Glycation end-product cross-link breaker reduces collagen and improves cardiac function in aging diabetic heart.
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The role of glucosamine-induced ER stress in diabetic atherogenesisDiabetic cardiomyopathy: pathophysiology and clinical featuresEmerging drugs for diabetic retinopathy.Expression and regulation of facilitative glucose transporters in equine insulin-sensitive tissue: from physiology to pathology.Structural arrangement of the cardiac collagen fibers of healthy and diabetic dogs.Assessment of cost and health resource utilization for elderly patients with heart failure and diabetes mellitusAge and the effectiveness of anti-hypertensive therapy on improvement in diastolic function.Screening for left ventricular hypertrophy in patients with type 2 diabetes mellitus in the community.Alagebrium in combination with exercise ameliorates age-associated ventricular and vascular stiffness.Reduced acute vascular injury and atherosclerosis in hyperlipidemic mice transgenic for lysozyme.Diabetic heart disease.Advanced glycation end product cross-linking: pathophysiologic role and therapeutic target in cardiovascular disease.Advanced glycation end products stimulate osteoblast apoptosis via the MAP kinase and cytosolic apoptotic pathways.Association of Metabolic Syndrome with the Cardioankle Vascular Index in Asymptomatic Korean Population.Advanced glycation end product cross-link breaker attenuates diabetes-induced cardiac dysfunction by improving sarcoplasmic reticulum calcium handling.Predictive value of advanced glycation end products for the development of post-infarction heart failure: a preliminary report.The diabetic myocardium.Remodeling of the rat distal colon in diabetes: function and ultrastructure.The impact of diabetes on heart failure: opportunities for intervention.Advanced glycation end-products (AGEs) and heart failure: pathophysiology and clinical implications.Aging and the cardiac collagen matrix: Novel mediators of fibrotic remodellingCardiac inflammation associated with a Western diet is mediated via activation of RAGE by AGEs.Advanced glycation end products accumulate in vascular smooth muscle and modify vascular but not ventricular properties in elderly hypertensive canines.The receptor for advanced glycation endproducts (RAGE) and cardiovascular disease.Emerging concepts in cardiac matrix biology.Diabetic Cardiomyopathy: An Immunometabolic Perspective.Targeting fibrosis for the treatment of heart failure: a role for transforming growth factor-β.Advanced glycation end products: role in pathology of diabetic cardiomyopathy.Targeting advanced glycation with pharmaceutical agents: where are we now?Atrial Remodeling And Atrial Fibrillation: Mechanistic Interactions And Clinical Implications.Cardioprotective effect of pioglitazone in diabetic and non-diabetic rats subjected to acute myocardial infarction involves suppression of AGE-RAGE axis and inhibition of apoptosis.In vitro model to study the effects of matrix stiffening on Ca2+ handling and myofilament function in isolated adult rat cardiomyocytes.Left ventricular diastolic dysfunction in asymptomatic type 2 diabetic patients: detection and evaluation by tissue Doppler imaging.Advanced glycation end products enhance expression of pro-apoptotic genes and stimulate fibroblast apoptosis through cytoplasmic and mitochondrial pathways.Blockage of receptor for advanced glycation end products prevents development of cardiac dysfunction in db/db type 2 diabetic mice.The presence of fructosamine in human aortic valves is associated with valve stiffness.Impaired left ventricular relaxation in type 2 diabetic rats is related to myocardial accumulation of N(epsilon)-(carboxymethyl) lysine.Left ventricular dysfunction in hypertensive patients with Type 2 diabetes mellitus.C16, a novel advanced glycation endproduct breaker, restores cardiovascular dysfunction in experimental diabetic rats.Phaseolamin treatment prevents oxidative stress and collagen deposition in the hearts of streptozotocin-induced diabetic rats.
P2860
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P2860
Glycation end-product cross-link breaker reduces collagen and improves cardiac function in aging diabetic heart.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Glycation end-product cross-li ...... ction in aging diabetic heart.
@en
Glycation end-product cross-li ...... ction in aging diabetic heart.
@nl
type
label
Glycation end-product cross-li ...... ction in aging diabetic heart.
@en
Glycation end-product cross-li ...... ction in aging diabetic heart.
@nl
prefLabel
Glycation end-product cross-li ...... ction in aging diabetic heart.
@en
Glycation end-product cross-li ...... ction in aging diabetic heart.
@nl
P2093
P2860
P1476
Glycation end-product cross-li ...... ction in aging diabetic heart.
@en
P2093
Ashwani Malhotra
Dorothy E Vatner
Garikiparthy N Jyothirmayi
Leonard G Meggs
Malthi R Masurekar
Stephen F Vatner
Timothy J Regan
P2860
P304
P356
10.1152/AJPHEART.00516.2003
P577
2003-08-28T00:00:00Z