A cytokine gene screen uncovers SOCS1 as genetic risk factor for multiple sclerosis.
about
Immunoregulatory molecules are master regulators of inflammation during the immune responseGene expression studies of a human monocyte cell line identify dissimilarities between differently manufactured glatiramoidsInvolvement of the janus kinase/signal transducer and activator of transcription signaling pathway in multiple sclerosis and the animal model of experimental autoimmune encephalomyelitis.Association of interleukin 7 receptor gene polymorphism rs6897932 with multiple sclerosis patients in Khuzestan.Fine mapping and functional analysis of the multiple sclerosis risk gene CD6Multiple Sclerosis Risk Allele in CLEC16A Acts as an Expression Quantitative Trait Locus for CLEC16A and SOCS1 in CD4+ T Cells.Interleukin-22 is increased in multiple sclerosis patients and targets astrocytesRegulation of Macrophage, Dendritic Cell, and Microglial Phenotype and Function by the SOCS Proteins.Elevated type I interferon-like activity in a subset of multiple sclerosis patients: molecular basis and clinical relevance.SncRNA (microRNA &snoRNA) opposite expression pattern found in multiple sclerosis relapse and remission is sex dependent.Specific aspects of modern life for people with multiple sclerosis: considerations for the practitionerOpportunities for Translation from the Bench: Therapeutic Intervention of the JAK/STAT Pathway in Neuroinflammatory Diseases.Emerging role of interleukin-22 in autoimmune diseasesCytokine gene polymorphisms and human autoimmune disease in the era of genome-wide association studiesFrom Identification to Characterization of the Multiple Sclerosis Susceptibility Gene CLEC16A.Toll-Like Receptor Pathways in Autoimmune Diseases.The impact of the interferon-lambda family on the innate and adaptive immune response to viral infections.Novel Insights into the Multiple Sclerosis Risk Gene ANKRD55.Regulation of suppressors of cytokine signaling as a therapeutic approach in autoimmune diseases, with an emphasis on multiple sclerosis.Suppressor of Cytokine Signaling (SOCS)1 Regulates Interleukin-4 (IL-4)-activated Insulin Receptor Substrate (IRS)-2 Tyrosine Phosphorylation in Monocytes and Macrophages via the Proteasome.Expression of interleukin-22 in myasthenia gravis.Molecular Genetic and Epigenetic Basis of Multiple Sclerosis.IFNL1 binds IL10RB:TYK2 and IFNLR1:JAK1IL-22 binding protein regulates murine skin inflammation.Methylation differences at the HLA-DRB1 locus in CD4+ T-Cells are associated with multiple sclerosis.
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A cytokine gene screen uncovers SOCS1 as genetic risk factor for multiple sclerosis.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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name
A cytokine gene screen uncovers SOCS1 as genetic risk factor for multiple sclerosis.
@en
A cytokine gene screen uncovers SOCS1 as genetic risk factor for multiple sclerosis.
@nl
type
label
A cytokine gene screen uncovers SOCS1 as genetic risk factor for multiple sclerosis.
@en
A cytokine gene screen uncovers SOCS1 as genetic risk factor for multiple sclerosis.
@nl
prefLabel
A cytokine gene screen uncovers SOCS1 as genetic risk factor for multiple sclerosis.
@en
A cytokine gene screen uncovers SOCS1 as genetic risk factor for multiple sclerosis.
@nl
P2093
P2860
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A cytokine gene screen uncovers SOCS1 as genetic risk factor for multiple sclerosis.
@en
P2093
A Antigüedad
B Swaminathan
G Izquierdo
J R Oksenberg
M Comabella
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P2888
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10.1038/GENE.2011.44
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P577
2011-06-30T00:00:00Z