FIP200, an essential component of mammalian autophagy is indispensible for fetal hematopoiesis.
about
Autophagy in stem cells.Mutation of kri1l causes definitive hematopoiesis failure via PERK-dependent excessive autophagy induction.Connecting autophagy: AMBRA1 and its network of regulation.Autophagy: regulation and role in developmentRegulation of autophagy in hematological malignancies: role of reactive oxygen species.Defective autophagy in Parkinson's disease: role of oxidative stress.Autophagy in cancer stem/progenitor cells.Autophagy in stem and progenitor cells.Mitochondrial fusion by pharmacological manipulation impedes somatic cell reprogramming to pluripotency: new insight into the role of mitophagy in cell stemness.Dissecting the function of Atg1 complex in Dictyostelium autophagy reveals a connection with the pentose phosphate pathway enzyme transketolase.
P2860
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P2860
FIP200, an essential component of mammalian autophagy is indispensible for fetal hematopoiesis.
description
2011 nî lūn-bûn
@nan
2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年學術文章
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2011年學術文章
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name
FIP200, an essential component ...... sible for fetal hematopoiesis.
@en
FIP200, an essential component ...... sible for fetal hematopoiesis.
@nl
type
label
FIP200, an essential component ...... sible for fetal hematopoiesis.
@en
FIP200, an essential component ...... sible for fetal hematopoiesis.
@nl
prefLabel
FIP200, an essential component ...... sible for fetal hematopoiesis.
@en
FIP200, an essential component ...... sible for fetal hematopoiesis.
@nl
P356
P1433
P1476
FIP200, an essential component ...... sible for fetal hematopoiesis.
@en
P2093
P304
P356
10.4161/AUTO.7.2.14125
P577
2011-02-01T00:00:00Z