Mobilization studies in mice deficient in either C3 or C3a receptor (C3aR) reveal a novel role for complement in retention of hematopoietic stem/progenitor cells in bone marrow. - Wikidata - awgldk - TriplyDB

Mobilization studies in mice deficient in either C3 or C3a receptor (C3aR) reveal a novel role for complement in retention of hematopoietic stem/progenitor cells in bone marrow.

Mobilization studies in mice deficient in either C3 or C3a receptor (C3aR) reveal a novel role for complement in retention of hematopoietic stem/progenitor cells in bone marrow.

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Beta-glucan enhances complement-mediated hematopoietic recovery after bone marrow injury How to Improve Cord Blood Engraftment?Complement activation in the context of stem cells and tissue repair Potential Reparative Role of Resident Adult Renal Stem/Progenitor Cells in Acute Kidney Injury Advances in umbilical cord blood manipulation-from niche to bedside The complement system and adverse pregnancy outcomes A novel role for bioactive lipids in stem cell mobilization during cardiac ischemia: new paradigms in thrombosis: novel mediators and biomarkers Complement fragment 3a priming of umbilical cord blood progenitors: safety profile.The bone marrow-expressed antimicrobial cationic peptide LL-37 enhances the responsiveness of hematopoietic stem progenitor cells to an SDF-1 gradient and accelerates their engraftment after transplantation Comparative analysis of the acute response of the trout, O. mykiss, head kidney to in vivo challenge with virulent and attenuated infectious hematopoietic necrosis virus and LPS-induced inflammation Mobilization studies in complement-deficient mice reveal that optimal AMD3100 mobilization of hematopoietic stem cells depends on complement cascade activation by AMD3100-stimulated granulocytes.The complement cascade as a mediator of tissue growth and regeneration.Haploidentical vs cord blood transplantation for adults with acute myelogenous leukemia.Novel evidence that crosstalk between the complement, coagulation and fibrinolysis proteolytic cascades is involved in mobilization of hematopoietic stem/progenitor cells (HSPCs)Complement C1q enhances homing-related responses of hematopoietic stem/progenitor cells.The CXCR4 and adhesion molecule expression of CD34+ hematopoietic cells mobilized by "on-demand" addition of plerixafor to granulocyte-colony-stimulating factor.Cells enriched in markers of neural tissue-committed stem cells reside in the bone marrow and are mobilized into the peripheral blood following stroke.Complement: a novel factor in basal and ischemia-induced neurogenesis Cellular players of hematopoietic stem cell mobilization in the bone marrow niche.Cyclophosphamide induces dynamic alterations in the host microenvironments resulting in a Flt3 ligand-dependent expansion of dendritic cells.The role of the anaphylatoxins in health and disease.The ins and outs of hematopoietic stem cells: studies to improve transplantation outcomes.Complement component 3 is necessary to preserve myocardium and myocardial function in chronic myocardial infarction.The Therapeutic Effects of Human Mesenchymal Stem Cells Primed with Sphingosine-1 Phosphate on Pulmonary Artery Hypertension The role of complement in the trafficking of hematopoietic stem/progenitor cells.The relative merits of therapies being developed to tackle inappropriate ('self'-directed) complement activation Evidence that a lipolytic enzyme--hematopoietic-specific phospholipase C-β2--promotes mobilization of hematopoietic stem cells by decreasing their lipid raft-mediated bone marrow retention and increasing the promobilizing effects of granulocytes.Bioactive lipids and cationic antimicrobial peptides as new potential regulators for trafficking of bone marrow-derived stem cells in patients with acute myocardial infarction.CXCR4 expression in CD34+ cells and unit predominance after double umbilical cord blood transplantation Mobilization of hematopoietic stem/progenitor cells: general principles and molecular mechanisms.The receptor for complement component C3a mediates protection from intestinal ischemia-reperfusion injuries by inhibiting neutrophil mobilization.Complement anaphylatoxin C3a is a potent inducer of embryonic chick retina regeneration.Genome-wide analysis of murine bone marrow‑derived very small embryonic-like stem cells reveals that mitogenic growth factor signaling pathways play a crucial role in the quiescence and ageing of these cells.Dynamic Cross Talk between S1P and CXCL12 Regulates Hematopoietic Stem Cells Migration, Development and Bone Remodeling Impaired mobilization of hematopoietic stem/progenitor cells in C5-deficient mice supports the pivotal involvement of innate immunity in this process and reveals novel promobilization effects of granulocytes Recent developments in low molecular weight complement inhibitors Complement-triggered pathways orchestrate regenerative responses throughout phylogenesis.Future of cord blood for oncology uses.Mobilization of Peripheral Blood Stem Cells and Changes in the Concentration of Plasma Factors Influencing their Movement in Patients with Panic Disorder.Innate immunity as orchestrator of stem cell mobilization.

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Mobilization studies in mice deficient in either C3 or C3a receptor (C3aR) reveal a novel role for complement in retention of hematopoietic stem/progenitor cells in bone marrow.

http://www.wikidata.org/entity/Q44646941

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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name

Mobilization studies in mice d ...... ogenitor cells in bone marrow.

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Mobilization studies in mice deficient in either C3 or C3a receptor

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type

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Mobilization studies in mice d ...... ogenitor cells in bone marrow.

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Mobilization studies in mice deficient in either C3 or C3a receptor

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Mobilization studies in mice d ...... ogenitor cells in bone marrow.

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Mobilization studies in mice deficient in either C3 or C3a receptor

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Anna Janowska-Wieczorek

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Daniel J Allendorf

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Gordon D Ross

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Janina Ratajczak

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Jarek T Baran

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Magda Kucia

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Marcin Majka

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Rick A Wetsel

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Ryan Reca

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2071-2078

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scholarly article

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10.1182/BLOOD-2003-06-2099

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103

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Mariusz Ratajczak

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2003-11-06T00:00:00Z

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9019896

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