474961AEQ28506013-0BBB24BC-99CB-4C1F-BCE3-7732DDBB5DD3Q28551361-EDAAC2AC-6F2D-414B-B60C-F32ECC8A1EB0Q29615098-6E4BFC95-A9C0-4331-82D6-40F926814D57Q30408788-1D1FF4AA-2CBA-4C12-AD60-9ABB38705FC0Q33255329-BADB4A39-235B-428B-8B9E-FA295C0C031EQ33559019-999FF449-829A-4601-B54C-3226CF439F77Q33573164-10FC6F1B-9674-4399-8F38-BB68F124C924Q33590099-178CE047-FA60-4912-903E-6BCBAD54C721Q33590103-A941C418-87C8-438E-B161-83B33B5176A7Q33590110-A4D26828-40F1-4035-B722-753BF8786970Q33596976-8868B27A-9EAA-4D4A-9C5D-46C93BC807FCQ33603041-4C71DC0C-5DE6-447D-9BA8-E28024BB3232Q33676729-210CC938-E4D4-4B94-8FEE-678F42E8F3BFQ33716990-F3C59256-637D-44DF-9598-55386A0E7E52Q33760914-A28BFF08-57EC-4CF8-9A8C-FAB877904B2BQ33767602-E207B03D-0A4A-476C-97D8-0CDB1948EBECQ33796833-21D4BBBC-9E09-400C-BC58-77C423145B69Q33801974-176F72AB-2773-4A09-A3BB-C616237C54E8Q33848737-67CCCE47-C8B1-4682-A484-ED7AFCFA4B6EQ33877458-88EFC331-0093-41F3-A2A4-2A9EE9AE575FQ33928484-73A1505C-F66F-4A8C-B94A-D006CB3CF703Q33946547-C59E9B74-1277-44FC-ABD7-383864B4E038Q34003618-474AFC5C-2798-4DD8-A633-381958C6324DQ34006736-CDEE9141-BE55-4745-8AC5-D54E77063D6FQ34051846-E3211955-6195-4379-817E-1BF7F16C8975Q34067481-05911889-9B79-4BC1-994E-90D86F0EDA96Q34110774-EE849F26-15E6-410A-A58C-EE5CF794E3ADQ34115899-9274BF2A-E4C9-43E4-9109-28A68C12D7C5Q34120905-7C612BC3-3DF2-4854-935B-A3F5692BA9D1Q34136306-0571ECDF-1053-49FB-9B64-D8E4B7047F34Q34152220-524D411A-CAEE-41B8-995E-70E54FD4AB0AQ34154441-A8A86EC9-37E9-4815-BEC4-3B135EF212A0
P2860
Early programming of T cell populations responding to bacterial infection.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年学术文章
@wuu
2000年学术文章
@zh
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
@yue
2000年學術文章
@zh-hant
name
Early programming of T cell populations responding to bacterial infection.
@en
Early programming of T cell populations responding to bacterial infection.
@nl
type
label
Early programming of T cell populations responding to bacterial infection.
@en
Early programming of T cell populations responding to bacterial infection.
@nl
prefLabel
Early programming of T cell populations responding to bacterial infection.
@en
Early programming of T cell populations responding to bacterial infection.
@nl
P2093
P1476
Early programming of T cell populations responding to bacterial infection.
@en
P2093
P304
P356
10.4049/JIMMUNOL.165.12.6833
P407
P577
2000-12-01T00:00:00Z