Increased persistent sodium currents in rat entorhinal cortex layer V neurons in a post-status epilepticus model of temporal lobe epilepsy.
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Enhancement of asynchronous release from fast-spiking interneuron in human and rat epileptic neocortexCurrent research on opioid receptor function.The neurobiology of antiepileptic drugsPhenytoin inhibits the persistent sodium current in neocortical neurons by modifying its inactivation propertiesLacosamide: a new approach to target voltage-gated sodium currents in epileptic disordersPersistent Sodium Current and Its Role in EpilepsyEvidence for a role of Nav1.6 in facilitating increases in neuronal hyperexcitability during epileptogenesis.Temporal lobe epilepsy induces intrinsic alterations in Na channel gating in layer II medial entorhinal cortex neurons.A selective interplay between aberrant EPSPKA and INaP reduces spike timing precision in dentate granule cells of epileptic rats.Reciprocal changes in phosphorylation and methylation of mammalian brain sodium channels in response to seizuresSaikosaponin a mediates the anticonvulsant properties in the HNC models of AE and SE by inhibiting NMDA receptor current and persistent sodium current.delta-Opioid receptors protect from anoxic disruption of Na+ homeostasis via Na+ channel regulationIntrinsic neurophysiological properties of hilar ectopic and normotopic dentate granule cells in human temporal lobe epilepsy and a rat model.Transient sodium current at subthreshold voltages: activation by EPSP waveformsMixed-mode oscillations in pyramidal neurons under antiepileptic drug conditions.Persistent sodium current drives conditional pacemaking in CA1 pyramidal neurons under muscarinic stimulationNa+ channelopathies and epilepsy: recent advances and new perspectives.Cav 1.3 channels play a crucial role in the formation of paroxysmal depolarization shifts in cultured hippocampal neurons.Carbamazepine and topiramate modulation of transient and persistent sodium currents studied in HEK293 cells expressing the Na(v)1.3 alpha-subunit.Activity-dependent alternative splicing increases persistent sodium current and promotes seizurePersistent sodium current in subicular neurons isolated from patients with temporal lobe epilepsy.Pro-excitatory alterations in sodium channel activity facilitate subiculum neuron hyperexcitability in temporal lobe epilepsy.Diversity and excitability of deep-layer entorhinal cortical neurons in a model of temporal lobe epilepsy.Layer-specific properties of the persistent sodium current in sensorimotor cortex.An increase in persistent sodium current contributes to intrinsic neuronal bursting after status epilepticus.
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P2860
Increased persistent sodium currents in rat entorhinal cortex layer V neurons in a post-status epilepticus model of temporal lobe epilepsy.
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2003 nî lūn-bûn
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name
Increased persistent sodium cu ...... del of temporal lobe epilepsy.
@en
Increased persistent sodium cu ...... del of temporal lobe epilepsy.
@nl
type
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Increased persistent sodium cu ...... del of temporal lobe epilepsy.
@en
Increased persistent sodium cu ...... del of temporal lobe epilepsy.
@nl
prefLabel
Increased persistent sodium cu ...... del of temporal lobe epilepsy.
@en
Increased persistent sodium cu ...... del of temporal lobe epilepsy.
@nl
P2093
P2860
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Increased persistent sodium cu ...... del of temporal lobe epilepsy.
@en
P2093
Angel Alonso
David S Ragsdale
Newton Agrawal
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P304
P356
10.1111/J.0013-9580.2003.23103.X
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2003-12-01T00:00:00Z