Transduction of NO-bioactivity by the red blood cell in sepsis: novel mechanisms of vasodilation during acute inflammatory disease.
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Hypoxic Vasodilation by Red Blood Cells: Evidence for an S-Nitrosothiol-Based SignalRed cell physiology and signaling relevant to the critical care settingCurrent perspectives and challenges in understanding the role of nitrite as an integral player in nitric oxide biology and therapyHemoglobin conformation couples erythrocyte S-nitrosothiol content to O2 gradients.Extrapulmonary effects of inhaled nitric oxide: role of reversible S-nitrosylation of erythrocytic hemoglobin.Inhaled NO accelerates restoration of liver function in adults following orthotopic liver transplantation.Water-soluble triarylphosphines as biomarkers for protein S-nitrosationInclusion of a nitric oxide congener in the insufflation gas repletes S-nitrosohemoglobin and stabilizes physiologic status during prolonged carbon dioxide pneumoperitoneum.Evolution of adverse changes in stored RBCsS-nitrosohemoglobin deficiency: a mechanism for loss of physiological activity in banked blood.Roles of dietary inorganic nitrate in cardiovascular health and disease.Erythrocyte-dependent regulation of human skeletal muscle blood flow: role of varied oxyhemoglobin and exercise on nitrite, S-nitrosohemoglobin, and ATP.Peroxiredoxin-2 recycling is inhibited during erythrocyte storage.A randomized clinical trial testing the anti-inflammatory effects of preemptive inhaled nitric oxide in human liver transplantation.Hemoglobin βCys93 is essential for cardiovascular function and integrated response to hypoxia.Requirement of transmembrane transport for S-nitrosocysteine-dependent modification of intracellular thiols.Erythrocytes are the major intravascular storage sites of nitrite in human blood.Hypoxia, red blood cells, and nitrite regulate NO-dependent hypoxic vasodilationArtery-to-vein differences in nitric oxide metabolites are diminished in sepsis.Poly(ADP-ribose) polymerase 1 inhibition improves coronary arteriole function in type 2 diabetes mellitus.Chronic inhibition of epidermal growth factor receptor tyrosine kinase and extracellular signal-regulated kinases 1 and 2 (ERK1/2) augments vascular response to limb ischemia in type 2 diabetic mice.Enzymatic mechanisms regulating protein S-nitrosylation: implications in health and disease.Effect of processing and storage on red blood cell function in vivoS-Nitrosothiol analysis via photolysis and amperometric nitric oxide detection in a microfluidic device.How do red blood cells cause hypoxic vasodilation? The SNO-hemoglobin paradigm.S-nitrosylation: integrator of cardiovascular performance and oxygen delivery.Nitrite as a vascular endocrine nitric oxide reservoir that contributes to hypoxic signaling, cytoprotection, and vasodilation.Paclitaxel injection concentrate for nanodispersion versus nab-paclitaxel in women with metastatic breast cancer: a multicenter, randomized, comparative phase II/III study.Transport and peripheral bioactivities of nitrogen oxides carried by red blood cell hemoglobin: role in oxygen delivery.Repletion of S-nitrosohemoglobin improves organ function and physiological status in swine after brain death.Protein kinase G oxidation is a major cause of injury during sepsis.Regulation of nitrite transport in red blood cells by hemoglobin oxygen fractional saturationSNO-hemoglobin is not essential for red blood cell-dependent hypoxic vasodilation.Activation and inhibition of soluble guanylyl cyclase by S-nitrosocysteine: involvement of amino acid transport system L.Antioxidant functions for the hemoglobin β93 cysteine residue in erythrocytes and in the vascular compartment in vivo.Hemoglobin, nitric oxide and molecular mechanisms of hypoxic vasodilationNitrate decreases xanthine oxidoreductase-mediated nitrite reductase activity and attenuates vascular and blood pressure responses to nitrite.Differential nitros(yl)ation of blood and tissue constituents during glyceryl trinitrate biotransformation in vivo.S-Nitrosothiol biology and therapeutic potential in metabolic disease.Nitric oxide formation versus scavenging: the red blood cell balancing act.
P2860
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P2860
Transduction of NO-bioactivity by the red blood cell in sepsis: novel mechanisms of vasodilation during acute inflammatory disease.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Transduction of NO-bioactivity ...... ng acute inflammatory disease.
@en
Transduction of NO-bioactivity ...... ng acute inflammatory disease.
@nl
type
label
Transduction of NO-bioactivity ...... ng acute inflammatory disease.
@en
Transduction of NO-bioactivity ...... ng acute inflammatory disease.
@nl
prefLabel
Transduction of NO-bioactivity ...... ng acute inflammatory disease.
@en
Transduction of NO-bioactivity ...... ng acute inflammatory disease.
@nl
P2093
P1433
P1476
Transduction of NO-bioactivity ...... ng acute inflammatory disease.
@en
P2093
Balu K Chacko
Barbora Piknova
Heather M Pruitt
Jack H Crawford
P304
P356
10.1182/BLOOD-2004-03-0880
P407
P577
2004-05-18T00:00:00Z