Ontogeny of sensorimotor gating and immune impairment induced by prenatal immune challenge in rats: implications for the etiopathology of schizophrenia.
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Inflammatory cytokines and neurological and neurocognitive alterations in the course of schizophreniaThe cytokine model of schizophrenia: emerging therapeutic strategiesInflammation and schizophreniaCytokine dysregulation in autism spectrum disorders (ASD): possible role of the environmentPrenatal inflammation-induced hypoferremia alters dopamine function in the adult offspring in rat: relevance for schizophrenia.Using a maternal immune stimulation model of schizophrenia to study behavioral and neurobiological alterations over the developmental course.Nicotine ameliorates schizophrenia-like cognitive deficits induced by maternal LPS exposure: a study in rats.Reductions in synaptic proteins and selective alteration of prepulse inhibition in male C57BL/6 mice after postnatal administration of a VIP receptor (VIPR2) agonist.Astaxanthin ameliorates prenatal LPS-exposed behavioral deficits and oxidative stress in adult offspring.Neurodevelopmental animal models of schizophrenia: role in novel drug discovery and developmentPriming of metabolic dysfunctions by prenatal immune activation in mice: relevance to schizophrenia.Models of neurodevelopmental abnormalities in schizophrenia.Effect of maternal lipopolysaccharide administration on the development of dopaminergic receptors and transporter in the rat offspring.Sex effects on neurodevelopmental outcomes of innate immune activation during prenatal and neonatal life.Schizophrenia and autism: both shared and disorder-specific pathogenesis via perinatal inflammation?Prenatal stress: role in psychotic and depressive diseases.Pallidal hyperdopaminergic innervation underlying D2 receptor-dependent behavioral deficits in the schizophrenia animal model established by EGFVariants of the RELA gene are associated with schizophrenia and their startle responsesPrenatal immune challenge is an environmental risk factor for brain and behavior change relevant to schizophrenia: evidence from MRI in a mouse modelNeuroimmunological aberrations and cerebral asymmetry abnormalities in schizophrenia: select perspectives on pathogenesisMaternal LPS exposure during pregnancy impairs testicular development, steroidogenesis and spermatogenesis in male offspring.Individual differences in maternal response to immune challenge predict offspring behavior: contribution of environmental factors.LPS exposure increases maternal corticosterone levels, causes placental injury and increases IL-1Β levels in adult rat offspring: relevance to autism.Effects of risperidone and paliperidone pre-treatment on locomotor response following prenatal immune activationLipopolysaccharide Exposure Induces Maternal Hypozincemia, and Prenatal Zinc Treatment Prevents Autistic-Like Behaviors and Disturbances in the Striatal Dopaminergic and mTOR Systems of OffspringInflammation impairs social cognitive processing: A randomized controlled trial of endotoxinFluoxetine and aripiprazole treatment following prenatal immune activation exert longstanding effects on rat locomotor response.Modulation of schizophrenia-related genes in the forebrain of adolescent and adult rats exposed to maternal immune activation.Effects of prenatal immune activation and peri-adolescent stress on amphetamine-induced conditioned place preference in the rat.Prenatal protein deprivation alters dopamine-mediated behaviors and dopaminergic and glutamatergic receptor binding.PRENATAL INFECTION, MATERNAL IMMUNE ACTIVATION, AND RISK FOR SCHIZOPHRENIA.Prenatal and postnatal animal models of immune activation: relevance to a range of neurodevelopmental disorders.The immune theory of psychiatric diseases: a key role for activated microglia and circulating monocytes.A starring role for microglia in brain sex differences.Maternal immune activation and abnormal brain development across CNS disorders.Disrupted Circadian Rhythm as a Common Player in Developmental Models of Neuropsychiatric Disorders.Maternal immune activation produces cerebellar hyperplasia and alterations in motor and social behaviors in male and female mice.Positive modulation of α5 GABAA receptors in preadolescence prevents reduced locomotor response to amphetamine in adult female but not male rats prenatally exposed to lipopolysaccharide.N-acetyl-cysteine prevents pyramidal cell disarray and reelin-immunoreactive neuron deficiency in CA3 after prenatal immune challenge in rats.Hypoactivity of the central dopaminergic system and autistic-like behavior induced by a single early prenatal exposure to lipopolysaccharide.
P2860
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P2860
Ontogeny of sensorimotor gating and immune impairment induced by prenatal immune challenge in rats: implications for the etiopathology of schizophrenia.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Ontogeny of sensorimotor gatin ...... tiopathology of schizophrenia.
@en
Ontogeny of sensorimotor gatin ...... tiopathology of schizophrenia.
@nl
type
label
Ontogeny of sensorimotor gatin ...... tiopathology of schizophrenia.
@en
Ontogeny of sensorimotor gatin ...... tiopathology of schizophrenia.
@nl
prefLabel
Ontogeny of sensorimotor gatin ...... tiopathology of schizophrenia.
@en
Ontogeny of sensorimotor gatin ...... tiopathology of schizophrenia.
@nl
P2093
P2860
P356
P1433
P1476
Ontogeny of sensorimotor gatin ...... tiopathology of schizophrenia.
@en
P2093
P2860
P2888
P304
P356
10.1038/MP.2008.44
P407
P577
2008-04-15T00:00:00Z
P5875
P6179
1036724768